ATP - sensitive potassium channelscausingmembrane depolarization
inhibit ATP - sensitive potassium channelscausingmembrane depolarization
ATP inhibit ATP - sensitive potassium channelscausingmembrane depolarization
allows entry of cations , including calcium ( Ca2 + ) , into the cellto causemembrane depolarization
which then promote closure of ATP - sensitive K+ ( KATP ) channelscausingmembrane depolarization
Inhibition of K+ channelscausesmembrane depolarization
by inhibition of potassium channels that otherwise maintain the resting potential(passive) is caused bymembrane depolarization
by inhibition of potassium channels that otherwise maintain the resting potential(passive) is causedmembrane depolarization
inhibition of potassium channels that otherwise maintain the resting potential(passive) is caused bymembrane depolarization
potassium channelscausingmembrane depolarization
Extracellular potassium accumulation is known ... , and has been associated with conduction block in central nervous system neuronsto causemembrane depolarization
High K+ stimulationprovokesmembrane depolarization
the inhibition of K(+ ) channel by NAcausedmembrane depolarization
that increases in intraluminal pressure leads to the activation of cation channels in smooth muscle cellsto causemembrane depolarization
by inhibition of potassium channels that otherwise maintain the resting potential.[4(passive) is caused bymembrane depolarization
by inhibition of potassium channels that otherwise maintain the resting potential.[4(passive) is causedmembrane depolarization
by inhibition of potassium channels that otherwise maintain the resting(passive) is caused bymembrane depolarization
by inhibition of potassium channels that otherwise maintain the resting(passive) is causedmembrane depolarization
The rapid movement into the cellcausesmembrane " depolarization
by increased content in unsaturated fatty acids(passive) caused bycellular membrane fluidity
activation of GABAA receptorscausesmembrane depolarization
by elevated extracellular K+(passive) caused bymembrane depolarization
Reduced KATP channel activity is in turn expectedto causemembrane depolarization
anion ) channels on the plasma membranecausingmembrane depolarization
to increased permeability of the membraneleadsto increased permeability of the membrane
in enhanced passive diffusion of the drugs ( 30resultingin enhanced passive diffusion of the drugs ( 30
alsoresultsalso
greater uptake of auranofin(passive) could be influenced bygreater uptake of auranofin
to the opening of voltage - dependent calcium channels and subsequent calcium entryleadingto the opening of voltage - dependent calcium channels and subsequent calcium entry
to plasma membrane blebbing and sheddingleadingto plasma membrane blebbing and shedding
to opening of voltage dependent calcium channelsleadingto opening of voltage dependent calcium channels
voltage - gated Ca2 + channels to opencausesvoltage - gated Ca2 + channels to open
to the opening of voltage - gated Ca2 + channels in the plasma membraneleadsto the opening of voltage - gated Ca2 + channels in the plasma membrane
Ca2 + entry through voltage - operated Ca2 + channels , activation of Ca2causingCa2 + entry through voltage - operated Ca2 + channels , activation of Ca2
opening of voltage dependent Ca2 +causesopening of voltage dependent Ca2 +
opening of voltage dependent Ca2causesopening of voltage dependent Ca2
to the generation of Ca2 + spike potentials and an increase in intracellular Ca2ledto the generation of Ca2 + spike potentials and an increase in intracellular Ca2
to calcium influx through L - type calcium channelsleadingto calcium influx through L - type calcium channels
to the opening of Ca2 + channelsleadingto the opening of Ca2 + channels
of a lipid bilayer of phospholipids with the hydrophobic edge screened by two amphipathic proteinscomposedof a lipid bilayer of phospholipids with the hydrophobic edge screened by two amphipathic proteins
to an influx of calciumleadingto an influx of calcium
Increase in potassium ion in extracellular(passive) is causedIncrease in potassium ion in extracellular
to cell contractionleadsto cell contraction
to Ca influx through L - type Ca channelsleadingto Ca influx through L - type Ca channels
release of the excitatory neurotransmitter glutamatecausesrelease of the excitatory neurotransmitter glutamate
to the delivery of active enzymes at the cell surfaceleadingto the delivery of active enzymes at the cell surface
in extracellular calcium entryresultingin extracellular calcium entry
to cell death by directly inhibiting potassium channelsleadsto cell death by directly inhibiting potassium channels
to firing of action potentialsleadingto firing of action potentials
to increased firingleadto increased firing
to calciumleadingto calcium
Ca2 + entrycausingCa2 + entry
to Ca2 +leadingto Ca2 +
to Ca2leadingto Ca2
in increasedresultingin increased
to the action potentialleadingto the action potential
to increased blockingleadsto increased blocking
to increased blockingleadsto increased blocking
in turnleadsin turn
a nerve impulse or action potential if a certain threshold is reached within the neuroncan causea nerve impulse or action potential if a certain threshold is reached within the neuron
an opening of the gates that previously had an inhibitory effect on the neuron they were gatingwill causean opening of the gates that previously had an inhibitory effect on the neuron they were gating
to excitationDoes ... always leadto excitation
to 7leadingto 7
Excitatory responses(passive) are caused byExcitatory responses