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The role of adenosine receptors in preconditioning is confirmed by the observation that adenosine A1‐receptor antagonists can block KATP channelsthereby preventingischaemic preconditioning
choices in the treatment of seams(passive) can be substantially influenced bypreconditioning
pharmacologic agents , including anesthetics(passive) can also be triggered byPreconditioning
carbon monoxide(passive) triggered byPreconditioning
bamiphylline , glibenclamide , and naloxone ( a non - selective opioid receptor antagonistpreventit.7,28–31 Ischaemic preconditioning
the release of metabolites eliciting the metaboreflexmay ... triggerpreconditioning
temporary occlusions(passive) is caused byIschaemic preconditioning
Inhibition of trafficking of KATP channel subunitspreventedpreconditioning
The effect of additional β - blocker treatment ... as these were previously shownto negatively influencepreconditioning
Adenosine receptor blockade ’s abilityto preventpreconditioning
Natural powderto preventpreconditioning
Nitric oxide as a mediator of delayed pharmacological ( A(1 ) receptortriggeredpreconditioning
isoflurane - induced mitochondrial ROS production in the aged myocardium ... sufficientto triggerpreconditioning
adenosine receptor activation(passive) caused bypreconditioning
that an A2a antagonist enhances the ability of an A3 / A1 agonist , MRS 580to causepreconditioning
endogenous cannibinoidsmay causeischaemic preconditioning
the ability of MRS1528to causepreconditioning
to reduce mortality and decrease sickness in calves(passive) is designedPreconditioning
the surface receptors ableto triggerpreconditioning
that all Gi - coupled receptors use the same signal transduction pathwaysto triggerpreconditioning
the differentiation induced by ischaemia / reperfusion injury of rat cardiac fibroblasts into myofibroblasts - ResearchOnline@JCU A to Z | @JCU | Current students | Staff |preventsthe differentiation induced by ischaemia / reperfusion injury of rat cardiac fibroblasts into myofibroblasts - ResearchOnline@JCU A to Z | @JCU | Current students | Staff |
endothelium - dependent vasomotor dysfunction(passive) can be prevented byendothelium - dependent vasomotor dysfunction
pronounced suppression of gene expression in response to ischaemia that is ordinarily injuriouscausedpronounced suppression of gene expression in response to ischaemia that is ordinarily injurious
lethal ischemic spinal cord injury in a swine modelto preventlethal ischemic spinal cord injury in a swine model
the liver inflammatory response to lung ischaemia / reperfusion in a swine lung autotransplant modelpreventsthe liver inflammatory response to lung ischaemia / reperfusion in a swine lung autotransplant model
myocardial damage during aortic aneurysm surgerypreventingmyocardial damage during aortic aneurysm surgery
functional vascular abnormalities.15 Clinical observations confirmed a reduced coronary flow reserve after ischaemia and reperfusioncan preventfunctional vascular abnormalities.15 Clinical observations confirmed a reduced coronary flow reserve after ischaemia and reperfusion
to a decrease in myocardial infarct sizeleadingto a decrease in myocardial infarct size
a complex multifactorial molecular process 20probably triggersa complex multifactorial molecular process 20
the increase in [ 3H]PtdIns(4,5)P2 in early reperfusionpreventedthe increase in [ 3H]PtdIns(4,5)P2 in early reperfusion
Ins(1,4,5)P3 responses in early reperfusionpreventedIns(1,4,5)P3 responses in early reperfusion
a significant improvement in several parameters of function during reperfusion in hearts from young control rats ( Table 4causeda significant improvement in several parameters of function during reperfusion in hearts from young control rats ( Table 4
apparentlyleadsapparently
in significant protection in Akt1+/+ mice against I - R injury ( 28.9 ± 1.4 % with IPC versus 45.5 ± 2.6 % in control , n = 6 , P = 0.0001resultedin significant protection in Akt1+/+ mice against I - R injury ( 28.9 ± 1.4 % with IPC versus 45.5 ± 2.6 % in control , n = 6 , P = 0.0001
about ~3-fold higher rate of exosome production from HUVEC and from isolatedcausedabout ~3-fold higher rate of exosome production from HUVEC and from isolated
Ins(1,4,5)P3 generationpreventedIns(1,4,5)P3 generation
iNOS expression in heartcausesiNOS expression in heart
no significant change in the effective refractory period ( ERP ) , VFT or DFT over the 90 min of the experimentscausedno significant change in the effective refractory period ( ERP ) , VFT or DFT over the 90 min of the experiments