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Infants up to 2 months of age- Use is not recommendedmay causekernicterus in neonates
the typical signs and symptoms of mucositis or treatment may helpto preventthrombocytopenia in neonates
Except as concurrent adjunctive therapy with pyrimethamine in the treatment of congenital toxoplasmosis or for the prophylaxis of Pneumocystis carinii pneumonia ( PCP ) in infants 4 weeks of age or over , the use of sulfamethoxazole and trimethoprim combination is contraindicated in infants up to 2 months of agemay causekernicterus in neonates
Pregnancy Category C , antifolate effectsmay causekernicterus in neonates
Zika fever and infection during pregnancy is hypothesizedto causemicocephaly in neonates
is contraindicated in infants up to 2 months of age for most indicationsmay causekernicterus in neonates
sulfonamidesmay causekernicterus in neonates
excessive concentration of bilirubin in the blood ,causingjoundice in neonates
Carnitine supplementation has been shownto preventsteatosis in neonates
an essential amino acidto preventcholestasis in neonates
as in adult(passive) can be causedThrombocytopenia in neonates
genetic TLR variantsinfluenceTregs in neonates
oftenresultsoften
sequels(passive) caused bysequels
kernicterus , porphyria – abnormalities in the chemical steps that lead to heme productionmay causekernicterus , porphyria – abnormalities in the chemical steps that lead to heme production
kernicterus ( central nervous system disorders caused by elevated bilirubinmay causekernicterus ( central nervous system disorders caused by elevated bilirubin
to retrolental fibroplasia manifested by damaged retinal blood vessels , varying degrees of scarring , and permanent visual impairmentcan leadto retrolental fibroplasia manifested by damaged retinal blood vessels , varying degrees of scarring , and permanent visual impairment
CNS infections(passive) caused byCNS infections
malabsorption and decreased expression of intestinal and pancreatic genes mediating food digestion and uptake throughcausesmalabsorption and decreased expression of intestinal and pancreatic genes mediating food digestion and uptake through
extensive skin necrosis(passive) caused byextensive skin necrosis
treatment of CLABSI(passive) caused bytreatment of CLABSI