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Restarting connmancausesloss of 3G/4G/
black and oolong tea leaves ... dried or fermented leavescausesloss of EGCG
deterioration(passive) caused byloss of phthalonitrile
Late pharmacological inhibition of ATP4causesloss of MCCs
A Bapx1 knockoutcausesloss of the gonium
The ultraviolet ( UV ) radiation in sunlight has been foundto causeloss of infectivity
Myocardial infarction ( MIcausesloss of cardiomyocytes
by cell death mechanisms other than the apoptosis(passive) was caused byloss of cardiomyocytes
by cell death mechanisms other than the apoptosis(passive) was causedloss of cardiomyocytes
TAU deficiency was shownto causeloss of RGCs
Injection of glutamatecausedloss of RGCs
to breakdowncausingloss of plumpness
Fragrance ( and even peg-40 hcocausesloss of viscousity
in turncausesloss of synchronism
the generator ... in turncausesloss of synchronism
mechanical limitations(passive) caused byLoss of synchronism
Amyotrophic lateral sclerosiscausesloss of motorneurons
to determinecausesloss of IP3R.
The larger the voltage sag , the less real electrical power the generator can supply and the more unbalance ... in turncausesloss of synchronism
Another thing , I was not able to activate this keyboard with GUI and any change made therecausedloss of medhA.
Antibiotics , 10 mg /kg q4hcan causeloss of painkilling
power increasecausingloss of control.[12][13
that the parent or parents have kept information from or lied to themcausingloss of trust.[21
lied to themcausingloss of trust.[21
raising the minimum wagecausesloss of jobs?but
to diminished heart contractility , scar formation and heart failureleadsto diminished heart contractility , scar formation and heart failure
Noise hazards(passive) are caused byNoise hazards
Heart failure(passive) is caused byHeart failure
in heart failure and eventually death of the patientsmight resultin heart failure and eventually death of the patients
the majority ( 60 % ) of microtubules to enter a ' paused ' state in which they are neither growing nor shrinkingcausesthe majority ( 60 % ) of microtubules to enter a ' paused ' state in which they are neither growing nor shrinking
in caspase activation and caspase - dependent apoptosisresultedin caspase activation and caspase - dependent apoptosis
to excess hair cell development in the mouse organ of Cortileadsto excess hair cell development in the mouse organ of Corti
Friedreich 's ataxia , a debilitating disease characterized by loss of coordination ( ataxiacausesFriedreich 's ataxia , a debilitating disease characterized by loss of coordination ( ataxia
changes in the positive histone markcauseschanges in the positive histone mark
Friedreich 's ataxiacausesFriedreich 's ataxia
to apoptosisleadsto apoptosis
in micecausesin mice
changes in the positive histone mark H3K4me2 as well as the negative mark H3K27me3causeschanges in the positive histone mark H3K4me2 as well as the negative mark H3K27me3
3 proteolytic activityleads3 proteolytic activity
in DRONC activationshould resultin DRONC activation
to a decrease in the expression of synaptic vesicle protein SV2 in brainledto a decrease in the expression of synaptic vesicle protein SV2 in brain
albuminuriamay causealbuminuria
to defects in hematopoietic stem cells ... and these defects result from an overaccumulation of ROSleadsto defects in hematopoietic stem cells ... and these defects result from an overaccumulation of ROS
activation of multiple immune signaling pathwayscausesactivation of multiple immune signaling pathways
in aberrant accumulation of Smo in ciliaresultingin aberrant accumulation of Smo in cilia
lethality in larval stagescauseslethality in larval stages
to shortened lifespan , locomotor defects ... and increased susceptibility to stressledto shortened lifespan , locomotor defects ... and increased susceptibility to stress
in a mislocalization of Chc1resultedin a mislocalization of Chc1
in a severe loss of NBsresultsin a severe loss of NBs
to reduced assembly of the dynein-2 holocomplex and reduced interactions with IFT particlesleadsto reduced assembly of the dynein-2 holocomplex and reduced interactions with IFT particles
in accumulation of FM4 - 64 puncta that colocalize with a late endosomal marker ( GFP - Pep12resultedin accumulation of FM4 - 64 puncta that colocalize with a late endosomal marker ( GFP - Pep12
a reduction in the amount of DHC2 / DYNC2H1 and TCTEX1 / DYNLT1 associated with HA - WDR34 ( Figure 9B and Ccauseda reduction in the amount of DHC2 / DYNC2H1 and TCTEX1 / DYNLT1 associated with HA - WDR34 ( Figure 9B and C
gain of function of oncogenes ( KCNMA1 , LOX ) , VEGFA and SEMA6A respectively and increased levels of miR-142 - 3p , miR-185 , mir-34a , miR-224causesgain of function of oncogenes ( KCNMA1 , LOX ) , VEGFA and SEMA6A respectively and increased levels of miR-142 - 3p , miR-185 , mir-34a , miR-224