free radicals during general anesthesia presents as observed lipid peroxidation products.13 Studies have shown that a variety of medications used in anesthesia have effects on the oxidant - antioxidant system(passive) caused byMembrane damage
reactive oxygen species ( ROS ) formation with subsequent lipid peroxidationcausesmembrane damage
free radicals via lipid peroxidation23,24 and PI labelling of membrane - compromised S. mutans cells as observed via confocal imaging ( Fig . 2a(passive) caused bymembrane damage
reactive oxygen species and DNA degradation(passive) provoked byMembrane damage
to be an efficient producer of reactive oxygen speciesoften leadingto membrane damage
Its role ... excess consumption of saturated fats , alcohol , and drugs , among otherscausemembrane damage
necrosis and cell deathcausingnecrosis and cell death
directly to cell deathleadsdirectly to cell death
to high cell deathleadsto high cell death
to rapid cell deathleadsto rapid cell death
in apoptotic cell deathresultingin apoptotic cell death
to cell necrosis and premature cell death ( Hildebrand , 1998leadingto cell necrosis and premature cell death ( Hildebrand , 1998
to the leakage of mitochondrial nucleic acids ... immune responses and/or cell deathleadsto the leakage of mitochondrial nucleic acids ... immune responses and/or cell death
to cell death during HR response ( Lee et al . , 2009leadingto cell death during HR response ( Lee et al . , 2009
to bacterial cell death.97 , 98 , 99 Structures of compounds in phase - I clinical trialsleadingto bacterial cell death.97 , 98 , 99 Structures of compounds in phase - I clinical trials
to bacterial cell death.97 , 98 , 99Figure 7.Structures of compounds in phase - I clinical trialsleadingto bacterial cell death.97 , 98 , 99Figure 7.Structures of compounds in phase - I clinical trials
to a direct intracellular attack which induces cell death that can not be repaired during the post - treatment storage period ( Huang et al .leadingto a direct intracellular attack which induces cell death that can not be repaired during the post - treatment storage period ( Huang et al .
to cell death ... 33 ... even if they can also have positive implications due to the possible signalling nature of some of the species produced [ 34 - 36leadsto cell death ... 33 ... even if they can also have positive implications due to the possible signalling nature of some of the species produced [ 34 - 36
measurement of electrolyte leakage(passive) caused bymeasurement of electrolyte leakage
internal cell materials ( potassium ion , galaktosidase , low molecular solutes , amino acids , RMA , DNA ) leakage , which results in cell deathcausesinternal cell materials ( potassium ion , galaktosidase , low molecular solutes , amino acids , RMA , DNA ) leakage , which results in cell death
to leakage of essential cytoplasmic constituents ( 15leadingto leakage of essential cytoplasmic constituents ( 15
to the leakage of essential cytoplasmic constituents ( 30leadingto the leakage of essential cytoplasmic constituents ( 30
to the hypersensitive reaction in lettuce Bestwick , CS ; Bennett , MH ; Mansfield , JW Recognition and response in plant – fungus interactions Mansfield , JW Dose - dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli Lennon , SV ; Martin , SJ ; Cotter , TG Hydrogen peroxide as a mediator of programmed cell death in the blastocyst Pierce , GBleadingto the hypersensitive reaction in lettuce Bestwick , CS ; Bennett , MH ; Mansfield , JW Recognition and response in plant – fungus interactions Mansfield , JW Dose - dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli Lennon , SV ; Martin , SJ ; Cotter , TG Hydrogen peroxide as a mediator of programmed cell death in the blastocyst Pierce , GB
electron leakage(passive) caused byelectron leakage
to increased solute leakagemight leadto increased solute leakage
to accumulation of high MDA , electrolyte leakage and reduction in NBT inhibition levels in cellsleadsto accumulation of high MDA , electrolyte leakage and reduction in NBT inhibition levels in cells
to lipid peroxidation and osmolytic leakageleadsto lipid peroxidation and osmolytic leakage
to rapid apoptosisleadsto rapid apoptosis
fluid leakage of ARDScausingfluid leakage of ARDS
leakage of cellular materialscausingleakage of cellular materials
leakage of intracellular materialcan causeleakage of intracellular material
in leakage of the cell membraneresultingin leakage of the cell membrane
in cell membrane leakageresulting ultimatelyin cell membrane leakage
in the activation of various UV response genes and consequent skin damageresultingin the activation of various UV response genes and consequent skin damage
to the apoptosis and necrosis of HeLa cellscontributedto the apoptosis and necrosis of HeLa cells
fлrex free radical production and further membrane damageleadsfлrex free radical production and further membrane damage
fluids leakage outside of cellscausesfluids leakage outside of cells
in leakage of ALT , ALP and bilirubinresultingin leakage of ALT , ALP and bilirubin
to leakage of toxic intracellular materialleadsto leakage of toxic intracellular material
Genest , P. C. , Setlow , B. , Melly , E. , Setlow , P. Killing of spores of Bacillus subtilis by peroxynitrite appears(passive) to be caused byGenest , P. C. , Setlow , B. , Melly , E. , Setlow , P. Killing of spores of Bacillus subtilis by peroxynitrite appears
cell cycle arrest in yeast [ 43can triggercell cycle arrest in yeast [ 43
to calcium influx [ 6leadingto calcium influx [ 6
in leakage of their contents into the cytosol and damage to cellsresultingin leakage of their contents into the cytosol and damage to cells
to leakage of intracellular components and denaturing of proteinsleadsto leakage of intracellular components and denaturing of proteins