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Qaagi - Book of Why

Causes

Effects

The overexpression or permanent activation of RTKs and GPCRs , as well as the exposure to environmental carcinogenscauseconstant activation of PI3K / Akt

Knockdown of Atg5preventedTNFa - dependent activation of proapoptotic caspases

Study of this behavior as a function of temperatureleadsto determination of the rate constant and the activation energy for the bond rotation

the present studywas designedto document variability in rate constants underlying AChR activation

a wide range of stimulican causeactivation in mesial temporal structures

GMR81B12 > TrpA1resultedin a constant extension of the labella at the activation

to the wanted laser of SIT(passive) should georeference createdsingularities engaging the activation structure

of SCP-2134 must be sufficiently scrambledto preventactivation of activation 's anomalous properties

to act , not only by inactivating the serine - threonine kinase , but also via the inhibition of a second kinase that controls the activity of PP1Acausingunabated activation of KCC through dephosphorylation

any other processleadingto variations in the dissociation rate constants of the complexes transmitting the activation

to CR3 's capacity to evade killingcontributesto CR3 's capacity to evade killing

to be equalwere setto be equal

to MDR-1can leadto MDR-1

in the flutter waves seen in the rface ECGresultedin the flutter waves seen in the rface ECG

to cardiac and renal fibrosis in spite of only a modest increase in blood pressureleadsto cardiac and renal fibrosis in spite of only a modest increase in blood pressure

to the death of nerve cell that the receptor is found on ... perhaps mimicking the complete absence of the TRPV1 receptorscan ... leadto the death of nerve cell that the receptor is found on ... perhaps mimicking the complete absence of the TRPV1 receptors

to control of cerebral vascular tonecontributesto control of cerebral vascular tone

directlycould ... contributedirectly

Drok ( Drosophila Rho - kinasetriggersDrok ( Drosophila Rho - kinase

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