Lipofectin , integrin - targeting peptides , and plasmid DNA 2006 Application to vascular adventitia of a nonviral vector for TIMP-1 gene therapyto preventintimal hyperplasia
mesenchymal stem cells after vascular injury Kinnaird et al(passive) contributed byintimal hyperplasia
Meng , Q. H. , Jamal , W. , Hart , S. L. & McEwan , J. R. Application to vascular adventitia of a nonviral vector for TIMP-1 gene therapyto preventintimal hyperplasia
Mesenchymal Stem Cells After Vascular Injury | Arteriosclerosis , Thrombosis , and Vascular Biology(passive) Contributed byAttenuate Intimal Hyperplasia
vascular inflammation such as atherosclerosis(passive) caused byintimal hyperplasia
Nonembryonic - Non - Human 2011 NHLBI 1R01HL106018 - 01 Site - specific therapiesto preventintimal hyperplasia CHAIKOF
to deliver therapeutic drugsto preventintimal hyperplasia
Eric Ducasse ; Jean - Marc Cosset ; Jocelyne Mazurier ; François Eschwege ; Paul Puppinck ; Eric Lartigau [ High - dose external ionizing radiationpreventsintimal hyperplasia
MSCs after vascular injury(passive) contributed byintimal hyperplasia
92(6):1570 - 1575 , Sep. 15 , 1995.5Liermann et al . , Prophylactic Endovascular Radiotherapyto PreventIntimal Hyperplasia
92(6):1570 - 1575 , Sep. 15 , 1995.6Liermann et al . , Prophylactic Endovascular Radiotherapyto PreventIntimal Hyperplasia
surgical preparative injury in grafts experiencing normal arterial hemodynamics(passive) triggered byintimal hyperplasia
chronic mechanical stimulation from stent(passive) caused bythe intimal hyperplasia
to delay PDT after balloon injuryto preventintimal hyperplasia
the activation of vascular smooth muscle cells and inflammation due to arterial stretch and injury(passive) caused byintimal hyperplasia
injury of the vessel wall during aggressive embolectomy(passive) caused byintimal hyperplasia
Conclusion Implantation of a sirolimus - eluting stent seemsto effectively preventintimal hyperplasia
to restenosis of the arteryleadingto restenosis of the artery
recurrent in - stent stenosis(passive) caused byrecurrent in - stent stenosis
to stenosis and occlusion of hSVleadingto stenosis and occlusion of hSV
to luminal stenosis and occlusionleadingto luminal stenosis and occlusion
the stenosis and possible occlusioncreatingthe stenosis and possible occlusion
in a flow - limiting stenosisresultingin a flow - limiting stenosis
to eventual restenosis.22–27leadingto eventual restenosis.22–27
to mild in - stent restenosis ... no obvious in stent restenosis ( > 50 %ledto mild in - stent restenosis ... no obvious in stent restenosis ( > 50 %
CAS for recurrent carotid stenosis(passive) is ... caused byCAS for recurrent carotid stenosis
in stenosis within the stent.3can resultin stenosis within the stent.3
narrowing of the blood vessels , a phenomenon called restenosis and itcan causenarrowing of the blood vessels , a phenomenon called restenosis and it
narrowing of the blood vessels , a phenomenon known as restenosiscan causenarrowing of the blood vessels , a phenomenon known as restenosis
to fibrotic response leading to stenosis and occlusionsleadingto fibrotic response leading to stenosis and occlusions
to restenosis after injury ( 19,27–32leadingto restenosis after injury ( 19,27–32
to stenosis around the anastomoses and bypass thrombosisleadingto stenosis around the anastomoses and bypass thrombosis
stenosis and markedly less patency rates [ 4causingstenosis and markedly less patency rates [ 4
the restenosis,2 it recently became clear that restenosis after PTA is caused mainly by shrinkage of the vessel wall ( “ constrictive vascular remodelingcausesthe restenosis,2 it recently became clear that restenosis after PTA is caused mainly by shrinkage of the vessel wall ( “ constrictive vascular remodeling
elastic recoil and negative remodeling which caused restenosis after PTCA mainly is counteracted by stents , so in - stent restenosis(passive) is ... caused byelastic recoil and negative remodeling which caused restenosis after PTCA mainly is counteracted by stents , so in - stent restenosis
a focal stenosis ( approximately 40 % ) of the distal aspect of the FDS(passive) probably caused bya focal stenosis ( approximately 40 % ) of the distal aspect of the FDS
agent in a time dependent mannerpreventingagent in a time dependent manner
Graft stenosis ... a pathology characterized by smooth muscle cell ( SMC ) proliferation and migration(passive) is caused byGraft stenosis ... a pathology characterized by smooth muscle cell ( SMC ) proliferation and migration
D. LIERMANN , Ruppert Bauernsachs , Bernd Schopohl , H - D Böttcher Restenosis in peripheral vessels(passive) caused byD. LIERMANN , Ruppert Bauernsachs , Bernd Schopohl , H - D Böttcher Restenosis in peripheral vessels
from the migration and proliferation of vascular smooth muscle cells ( VSMCsresultingfrom the migration and proliferation of vascular smooth muscle cells ( VSMCs