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the TH enzymeleadingto low mitochondrial ATP production
Any effective mutation in these subunitscausesloss of ATP production
equilibrium ... a few hoursresultedin the decline of ATP Production
that l - methyl-4-phenylpyridinium ( MPP+ ) , the active metabolite of MPTP , can block electron transport by binding to the same site as the classic Complex I inhibitor , rotenoneleadingto a loss of ATP production
cell mitochondrial metabolismleadingto less ATP production
cell mitochondrial metabolismleadingto less ATP production
that important cellular targets are several mitochondrial respiratory enzymesleadingto reduced ATP production
that acute hypoxia generated more ROS in complex III and increased mitochondrial oxygen consumptionleadingto less ATP production
And again , these mutations in your mitochondria iscausesless ATP production
fatty acid oxidationleadsto high ATP production
a decrease in the levels of glucose exertscausesa decrease in ATP production
mitochondrial failureresultingin poor production of ATP
abnormal mitochondriacausinglower ATP production
the mitochondrial defectsleadingto low ATP production
the accumulation of hydrogen ions along with lactate in the blood(passive) is prompted byThat drop in ATP production
the accumulation of hydrogen ions along with the lactate in the blood(passive) is prompted byThat drop in ATP production
Reduced cytochrome c oxidase activitymay resultin low ATP production
that oxidative stress damages the mitochondriaresultingin less ATP production
in turnwould ... resultin reduced ATP production
the level of module 3contributingto reduced ATP production
These mutations ... dysfunctional mitochondrial assemblyleadsto reduced ATP production
the oxidative phosphorylation ( OXPHOS ) pathway , ... elevated reactive oxygen species ( ROS ) levels , which cause oxidative stressresultingin low ATP production
Current levels above 1000 micro ampscausedATP production to decline
reduced the transcription of the mtDNAresultingin low ATP production
Mitochondrial alterationsleadingto reduced ATP production
the OXPHOS pathwayresultingin low ATP production
Reduced NAD+also causesreduced ATP production
to decline in older animalspresumably causingreduced ATP production
the increased cystine concentration in the lysosomeleadsto a lower ATP production
dysfunctional mitochondrial assemblyleadsto reduced ATP production
impair the oxidative phosphorylation ( OXPHOS ) pathwayresultingin low ATP production
Dysfunction of mitochondrial energy metabolism have been described following acute and chronic ethanol exposureleadingto reduced ATP production
impair the OXPHOS pathway ,resultingin low ATP production
The lack of oxygen as the last acceptor of electrons in the respiratory chaincausesa decrease in ATP production
The reduced Ca2 + concentration in the mitochondrialeadsto a decreased ATP production
This interaction was accompanied by a decrease of OSCP and loss of ATPase function ,leadingto reduced ATP production
Decreased ETCcausingdecreased ATP production
a reduction of these overexpressed enzymes in Phyllanthus - treated infected cellsmay leadto a low production of ATP
iron deficiencycausingdecreased ATP production
phosphates deposited in tissues(passive) is caused byLow ATP level
fatiguecan causefatigue
to seizuresto contributeto seizures
to cell depolarizationleadingto cell depolarization
to autoimmunity via an influence on the death of cellsmay leadto autoimmunity via an influence on the death of cells
to autoimmunitymay leadto autoimmunity
to the ability to train harder and recover fasteralso leadsto the ability to train harder and recover faster
our cells to be sickcauseour cells to be sick
cells to be sickcausecells to be sick
to or resulted from the development of infectioncontributedto or resulted from the development of infection
to muscle weakness and painmay contributeto muscle weakness and pain
howeverleadhowever
from insufficient delivery of nutrient and oxygenmight ... resultfrom insufficient delivery of nutrient and oxygen
the effect role of ATP and the ripple effect(passive) caused bythe effect role of ATP and the ripple effect
to exhaustionleadto exhaustion
ATP / AMPK signallingmay influenceATP / AMPK signalling
to the increased muscular activityleadsto the increased muscular activity
to higher adenosine levels , which ultimately may help with DNA demethylation , hence gene activationleadsto higher adenosine levels , which ultimately may help with DNA demethylation , hence gene activation
in the depolarization of neurons due to the failure of Na / K pumps of the cell membranesresultin the depolarization of neurons due to the failure of Na / K pumps of the cell membranes
to death of alveolarleadsto death of alveolar
to death of alveolarleadsto death of alveolar
The lower cell concentration(passive) was also caused byThe lower cell concentration
our cells to be sick , and decreases their ability to heal , regenerate , or function properlycauseour cells to be sick , and decreases their ability to heal , regenerate , or function properly
Protracted autophagy(passive) caused byProtracted autophagy
in slow growth and higher glucose uptake ratewould resultin slow growth and higher glucose uptake rate
to cellular dysfunctionleadingto cellular dysfunction
reduced glycogen synthesis which causes insulin resistancecausesreduced glycogen synthesis which causes insulin resistance
to cell growth inhibition and cell deathleadingto cell growth inhibition and cell death
in illnesses and diseaseresultingin illnesses and disease
immobilization and death of the wormcausesimmobilization and death of the worm
in slowing of muscle contractionresultingin slowing of muscle contraction
to an activation of a cellular energy sensor , AMPK , and subsequent downregulation of mTOR signaling pathway , which is associated with decreased cellular proliferationleadsto an activation of a cellular energy sensor , AMPK , and subsequent downregulation of mTOR signaling pathway , which is associated with decreased cellular proliferation
in illnessesresultingin illnesses
in apoptosis and tumor cell killingresultingin apoptosis and tumor cell killing
to perturbation of the Na+/K+-ATPaseleadsto perturbation of the Na+/K+-ATPase
membrane plasma depolarization , opening of calcium channels and disruption of potential mitochondrial membranescausesmembrane plasma depolarization , opening of calcium channels and disruption of potential mitochondrial membranes
to activate AMP - activated protein kinaseleadingto activate AMP - activated protein kinase
to cellular degenerationleadingto cellular degeneration
in neuron deathresultingin neuron death
to cellular organelle dysfunction and cellular death in high - metabolic end organs such as the heart , brain , and kidneyleadsto cellular organelle dysfunction and cellular death in high - metabolic end organs such as the heart , brain , and kidney
in ATP / ADPresultedin ATP / ADP