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that disseminated prostate cancer cells compete for occupancy of the osteoblastic HSC niche with HSCsto createmetastatic foci
Another possible mechanism for neuronal damage ... vasogenic edemamay originatefrom ischemic foci
transcortical and medullary arteriescausinginfarction foci
irritation(passive) caused byneoplastic foci
radiation - induced DSBs(passive) caused by53BP1 foci
of < 20 cells ( three times larger than lymphocytes(passive) were composedMetastatic foci
the deposition of a peculiar substance(passive) were caused bymiliary foci
of an inner layer of epithelial cells surrounded by myoepithelial cells with ample and clear cytoplasm(passive) are composedHyperplastic foci
off - target DSBs(passive) created by53BP1 foci
RecA overproduction in ΔrecQ cells(passive) caused byRDG foci
to excess antisense peptides , mislocalization of TDP-43 , and neurodegenerationleadsto excess antisense peptides , mislocalization of TDP-43 , and neurodegeneration
mild mass effect and midline - shift in keeping with a haemorrhagic infarction occupying almost completely the right frontal lobecausingmild mass effect and midline - shift in keeping with a haemorrhagic infarction occupying almost completely the right frontal lobe
from myocardial fiber fragmentationresultingfrom myocardial fiber fragmentation
of bone marrow tissuecomposedof bone marrow tissue
from carotid stent treatmentresultingfrom carotid stent treatment
from expression of the enhanced green fluorescent protein by the reconstituted virusesresultingfrom expression of the enhanced green fluorescent protein by the reconstituted viruses
of a considerable number of monomersare ... composedof a considerable number of monomers
to persistent DNA damageleadingto persistent DNA damage
to an overall reduction in average foci number close to the background level of 1.8 ± 0.5 and 2.9 ± 0.5 respectively for X - rays and protonsleadingto an overall reduction in average foci number close to the background level of 1.8 ± 0.5 and 2.9 ± 0.5 respectively for X - rays and protons