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The overexpression or permanent activation of RTKs and GPCRs , as well as the exposure to environmental carcinogenscauseconstant activation of PI3K / Akt
GMR81B12 > TrpA1resultedin a constant extension of the labella at the activation
any other processleadingto variations in the dissociation rate constants of the complexes transmitting the activation
to cardiac and renal fibrosisleadsto cardiac and renal fibrosis
to the death of nerve cell that the receptor is found on ... perhaps mimicking the complete absence of the TRPV1 receptorscan ... leadto the death of nerve cell that the receptor is found on ... perhaps mimicking the complete absence of the TRPV1 receptors
to its capacity to evade killingcontributesto its capacity to evade killing