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non - homologous chromosome may reattach back to the original chromosome , but in reverse orientationcausingchromosomal inversion
major factorscontributingto chromosomal inversion [ 43]–[47
inreverse orientationcausingchromosomal inversion
Schematic pathway for intramolecular replicative transposition of IS26causinga chromosomal inversion
Gamma Irradiation Contributes to Downregulation of a WBC11 Homolog in Bloomless Sorghum(passive) Caused byGenomic Inversion
a fusion generesultingfrom chromosome inversion
The bovine Xp painting probe identified an interstitially located homologous segment in the sheep and goat Xq regionmost probably resultingfrom chromosome inversion
authors Darius Juskeviciusresultingfrom chromosomal translocation / inversion
IS6100 ( 28 ) , which belongs to the same family as IS26 , and may also account for the formation of Tn6029 ( 29(passive) caused bya chromosomal inversion
likely replication origin ( dnaA ) and terminus ( dif ) and highlight a replichore imbalance(passive) caused bylikely replication origin ( dnaA ) and terminus ( dif ) and highlight a replichore imbalance
the long stretch of linkage disequilibrium(passive) caused bythe long stretch of linkage disequilibrium
in C3H / HeJ mice JAX Notes 491:15discoveredin C3H / HeJ mice JAX Notes 491:15
from a broken chromosome that was put back together incorrectlyresultingfrom a broken chromosome that was put back together incorrectly
with a lack of recombination between the original and inverted variantresultingwith a lack of recombination between the original and inverted variant
to the fusion of the anaplastic lymphoma kinase ( ALK ) gene with the echinoderm microtubule - associated protein - likeleadingto the fusion of the anaplastic lymphoma kinase ( ALK ) gene with the echinoderm microtubule - associated protein - like
to dominant expression of a second S layer proteinleadingto dominant expression of a second S layer protein
almost 50 % of the cases of severe Factor VIII deficiencycausesalmost 50 % of the cases of severe Factor VIII deficiency
in a disruption of a gene , which may contribute to the syndromeresultsin a disruption of a gene , which may contribute to the syndrome
Shh relocationcausesShh relocation
severe hemophilia A. NAHR between two homologous regions , one in intron 1 of the F8 gene ( here named homolog 1 ) and the other located 140 kbp upstream ( homolog 2causessevere hemophilia A. NAHR between two homologous regions , one in intron 1 of the F8 gene ( here named homolog 1 ) and the other located 140 kbp upstream ( homolog 2
to cancerledto cancer
The EML4-ALK gene fusion(passive) is caused byThe EML4-ALK gene fusion
in a similarly truncated human HMGA2 generesultsin a similarly truncated human HMGA2 gene
to dominant expression of SlpB.leadingto dominant expression of SlpB.
A dominant Antp mutation(passive) caused byA dominant Antp mutation
just this type of agent in B - RAFcreatesjust this type of agent in B - RAF
in gene silencing in some cells ( white ) due to heterochromatin spreading over the w gene in a stochastic process that allows expression to be maintained in other cells ( redresultsin gene silencing in some cells ( white ) due to heterochromatin spreading over the w gene in a stochastic process that allows expression to be maintained in other cells ( red
these two genotypescausedthese two genotypes
Shh to be expressed ectopically in the phalangescausesShh to be expressed ectopically in the phalanges
in suppression of the senseresultingin suppression of the sense
to adaptation and ... in turn , reproductive isolationcontributesto adaptation and ... in turn , reproductive isolation
to loss of the entire E(splleadingto loss of the entire E(spl
the Trsp1 gene(passive) caused bythe Trsp1 gene
embryogenesis(passive) caused byembryogenesis