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the accumulation of cells and lipids ( Fig(passive) caused byintimal thickening
proliferation of smooth muscle cells(passive) caused byIntimal thickening
0047]Vascular injurycausingintimal thickening
the precise mechanism by whichpreventsintimal thickening
poor runoff conditions in the canine autologous vein grafts(passive) caused byintimal thickening
balloon injury of the carotid artery(passive) caused byintimal thickening
overstretching of arteries during balloon angioplasty(passive) triggered byintimal thickening
the prevention and treatment of vascular injurycausingintimal thickening
Combined Therapy Vascular injurycausingintimal thickening
687 - 694 Anipamilpreventsintimal thickening
the local underlying biomechanical stimulimay influenceintimal thickening
a proliferation of elongated and concentrically arranged smooth muscle cells with deposition of collagen(passive) caused byintimal thickening
Prevention of the upregulation of this transcription factor , observed in carotid artery from rats subjected to balloon injurymight preventintimal thickening
the accumulation of cells , infiltration of inflammatory cells , lipids and connective tissues that can lead to cardiac and cerebral infarction ( such as heart attack and stroke(passive) caused byintimal thickening
Next , the smooth muscle cells in intima proliferate accompanied with deposition on the substratethereby causingintimal thickening
Smooth muscle cells ( SMC ) migrate to the intimacausingintimal thickening
migration of vascular smooth muscle cells ( VSMCs ) from the media(passive) is caused byIntimal thickening
oedema and fibrocellular proliferation(passive) caused byintimal thickening
the graft vascular bedcausescircumferential intimal thickening
leaky and vascular smooth muscle cells ... ECM proteinscauseintimal thickening
accumulation of smooth muscle - like cells and a loose proteoglycan rich stroma focal calcification usually within residual tubular segments extensive deposition of calcium oxalate crstals in tubules and interstitium(passive) caused byarteial intimal thickening
an inflammatory response ( ie , arteritiscausesendothelial thickening
an accumulation of VSMCs and macrophages(passive) caused byIntimal thickening
hyperproliferation as well as migrate to the intimal(passive) caused byintimal thickening
a combination of cellular proliferation and extracellular matrix accumulation(passive) is created byIntimal thickening
proliferation of alpha - smooth muscle actin - positive cells(passive) caused byintimal thickening
proliferation of α - smooth muscle actin - positive cells(passive) caused byintimal thickening
edema , mucinous degeneration , and increase in cells , collagen , and elastica(passive) caused bynodular intimal thickening
NO generated from iNOSmay preventintimal thickening
This treatment peeled off the endothelium of blood vessels and damaged tunica media of smooth muscleto createintimal thickening
accumulation of smooth muscle cells ( SMCs ) both from donor and recipient(passive) caused byintimal thickening
Nonatheromatous arteriosclerosiscausesintimal thickening
endothelial cell injuriescan causeintimal thickening
spindle shaped cells arranged(passive) caused byIntimal thickening
the prevention and treatment of injurycausingintimal thickening
Intimal hyperplasiacausesAVF intimal thickening
Blood flow obstruction ( by the presence of worms ) andcan leadendothelial thickening
embolism of tumor cells into pulmonary vasculature(passive) caused byfibrocellular intimal thickening
also(passive) can ... be triggeredIntimal thickening
A variety of stent coatings and compositions have been proposed for the prevention and treatment of , for examplecausingintimal thickening
to 20 % stenosisleadingto 20 % stenosis
in early graft stenosis and occlusionresultingin early graft stenosis and occlusion
to the development of stenosis 9leadingto the development of stenosis 9
to luminal narrowing and vessel occlusioncan leadto luminal narrowing and vessel occlusion
of proliferative smooth muscle cells , matrix , and other cellularcomposedof proliferative smooth muscle cells , matrix , and other cellular
of vascular smooth muscle cells and extracellular matrixwas ... composedof vascular smooth muscle cells and extracellular matrix
to graft ischemiacan leadto graft ischemia
to proliferative restenosismay leadto proliferative restenosis
from accumulation of vascular smooth muscle cells and deposition of the extracellular matrixoriginatesfrom accumulation of vascular smooth muscle cells and deposition of the extracellular matrix
mild stenosis in external iliac artery ( ccausingmild stenosis in external iliac artery ( c
in moderate up to subtotal stenosis of the lumen ( Figure 2Aresultingin moderate up to subtotal stenosis of the lumen ( Figure 2A
to the restenosis of the vein graft ... 7 , 8may leadto the restenosis of the vein graft ... 7 , 8
to restenosis.34 Howeverleadsto restenosis.34 However
towards arterial lumen occlusion , a process called negative remodellingpossibly leadstowards arterial lumen occlusion , a process called negative remodelling
A review Seung - Kee Min MD a , Richard D. Kenagy PhD b and Alexander W. Clowes MD b , , a b intimal thickeningendothelial injury and inflammationsmooth muscle cell hyperplasia , extracellular matrix accumulation After vascular reconstruction , luminal narrowing is in part(passive) caused byA review Seung - Kee Min MD a , Richard D. Kenagy PhD b and Alexander W. Clowes MD b , , a b intimal thickeningendothelial injury and inflammationsmooth muscle cell hyperplasia , extracellular matrix accumulation After vascular reconstruction , luminal narrowing is in part
mostly from the accumulation of VSMCs within the intimaresultingmostly from the accumulation of VSMCs within the intima
from Subnormal Wall Shear Stress Chronic kamagra jel izmir in blood flowResultingfrom Subnormal Wall Shear Stress Chronic kamagra jel izmir in blood flow
from an accumulation of lipidsresultingfrom an accumulation of lipids
to loss of luminal caliber , which decreases vascular complianceleadsto loss of luminal caliber , which decreases vascular compliance
to endothelial injury of intracranial veinsleadingto endothelial injury of intracranial veins
an increasing of pulmonary arterial pressurecausesan increasing of pulmonary arterial pressure
from infiltration of inflammatory cellsresultingfrom infiltration of inflammatory cells
the constriction(passive) caused bythe constriction
of macrophages distended by lipid droplets ( known as foam cells ) , lymphocytes , and smooth muscle cellscomposedof macrophages distended by lipid droplets ( known as foam cells ) , lymphocytes , and smooth muscle cells
from Subnormal Wall Shear Stress Chronic reduction in blood flow volume and blood flow velocityResultingfrom Subnormal Wall Shear Stress Chronic reduction in blood flow volume and blood flow velocity
from mechanical influences on the vessel wallresultingfrom mechanical influences on the vessel wall
to platelets aggregation ... and finally results in the formation of thrombus in the vessel [ 24 - 26leadsto platelets aggregation ... and finally results in the formation of thrombus in the vessel [ 24 - 26
a narrowed lumen(passive) caused bya narrowed lumen
to vessel obliterationleadingto vessel obliteration
in changes of ECM contents ( e.g. , fibrin vs. collagen ) and the functions of the recruited vascular cells ... thus interfering with the synthesis and assembly of elastinresultedin changes of ECM contents ( e.g. , fibrin vs. collagen ) and the functions of the recruited vascular cells ... thus interfering with the synthesis and assembly of elastin
of smooth muscle cells [ 5composedof smooth muscle cells [ 5
from the cholesterol supplemental dietresultingfrom the cholesterol supplemental diet
of T cells , macrophages , and smooth muscle cellscomposedof T cells , macrophages , and smooth muscle cells
of SMCscomposedof SMCs
of collagen , elastin , and smooth musclecomposedof collagen , elastin , and smooth muscle
of variable combinations of fibrous and fatty materialcomposedof variable combinations of fibrous and fatty material
to the tensile support.6may ... contributeto the tensile support.6
from loose fibrous remodelingresultingfrom loose fibrous remodeling
from smooth muscle cell migration and proliferationresultingfrom smooth muscle cell migration and proliferation
from localized proliferation of intimal fibroblasts caused by growth factors released by hemodynamic stressesmay resultfrom localized proliferation of intimal fibroblasts caused by growth factors released by hemodynamic stresses