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Smart Reasoning:

C&E

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Qaagi - Book of Why

Causes

Effects

I expression ( e.g. virally infected or neoplastic cells escaping conventional cytotoxic T cellsprovokesnatural killer cells

from existing pluripotent stem cells(passive) could be originatedNatural killer cells

many conditionscan causelow natural killer cell function

This proteintriggersnatural killer cells

to target and kill cancer and pathogenically infected cells as part of the innate immune system , the non - specific and immediate response to disease in the human body(passive) are designedNatural killer cells

researchers at University of California San Diego School of Medicinehave creatednatural killer cells

in bone marrow(passive) are createdNatural killer cells

Other immune cells including proinflammatory and anti - inflammatory / regulatory CD4 + T cells ( helper T lymphocytes ) , CD8 + T cells ( cytotoxic T lymphocytes ) , myeloid cell subsets , B cells , andcontributenatural killer cells

because of their ability to recognize and attack tumors(passive) were first discoveredNatural killer cells

soaking in the damaging rayscauseslow levels of Natural Killer cells

Shraga Segal Department of Microbiology and Immunology , Ben - Gurion University of the Negev , Beer Sheva 84105 , Israel ; National Institute for Biotechnology in the Negev , Ben - Gurion University of the Negev , Beer Sheva 84105 , Israel;Department of Life Sciences , Ben - Gurion University of the Negev , Beer Sheva 84105 , Israel ; Innate Immunity Group , German Cancer Research Center , Heidelberg 69120 , Germany ; and Shraga Segal Department of Microbiology and Immunology , Ben - Gurion University of the Negev , Beer Sheva 84105 , Israel;National Institute for Biotechnology in the Negev , Ben - Gurion University of the Negev , Beer Sheva 84105 , IsraelcontributeNatural killer cells

activation of monocytes / macrophages(passive) caused bynatural killer cells

this exchangecould preventnatural killer cells

the tumor microenvironment and we develop combinational strategies to enhance clinical efficacy in haematological and solid tumors(passive) are influenced byNatural Killer cells

These changespreventnatural killer cells

This processtriggersNatural Killer cells

Eva Klein , a renowned immunologist at the Karolinska Institutediscoverednatural killer cells

Inflammation of the endometriumcan causeNatural Killer cells

in the late 1960s(passive) were discoveredNatural killer cells

the taste of camphor with a drugpromptsnatural killer cells

Delivery , at the tumor site , of therapeutic amounts of cytokinescan triggernatural killer cells

Delivery ... cytokinescan triggernatural killer cells

female and male genestriggeringNatural Killer Cells

either autoimmune implantation disfunction , alloimmune implantation disfunction or both(passive) caused bynatural killer cells

Our body cells use Metylfolate for different purposes such as building and repairing DNA ,creatingnatural killer cells

a compound to helpto createNatural Killer Cells

the body ... in particular , the immune systemcreatesnatural killer cells

Sabrina Maria Calleja ... what treatment did you haveto preventnatural killer cells

nature in order to protect us from things like viral infections(passive) are designed byNatural killer cells

in the year 1970(passive) was discoveredNatural Killer cells

adrenaline(passive) caused bynatural killer cells

Interferon - γ production by tubulointerstitial human CD56(brightcontributesnatural killer cells

their cytotoxicity against tumor cellsinfluencedtheir cytotoxicity against tumor cells

to inhibition of cytotoxicityleadingto inhibition of cytotoxicity

to hepatocellular inflammationmay contributeto hepatocellular inflammation

from bone - marrow precursorsoriginatefrom bone - marrow precursors

to non - specific immunity through cell - mediated cytotoxicity ( CMCalso contributeto non - specific immunity through cell - mediated cytotoxicity ( CMC

apoptosis of infected cellscan causeapoptosis of infected cells

minor inflammation for a timecauseminor inflammation for a time

a process called inflammationcreatinga process called inflammation

cytotoxicity through binding of glycans on target cells including sialyl Lewis X antigen ( Higai 2009 ) peptide - selective NK cell receptor , andtriggeringcytotoxicity through binding of glycans on target cells including sialyl Lewis X antigen ( Higai 2009 ) peptide - selective NK cell receptor , and

to this endothelial cell dysfunction through the production of signaling proteins that affect the migration of the endothelial cells [ 50may contributeto this endothelial cell dysfunction through the production of signaling proteins that affect the migration of the endothelial cells [ 50

to pathophysiology of placenta leading to miscarriage in fetal and neonatal alloimmune thrombocytopeniacontributeto pathophysiology of placenta leading to miscarriage in fetal and neonatal alloimmune thrombocytopenia

to increased immune responseleadto increased immune response

the response that caused rejection of donor stem cells in bone marrow transplantationsledthe response that caused rejection of donor stem cells in bone marrow transplantations

to the lethality of a murine model of Escherichia coli infection Department of Surgerycontributeto the lethality of a murine model of Escherichia coli infection Department of Surgery

in producing proinflammatory mediators and secreting chemokine to Essay about The Validity Insanityresultin producing proinflammatory mediators and secreting chemokine to Essay about The Validity Insanity

in the production of cytokines.(30resultingin the production of cytokines.(30

to an exaggerated but ineffective immune responseleadingto an exaggerated but ineffective immune response

to a decrease in their cytotoxicity and cell killing abilityleadingto a decrease in their cytotoxicity and cell killing ability

to liver inflammation in chronic HCV infectioncontributeto liver inflammation in chronic HCV infection

recurrent miscarriagemight causerecurrent miscarriage

in bone marrow from CD34+Lin - CD45RA+CD10originatein bone marrow from CD34+Lin - CD45RA+CD10

in recurrent miscarriagesresultingin recurrent miscarriages

to kill off infected cells in the bodydesignedto kill off infected cells in the body

direct anterior - to - posterior spread of herpes simplex virus type 1 in the eyepreventdirect anterior - to - posterior spread of herpes simplex virus type 1 in the eye

to release of one or more enzymes or other granule components , such as esterases ( e.g. , serine esterases ) , perforin , and/or granzymes ) , inflammatory mediator release ( such as release of bioactive lipids such as leukotrienes ( e.g. , leukotriene C4 ) ) , and respiratory burstleadingto release of one or more enzymes or other granule components , such as esterases ( e.g. , serine esterases ) , perforin , and/or granzymes ) , inflammatory mediator release ( such as release of bioactive lipids such as leukotrienes ( e.g. , leukotriene C4 ) ) , and respiratory burst

in the phagocytosis and death of the tumor cells ( 75resultsin the phagocytosis and death of the tumor cells ( 75

with bone marrow - derived dendritic cells cultured in the presence of retinoic acidtriggeredwith bone marrow - derived dendritic cells cultured in the presence of retinoic acid

tumors and virus infected cells to self destructcausetumors and virus infected cells to self destruct

the actions of the more deadly T cells by setting In exploring how inflammation of the blood vessels occurstriggerthe actions of the more deadly T cells by setting In exploring how inflammation of the blood vessels occurs

CD28-mediated Foxp3 transcription in CD4+CD25- T lymphocytespreventCD28-mediated Foxp3 transcription in CD4+CD25- T lymphocytes

a miscarriage each time you get pregnantwill causea miscarriage each time you get pregnant

in the production of cytokines , including interleukins , tumor necrosis factor , and interferonresultingin the production of cytokines , including interleukins , tumor necrosis factor , and interferon

to mitochondrial dysfunction in response to placental ischemia in reduced uterine perfusion pressurecontributeto mitochondrial dysfunction in response to placental ischemia in reduced uterine perfusion pressure

in the production of cytokines , including interleukins , tumour necrosis ( cell death ) factor - á and interferonsresultingin the production of cytokines , including interleukins , tumour necrosis ( cell death ) factor - á and interferons

tumor cell apoptosis and inhibit tumor angiogenesistriggertumor cell apoptosis and inhibit tumor angiogenesis

infected cells to lyse PERFORIN cancer cells 7 Specific Immunity : Third Line of Defense Comes into play when non - specificcauseinfected cells to lyse PERFORIN cancer cells 7 Specific Immunity : Third Line of Defense Comes into play when non - specific

to immunodeficiency characterized by recurrent sinopulmonary and herpes simplex virus infections and poor responses to immunizationleadingto immunodeficiency characterized by recurrent sinopulmonary and herpes simplex virus infections and poor responses to immunization

failed implantation The theory goes that the problem for people like me is that there is an abnormally high concentration of natural killer cells , specifically in the uterus , which fail to get the memo about not attacking the implanting embryocausefailed implantation The theory goes that the problem for people like me is that there is an abnormally high concentration of natural killer cells , specifically in the uterus , which fail to get the memo about not attacking the implanting embryo

cell lysisthus preventingcell lysis

in automimmune conditions and degenerationcan resultin automimmune conditions and degeneration

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Smart Reasoning:

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