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Our experimentsledto a kinetic model for the reactivation phenomenon for which rate constants were determined
Hypomethylation at the pericentric repeat regionsalso leadsto an increase in instability without the activation of transposable elements
coupledleadingto either activation of phospholipase C , direct activation of K - channels
any other processleadingto variations in the dissociation rate constants of the complexes transmitting the activation
An example of this work ... workleadsto activation energies and rate constants for radical reactions
Cone - specific Ranbp2 ablationcausesthe temporal activation of a cone - intrinsic molecular cascade highlighted by the early activation of metalloproteinase
each reviewresultsin a uniform activation of the underlying synaptic structure
ureaseleadto a putative structure for the urease activation complex
BRAF ... a dominant transforming proteincausesconstitutive activation of the MAPK pathway , independent of RAS activation
structural models of such proteins ... K. aerogenes ureaseleadto a putative structure for the urease activation complex
The initiation of the caspases cascademay leadto the activation of endonucleases like caspase - activated DNAase
G2 / M arrestresultsfrom untimely activation of the structure - specific endonuclease
K. aerogenes ureaseleadto a putative structure for the urease activation complex
strongly(passive) is ... influencedThe time constant of activation
The oxidation of the drug intermediate 1-(2-hydroxyethyl)piperidine by [ Ag(HIO6)2]5- was carefully studied by spectroscopic techniquesleadingto the determination of the rate constants and associated activation parameters
The induction of apoptosis ... a 40-kDa membrane protein ( 14 ) allowing recruitment of the adaptor protein Fas - associated death domain ( FADD ) ( 15 ) and binding of procaspase-8 , resulting in the formation of the death - inducing signaling complex ( DISC ) ( 16 , 17leadsto the activation of effector caspase-3 by active Caspase-8
GMR81B12 > TrpA1resultedin a constant extension of the labella at the activation
binding of the death receptorscausesactivation of caspase-8 , an initiator caspase
Inactivation of AP-1causesligand - dependent activation of Notch
indeterminant processesresultingfrom the activation of open - ended structures
The overexpression or permanent activation of RTKs and GPCRs , as well as the exposure to environmental carcinogenscauseconstant activation of PI3K / Akt
a detrimental positive feedback loopcausingthe constant activation of AhR.
other factorscontributetransients elicited by activation of GPCRs
to an increase in chemiluminescenceleadingto an increase in chemiluminescence
to initiation of further repair cascademay leadto initiation of further repair cascade
The change in steady - state activation(passive) may be caused byThe change in steady - state activation
to abnormal checkpoint activation and apoptosis ... 42leadsto abnormal checkpoint activation and apoptosis ... 42
to inactivation of mTOR - activated GTP - binding protein Rhebleadingto inactivation of mTOR - activated GTP - binding protein Rheb
to a reduction in the level of cGMP within the photoreceptoreventually leadingto a reduction in the level of cGMP within the photoreceptor
fibrosiscausesfibrosis
to increased proliferation and reduced apoptosis of melanoma cells under anchorage - deprived conditionscontributesto increased proliferation and reduced apoptosis of melanoma cells under anchorage - deprived conditions
phosphorylation earlier than the TLR2 agonist ( 25causedphosphorylation earlier than the TLR2 agonist ( 25
a constant displacement of the proboscis consistent with the contraction of muscle 9 ( Gordon and Scott , 2009causeda constant displacement of the proboscis consistent with the contraction of muscle 9 ( Gordon and Scott , 2009
to a cascade of intracellular protein - protein interactions involving a set of adapter proteins that ultimately leads to the appropriate cellular effectsleadsto a cascade of intracellular protein - protein interactions involving a set of adapter proteins that ultimately leads to the appropriate cellular effects
in cleavage of glycophorins from the erythrocyte surface and facilitating com- plement - mediated lysisresultingin cleavage of glycophorins from the erythrocyte surface and facilitating com- plement - mediated lysis
to its capacity to evade killingcontributesto its capacity to evade killing
in thick lamellipodial ( dSMAC ) and lamellar ( pSMAC ) F - actin meshworksresultingin thick lamellipodial ( dSMAC ) and lamellar ( pSMAC ) F - actin meshworks
to filopodia and focal complex formationleadsto filopodia and focal complex formation
to apoptosisat least partly ... contributesto apoptosis
to the death of nerve cell that the receptor is found on ... perhaps mimicking the complete absence of the TRPV1 receptorscan ... leadto the death of nerve cell that the receptor is found on ... perhaps mimicking the complete absence of the TRPV1 receptors
significant degradation of DCA and DCA removal efficiency increased with the increasing persulfate concentrationcausedsignificant degradation of DCA and DCA removal efficiency increased with the increasing persulfate concentration
to cardiac and renal fibrosisleadsto cardiac and renal fibrosis