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Smart Reasoning:

C&E

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Qaagi - Book of Why

Causes

Effects

free radicals during inflammation(passive) is caused bymembrane damage

free radicals.2(passive) caused bymembrane damage

free radicals ( 19(passive) caused bymembrane damage

free radicals Majority of studies(passive) caused bymembrane damage

free radicals and enhancing memory and cognitive function(passive) caused bymembrane damage

e.g. silica reacts with free radicals and other chemical groupsto causemembrane damage

In order to determine whether or notcan causemembrane damage

Both lipid peroxidation and oxidative stresscan causemembrane damage

ROS - induced lipid peroxidation(passive) caused byMembrane damage

oxidative stress [ 18(passive) caused bymembrane damage

Treatment by both compoundscausedmembrane damage

high lipid peroxidationcan causemembrane damage

ROS stress(passive) caused bymembrane damage

free radicals during general anesthesia presents as observed lipid peroxidation products.13 Studies have shown that a variety of medications used in anesthesia have effects on the oxidant - antioxidant system(passive) caused byMembrane damage

reactive oxygen species ( ROS ) formation with subsequent lipid peroxidationcausesmembrane damage

free radicals via lipid peroxidation23,24 and PI labelling of membrane - compromised S. mutans cells as observed via confocal imaging ( Fig . 2a(passive) caused bymembrane damage

carolacton treatment [ 5(passive) caused bythe membrane damage

pore - forming toxins during L. monocytogenes infection limiting cytotoxicity [ 38(passive) caused bymembrane damage

pore - forming toxins aerolysin and α - toxin [ 35(passive) caused bythe membrane damage

reactive oxygen species ( ROS ) during abiotic stress(passive) caused bythe membrane damage

lipid peroxidation in myocardial ischemia(passive) caused byMembrane damage

exacerbates membrane lipid peroxidationcausingmembrane damage

both DNA damage and lipid peroxidationcan causemembrane damage

lipid peroxidation in brain mitochondria(passive) caused bymembrane damage

the ability of Aβto causemembrane damage

i.e. , the abilityto causemembrane damage

the ability of BCto causemembrane damage

the ability of LLOMEto causemembrane damage

ROS.PMID(passive) caused bymembrane damage

over - accumulation of ROS(passive) caused bymembrane damage

the activity of reactive oxygen species(passive) caused bymembrane damage

The larger changes in pressurecould causedamage to the membrane

Reactive oxygen species ( ROS ) , occurring during dehydration and cryopreservationmay also causemembrane damage

Heat stresscan causemembrane damage

the lipopeptides(passive) caused bymembrane damage

pore - forming toxins ( 29(passive) caused bymembrane damage

NaCl stress(passive) caused bymembrane damage

reactive oxygen species and DNA degradation(passive) provoked byMembrane damage

to be an efficient producer of reactive oxygen speciesoften leadingto membrane damage

Its role ... excess consumption of saturated fats , alcohol , and drugs , among otherscausemembrane damage

necrosis and cell deathcausingnecrosis and cell death

directly to cell deathleadsdirectly to cell death

to high cell deathleadsto high cell death

to rapid cell deathleadsto rapid cell death

in apoptotic cell deathresultingin apoptotic cell death

to cell necrosis and premature cell death ( Hildebrand , 1998leadingto cell necrosis and premature cell death ( Hildebrand , 1998

to the leakage of mitochondrial nucleic acids ... immune responses and/or cell deathleadsto the leakage of mitochondrial nucleic acids ... immune responses and/or cell death

to cell death during HR response ( Lee et al . , 2009leadingto cell death during HR response ( Lee et al . , 2009

to bacterial cell death.97 , 98 , 99 Structures of compounds in phase - I clinical trialsleadingto bacterial cell death.97 , 98 , 99 Structures of compounds in phase - I clinical trials

to bacterial cell death.97 , 98 , 99Figure 7.Structures of compounds in phase - I clinical trialsleadingto bacterial cell death.97 , 98 , 99Figure 7.Structures of compounds in phase - I clinical trials

to a direct intracellular attack which induces cell death that can not be repaired during the post - treatment storage period ( Huang et al .leadingto a direct intracellular attack which induces cell death that can not be repaired during the post - treatment storage period ( Huang et al .

to cell death ... 33 ... even if they can also have positive implications due to the possible signalling nature of some of the species produced [ 34 - 36leadsto cell death ... 33 ... even if they can also have positive implications due to the possible signalling nature of some of the species produced [ 34 - 36

measurement of electrolyte leakage(passive) caused bymeasurement of electrolyte leakage

internal cell materials ( potassium ion , galaktosidase , low molecular solutes , amino acids , RMA , DNA ) leakage , which results in cell deathcausesinternal cell materials ( potassium ion , galaktosidase , low molecular solutes , amino acids , RMA , DNA ) leakage , which results in cell death

in cell lysis [ 49could resultin cell lysis [ 49

from electrolyte leakage Plasma membrane damageresultingfrom electrolyte leakage Plasma membrane damage

to leakage of essential cytoplasmic constituents ( 15leadingto leakage of essential cytoplasmic constituents ( 15

to the leakage of essential cytoplasmic constituents ( 30leadingto the leakage of essential cytoplasmic constituents ( 30

to the hypersensitive reaction in lettuce Bestwick , CS ; Bennett , MH ; Mansfield , JW Recognition and response in plant – fungus interactions Mansfield , JW Dose - dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli Lennon , SV ; Martin , SJ ; Cotter , TG Hydrogen peroxide as a mediator of programmed cell death in the blastocyst Pierce , GBleadingto the hypersensitive reaction in lettuce Bestwick , CS ; Bennett , MH ; Mansfield , JW Recognition and response in plant – fungus interactions Mansfield , JW Dose - dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli Lennon , SV ; Martin , SJ ; Cotter , TG Hydrogen peroxide as a mediator of programmed cell death in the blastocyst Pierce , GB

electron leakage(passive) caused byelectron leakage

to increased solute leakagemight leadto increased solute leakage

to accumulation of high MDA , electrolyte leakage and reduction in NBT inhibition levels in cellsleadsto accumulation of high MDA , electrolyte leakage and reduction in NBT inhibition levels in cells

to lipid peroxidation and osmolytic leakageleadsto lipid peroxidation and osmolytic leakage

to rapid apoptosisleadsto rapid apoptosis

fluid leakage of ARDScausingfluid leakage of ARDS

leakage of cellular materialscausingleakage of cellular materials

leakage of intracellular materialcan causeleakage of intracellular material

in leakage of the cell membraneresultingin leakage of the cell membrane

in cell membrane leakageresulting ultimatelyin cell membrane leakage

in the activation of various UV response genes and consequent skin damageresultingin the activation of various UV response genes and consequent skin damage

to the apoptosis and necrosis of HeLa cellscontributedto the apoptosis and necrosis of HeLa cells

fлrex free radical production and further membrane damageleadsfлrex free radical production and further membrane damage

fluids leakage outside of cellscausesfluids leakage outside of cells

in leakage of ALT , ALP and bilirubinresultingin leakage of ALT , ALP and bilirubin

to leakage of toxic intracellular materialleadsto leakage of toxic intracellular material

Genest , P. C. , Setlow , B. , Melly , E. , Setlow , P. Killing of spores of Bacillus subtilis by peroxynitrite appears(passive) to be caused byGenest , P. C. , Setlow , B. , Melly , E. , Setlow , P. Killing of spores of Bacillus subtilis by peroxynitrite appears

cell cycle arrest in yeast [ 43can triggercell cycle arrest in yeast [ 43

to calcium influx [ 6leadingto calcium influx [ 6

in leakage of their contents into the cytosol and damage to cellsresultingin leakage of their contents into the cytosol and damage to cells

to leakage of intracellular components and denaturing of proteinsleadsto leakage of intracellular components and denaturing of proteins

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Smart Reasoning:

C&E

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