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the use of a too large pfncan provokeloss of reduc
Designedto preventloss of traction(Grip
deletion of the gene encoding the catalytic PRC2 subunit , set-7(passive) caused byLoss of H3K27me3
AB - Myocardial ischemia / reperfusion ( MI / Rcausesloss of cardiomyocytes
N2 - Myocardial ischemia / reperfusion ( MI / Rcausesloss of cardiomyocytes
I ) PvrRNAiexpression in differentiating cells ( Hml - gal4 UAS-2xEGFP UAS - PvrRNAicausesloss of progenitors
this oxidant stress paradigmcausesloss of ΔΨm
The changes at KEIGHLEYwill causeloss of PSB1
yet not so largeto causeloss of stereofusion
to avoid further damage to the muscle and/orto preventloss of life.[60
human organotypic retinal cultures ( HORCs ... knownto causeloss of RGCs
they are stored in conducive conditionsto preventloss of affectivity
a community for 9 yearsmessage the moderatorsMODERATORSilluminatedwaxKBPrinceORepairer of ReputationsLiruabout moderation team » discussions in r / webcomics<>X686 · 20 comments Side effectsmay causeloss of life105
an excessive frequency with which the user requires data(passive) caused byloss of synchronism
BarleyconditionsdomesticationEnvironmentalraterecombinationShapedvariation RNA viruses ... beyond whichcauseloss of infectivity
felling trees , demolition or changes to buildings(passive) caused byloss of roosts
error"AvoidingCatastropherecombination RNA viruses ... beyond whichcauseloss of infectivity
Avoid sample contact with any glass during the sampling proceduremay causeloss of analytes
the rotor to slip poles during disturbances such as load changes or faultscausingloss of synchronism
The frequency limiting unit 9 limits the frequency against any further increase of the rotating speedto preventloss of synchronism
cardioprotection against ischemia / reperfusion injury via FBXW7 / notch pathway — Augusta University Research Profiles Zixin Chen , Xuan Su , Yan Shen , Yue Jin , Tong Luo , Il - man Kim , Neal Lee Weintraub , Yao Liang Tang Myocardial ischemia / reperfusion ( MI / Rcausesloss of cardiomyocytes
the gain reductioncausingloss of audibility
A lack of permanent staffwill causeloss of effency
Late pharmacological inhibition of ATP4causesloss of MCCs
performancesto preventloss of synchronism
estimating the amount of generation - shedding required , early in the disturbance sequenceto preventloss of synchronism
A severe faultmay causeloss of synchronism
Both phenomena ... open - loopcausesloss of synchronism
needto preventloss of fitnessWhat
to Keep AppointmentscausingLoss of MoneyIn
the critical clearing angle ( cr ) to exceedcausingloss of synchronism
Optimizationcausedloss of accurracy
PBA treatmentpreventedloss of RGCs
deletion of Tim8pcausesloss of Tim13p
increased levels of free choline in the brain , which is consistent with a response to an infection resulting in increased breakdown of cell membraneswould causeloss of function.36,37,38
mechanical limitations(passive) caused byLoss of synchronism
a big impact on people and society in the immediate adverse results of medicine and practical consequences of altered neuroplasticity during the Mindwill causeloss of productiveness
triggering control actions early enoughto preventloss of synchronism
quickly producing sufficient decelerating energy to counteract accelerating energy gained during(passive) is prevented byLoss of synchronism
the step rate of a motorgradually to preventloss of synchronism
a myeloproliferative syndrome with splenomegaly and increased hematopoietic progenitors ( HPs ) that are hypersensitive to cytokinescausesa myeloproliferative syndrome with splenomegaly and increased hematopoietic progenitors ( HPs ) that are hypersensitive to cytokines
severe defects in integrin signalling , cell - ECM adhesion and cytoskeletal organization , resulting in early embryonic lethality ( kindlin-2causessevere defects in integrin signalling , cell - ECM adhesion and cytoskeletal organization , resulting in early embryonic lethality ( kindlin-2
to a dramatic loss of trigeminal neurons due to increased apoptosis ( Huang et al . , 1999bledto a dramatic loss of trigeminal neurons due to increased apoptosis ( Huang et al . , 1999b
to the accumulation of the protein p85leadsto the accumulation of the protein p85
in heart failure and eventually death of the patientsmight resultin heart failure and eventually death of the patients
proliferation of pancreatic islet cells , but not the adjacent exocrine cells that secrete proteins other than insulincan causeproliferation of pancreatic islet cells , but not the adjacent exocrine cells that secrete proteins other than insulin
to diminished heart contractility , scar formation and heart failureleadsto diminished heart contractility , scar formation and heart failure
the progress of heart failure and substantially extends the survival of CnA transgenic micepreventsthe progress of heart failure and substantially extends the survival of CnA transgenic mice
the progress of heart failure and substantially extends the survival of calcineurin transgenic micepreventsthe progress of heart failure and substantially extends the survival of calcineurin transgenic mice
increased apoptosis ( TUNEL staining )(passive) caused byincreased apoptosis ( TUNEL staining )
permanent damage of heart that progressively decreases its functionality and could eventually lead to heart failure and deathcausespermanent damage of heart that progressively decreases its functionality and could eventually lead to heart failure and death
a defect in embryonic programmed cell deathcausesa defect in embryonic programmed cell death
long term damage of heart that gradually decreases its features and could eventually lead to heart failure and deathcauseslong term damage of heart that gradually decreases its features and could eventually lead to heart failure and death
in a loss of JNK signaling and an abrogation of p75-mediated apoptosisresultedin a loss of JNK signaling and an abrogation of p75-mediated apoptosis
to a dramatic increase in apoptosis and severe loss of neurons in sensory ganglialeadsto a dramatic increase in apoptosis and severe loss of neurons in sensory ganglia
to the failure of endocrine cell development ... altered pancreatic structure and a markedly decreased number of cells expressing neurogenin 3 , indicating that menin is also required at this stage of the endocrine pancreas developmentleadsto the failure of endocrine cell development ... altered pancreatic structure and a markedly decreased number of cells expressing neurogenin 3 , indicating that menin is also required at this stage of the endocrine pancreas development
in a decrease of glucose - stimulated insulin secretion and in an increase of β - cell apoptosisalso resultsin a decrease of glucose - stimulated insulin secretion and in an increase of β - cell apoptosis
a mild cytokinesis defect that is made far worse by loss of E - cadherincausesa mild cytokinesis defect that is made far worse by loss of E - cadherin
a cell - intrinsic defect in TFH cell differentiation starting from early after infection ( A – Ecausesa cell - intrinsic defect in TFH cell differentiation starting from early after infection ( A – E
to the aberrant regulation of multiple E3 ubiquitin ligases , which causes the degradation of c - JUN protein through ubiquitin - proteasome system in satellite cellsleadsto the aberrant regulation of multiple E3 ubiquitin ligases , which causes the degradation of c - JUN protein through ubiquitin - proteasome system in satellite cells
the MEN1 syndrome in humans , which is an autosomal dominant disorder characterized by development of neuroendocrine tumors , glucose intolerance and type 2 diabetes.1 , 2 , 3causesthe MEN1 syndrome in humans , which is an autosomal dominant disorder characterized by development of neuroendocrine tumors , glucose intolerance and type 2 diabetes.1 , 2 , 3
lethality in larval stagescauseslethality in larval stages
retinal degeneration ... possibly because of impaired generation of the evaginations and/or impaired conversion of the evaginations to diskscausesretinal degeneration ... possibly because of impaired generation of the evaginations and/or impaired conversion of the evaginations to disks
to decreased cardiac performance that current pharmacological therapies can only modestly improveleadsto decreased cardiac performance that current pharmacological therapies can only modestly improve
to loss of cardiac massleadsto loss of cardiac mass
in severe retardation in development of the axon projections to the cochlea and the posterior vertical canalresultsin severe retardation in development of the axon projections to the cochlea and the posterior vertical canal
to adverse LV remodeling and dysfunction , which is accompanied by increased death and rates of ventricular rupture.16leadsto adverse LV remodeling and dysfunction , which is accompanied by increased death and rates of ventricular rupture.16
to midbrain - hindbrain boundary ( MHB ) defectsleadsto midbrain - hindbrain boundary ( MHB ) defects
to bone marrow failureleadsto bone marrow failure
a slow rate of defecation ( f ) decreased body length ( gcausesa slow rate of defecation ( f ) decreased body length ( g
a reduction in size and number of desmosomes ... resulting in ectodermal dysplasia / skin fragility syndrome.53causesa reduction in size and number of desmosomes ... resulting in ectodermal dysplasia / skin fragility syndrome.53
to a shortened palate and Meckelleadsto a shortened palate and Meckel
through deletion of deletion 's putative methyltransferase Ezl3pcausesthrough deletion of deletion 's putative methyltransferase Ezl3p
to decreased tellurite resistanceleadsto decreased tellurite resistance
to a reduction in erythrocyte lifespan as well as an enhanced mitotic arrest in intermediate ( Ter119+CD71 +ledto a reduction in erythrocyte lifespan as well as an enhanced mitotic arrest in intermediate ( Ter119+CD71 +
in a mislocalization of Chc1resultedin a mislocalization of Chc1
to impaired placental development and intrauterine growth restriction ( IUGR ) , a frequent complication of human pregnancy that limits the supply of oxygen and nutrients from the placenta to the developing fetus and is associated with a reduction of embryonic β cell growth and the onset of T2DM later in lifeleadsto impaired placental development and intrauterine growth restriction ( IUGR ) , a frequent complication of human pregnancy that limits the supply of oxygen and nutrients from the placenta to the developing fetus and is associated with a reduction of embryonic β cell growth and the onset of T2DM later in life
in decreased survivalresultedin decreased survival
a metaphase delay in embryos ( Steffensen et al . , 2001 ) and even more pronounced prometaphase delay ( data not showncausesa metaphase delay in embryos ( Steffensen et al . , 2001 ) and even more pronounced prometaphase delay ( data not shown
to a massive ER swelling and cell death in growth plate cells culminating in impaired bone formationledto a massive ER swelling and cell death in growth plate cells culminating in impaired bone formation