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propofol infusionresultingin lipid emulsion
of 30 % soybean oil and 15 % fish oil(passive) is composedThe SMOF lipid emulsion
of EPC , DCP , caprylate and caprate triglyceride(passive) was composedLipid emulsion
to be infused slowly as part of a total parenteral nutrition regimen(passive) is designedLipid emulsion
to considerwhen designinglipid emulsion
The administration of parenteral nutrition containing soybean oil – based and olive oilresultedbased lipid emulsion
the formulation of base of the common anesthetic , propofol(passive) was discoveredLipid emulsion
Imminent cardiac arrest in caffeine toxicityshould promptintravenous lipid emulsion
of the machine or installation it should be available with scientists involved(passive) is composedThe lipid emulsion
of SMOFlipid(passive) is composedThe lipid emulsion of SmofKabiven
a lipid phase that extracts the lipid - soluble bupivacaine molecules from the aqueous plasma phase ( and therefore out of the tissue ) , or alternatively , the lipid infusion diffuses directly into tissue and interacts with bupivacaine at that level.2createsa lipid phase that extracts the lipid - soluble bupivacaine molecules from the aqueous plasma phase ( and therefore out of the tissue ) , or alternatively , the lipid infusion diffuses directly into tissue and interacts with bupivacaine at that level.2
of 80 % olive oil and 20 % soybean oilcomposedof 80 % olive oil and 20 % soybean oil
of olive oil ( 80 % ) and soybean oil ( 20 %composedof olive oil ( 80 % ) and soybean oil ( 20 %
of 30 % soybean oil , 30 % MCT , 25 % olive oil and 15 %composedof 30 % soybean oil , 30 % MCT , 25 % olive oil and 15 %
in significant dye acquisition by the lipid compartment accompanied by a reduction in the colour intensity of the aqueous phase that could be readily observedresultedin significant dye acquisition by the lipid compartment accompanied by a reduction in the colour intensity of the aqueous phase that could be readily observed
cardiovascular toxicity but also relieve nervous system toxicitycan preventcardiovascular toxicity but also relieve nervous system toxicity
of a bunch of tiny oil - bubbles bordered by an emulsifiercomposedof a bunch of tiny oil - bubbles bordered by an emulsifier
possiblyinfluencepossibly
Essential fatty acid deficiency(passive) prevented byEssential fatty acid deficiency
a separate lipid compartment in the plasma , which draws the lipophilic drug out of the tissue. createsa separate lipid compartment in the plasma , which draws the lipophilic drug out of the tissue. 
amlodipine concentrations(passive) caused byamlodipine concentrations
of soybean oil ( long - chain triglycerides , LCTcomposedof soybean oil ( long - chain triglycerides , LCT
of linoleate ( 18:2primarily composedof linoleate ( 18:2
to maximize the ratio of omega 3 to omega 6 fatty acidshas been designedto maximize the ratio of omega 3 to omega 6 fatty acids
to enhanced redistribution of local anesthetics or enhanced delivery of local anesthetics into the liver , which has been described as a lipid shuttle or subway [ 36 - 43can contributeto enhanced redistribution of local anesthetics or enhanced delivery of local anesthetics into the liver , which has been described as a lipid shuttle or subway [ 36 - 43
the reduction in amlodipine concentration(passive) caused bythe reduction in amlodipine concentration
only minimallyinfluencesonly minimally
from either techniqueresultingfrom either technique
metabolic pathways(passive) influenced bymetabolic pathways
of long chain fatty acidscomposedof long chain fatty acids
a “ lipid sink ” that sequesters lipophilic drugs from their sites of action and decreases the amount of free drug that is available ( 1 - 4createsa “ lipid sink ” that sequesters lipophilic drugs from their sites of action and decreases the amount of free drug that is available ( 1 - 4
hyperacidity or inappropriate amount of bivalent cations ... Ca 2 + and Mg 2 +can causehyperacidity or inappropriate amount of bivalent cations ... Ca 2 + and Mg 2 +
in similar rates of infectious and noninfectious complications and no differences in glycemic control , inflammatory and oxidative stress markers , and immune function in critically ill adultsresultedin similar rates of infectious and noninfectious complications and no differences in glycemic control , inflammatory and oxidative stress markers , and immune function in critically ill adults
of droplets with a mean ( range ) size of 0.19 ( 0.15–0.3composedof droplets with a mean ( range ) size of 0.19 ( 0.15–0.3
to an increased inactivation of the Na+ channels ... sensitizing INa for mepivacaineledto an increased inactivation of the Na+ channels ... sensitizing INa for mepivacaine
the toxic drugs to pass into the plasma , where they then pass to the lipid phase [ 9,10leadsthe toxic drugs to pass into the plasma , where they then pass to the lipid phase [ 9,10
an expanded lipid phase , resulting in redistributioncreatesan expanded lipid phase , resulting in redistribution
in increased adenosine triphosphate ( ATP ) productioncan also resultin increased adenosine triphosphate ( ATP ) production
in the shuttling of local anesthetic away from the thalamocortical neuronsresultsin the shuttling of local anesthetic away from the thalamocortical neurons
to skeletal muscle damage in clinical settingscontributesto skeletal muscle damage in clinical settings
out in claim 1setout in claim 1
local anesthetic toxicity(passive) caused bylocal anesthetic toxicity
significant hemodynamic changes with hypertension , particularly in the pulmonary circulation and increase in vascular resistance , which is a factor to consider prior to use of these solutionscausesignificant hemodynamic changes with hypertension , particularly in the pulmonary circulation and increase in vascular resistance , which is a factor to consider prior to use of these solutions
of polystyrene cross - linked with divinyl bcnzcneis composedof polystyrene cross - linked with divinyl bcnzcne
from such additionsmay resultfrom such additions
further hemodynamic instability in the high doses necessarywill causefurther hemodynamic instability in the high doses necessary
phosphorylation of Aktwill causephosphorylation of Akt
long - term neurological damage from chemical agentscan preventlong - term neurological damage from chemical agents
to pulmonary infections or septicaemiacan ledto pulmonary infections or septicaemia
six papers totaling 139 pages that cover specific topics such as adverse events and laboratory interference(passive) caused bysix papers totaling 139 pages that cover specific topics such as adverse events and laboratory interference