by the activation and uncontrolled proliferation of T lymphocytes and well - differentiated macrophages(passive) to be caused by1 ] Macrophage activation syndrome
mutationcausesautoinflammation with recurrent macrophage activation syndrome
by excessive activation and proliferation of T lymphocytes and macrophages(passive) to be caused byMacrophage activation syndrome is thought
by excessive activation and proliferation of T lymphocytes and macrophages(passive) to be caused byMacrophage activation syndrome ( MAS
by the activation and uncontrolled proliferation of T lymphocytes and macrophages(passive) to be caused byMacrophage activation syndrome ( MAS
by the activation and uncontrolled proliferation of T lymphocytes and well - differentiated macrophages(passive) to be caused byMacrophage activation syndrome
by the immune disease irritating their mast cells so much(passive) caused bymast cell activation syndrome
the immune disease irritating their mast cells so much(passive) caused bymast cell activation syndrome
These inflammation phenotypescan leadto macrophage activation syndrome
These inflammation phenotypescan leadto macrophage activation syndrome Macrophage activation syndrome
Neurological symptoms associated with renal or liver impairment , fever , lymphadenopathy and disseminated intravascular coagulationshould promptconsideration of macrophage activation syndrome
by Lyme Disease(passive) caused byMast Cell Activation Syndrome
many thingscan setoff mast cell activation syndrome
the activation and uncontrolled proliferation of T lymphocytes and well - differentiated macrophages , leading to widespread hemophagocytosis and cytokine overproduction [ 1,2,6(passive) to be caused byMacrophage activation syndrome
Relatedto causeactivation syndrome
any drugcausingACTIVATION SYNDROME
ASPIRIN ... other drugscauseACTIVATION SYNDROME
ACETAMINOPHEN ... other drugscauseACTIVATION SYNDROME
a possibilitycould causeActivation Syndrome
Tryptophancan causeactivation syndrome
CYMBALTA ... other drugscauseACTIVATION SYNDROME
CYMBALTAcausingACTIVATION SYNDROME
you evaluatecausesACTIVATION SYNDROME
you evaluateACETAMINOPHEN causesACTIVATION SYNDROME
Disposition of antidepressantscan causeactivation syndrome
Some of the injury stimulicausingendothelial dysfunction / activation
Reactivation of the virus in immunocompromised patientscausesactivation of latent virus
Cns ... the processresultsin the cell activation syndrome
the two viral infectionsto causemonocyte / macrophage activation
from uncontrolled activation and proliferation of T cellsto resultfrom uncontrolled activation and proliferation of T cells
from uncontrolled activation and proliferation of T cells and excessive activation of macrophagesto resultfrom uncontrolled activation and proliferation of T cells and excessive activation of macrophages
to the death of more than half of the patientsmay have contributedto the death of more than half of the patients
in deathresultsin death
to ongoing health problemsleadingto ongoing health problems
severe allergic - like symptomscausessevere allergic - like symptoms
severe organ inflammationis causingsevere organ inflammation
specific symptoms(passive) caused byspecific symptoms
temporary symptomscan causetemporary symptoms
alsocan ... causealso
temporary symptoms in multiple organ systemscan causetemporary symptoms in multiple organ systems
allergic reactioncausesallergic reaction
histamine intolerancecauseshistamine intolerance
the release of histamine , a substance that causes chronic inflammationcausesthe release of histamine , a substance that causes chronic inflammation
the POTSis causingthe POTS
in pulmonary diseasecan resultin pulmonary disease
headachescan causeheadaches
any harmmay causeany harm
in animalsmay resultin animals
to inflammation and neuronal injury in a number of neurological disorders [ 2 , 3contributesto inflammation and neuronal injury in a number of neurological disorders [ 2 , 3
the graft(passive) caused bythe graft
in very high fatality in childrenresultsin very high fatality in children
the oxidative burst(passive) caused bythe oxidative burst
to removal of cellular debris and protein aggregates by phagocytosis and 2 ) ITGB3 , Galctin-3 and TGFB1leadsto removal of cellular debris and protein aggregates by phagocytosis and 2 ) ITGB3 , Galctin-3 and TGFB1
oftenleadsoften
to the release of proinflammatory cytokines , reactive oxygen species ( ROS ) , proteolytic enzymes involved in matrix degradation and thus in atherosclerotic plaque destabilizationleadsto the release of proinflammatory cytokines , reactive oxygen species ( ROS ) , proteolytic enzymes involved in matrix degradation and thus in atherosclerotic plaque destabilization
to the observed responsecontributeto the observed response
to the control of the disease and to the immunosuppression observed during the course of the infectioncontributesto the control of the disease and to the immunosuppression observed during the course of the infection
to the activation of stellate cellscontributesto the activation of stellate cells
to production of tumor necrosis factor ( TNF - leadingto production of tumor necrosis factor ( TNF -
in increased phagocytosisresultingin increased phagocytosis
to the pathogenesis of inflammatory diseases including atherosclerosiscontributes importantlyto the pathogenesis of inflammatory diseases including atherosclerosis
to sustained recruitment of CCR5leadsto sustained recruitment of CCR5
to inflammationleadsto inflammation
to a Cellleadsto a Cell
to production ofleadingto production of
in the release oresultingin the release o
to growth or differentiationleadingto growth or differentiation
to Ca++ entry into the cell triggering protein kinase C activationleadsto Ca++ entry into the cell triggering protein kinase C activation
injury to normal tissuescan causeinjury to normal tissues