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Qaagi - Book of Why

Causes

Effects

KLF4 activitycausinginhibition of apoptosis

PI3 K inhibition by LY294002causedinhibition of Akt phosphorylation

The results ... productcauses50 % inhibition of the polymerization

productcauses50 % inhibition of the polymerization

activating small Rho GTPaseleadingto inhibition of actin polymerization

E6 / E6APcausinginhibition of apoptosis

These processes depend on deactivation of small Rho GTPaseleadingto inhibition of actin polymerization

In addition , it has been suggestedmay leadto inhibition of microtubule polymerization

Activation of cannabinoid receptorscausesinhibition of spasticity

by dual inhibition of PDGFR - beta and VEGFR-2 kinases(passive) is caused byinhibition of angiogenesis

the ADAMs cytotoxic propertiesmay resultfrom the inhibition of tubulin polymerization

Extracellular secretion of PGK1 by tumor cells increases angiostatin activitycausinginhibition of angiogenesis

by excessive addition(passive) caused bypolymerization inhibition

Activation of both receptors by chemerin is via coupling to Gi / ocausinginhibition of adenylyl cyclase

in the UV polymerization(passive) is ... causedinhibition of polymerization

by oxygen(passive) caused bypolymerization inhibition

by oxygen(passive) to be caused bypolymerization inhibition

by oxygen(passive) caused bythe polymerization inhibition

Binding by tacrolimuscausesinhibition of calcineurin

likelywould resultin an inhibition of polymerization

so it is likelywould resultin an inhibition of polymerization

the polymerization rate ... tendto causeinhibition of polymerization

ammonia , a product of MEAw degradation(passive) can be caused byinhibition of acetoclastic methanogenesis

disrupt actinleadingto inhibition of its polymerization

the redox reaction ... the adhesion interfacewhere ... is causedthe polymerization inhibition

the oligomerdoes ... causepolymerization inhibition

Serum withdrawalcausedinhibition of AKT

Local release of blebbistatin at the equatorcausedinhibition of cytokinesis

Diabetescausesinhibition of glucose-6-phosphate dehydrogenase

transition metalscausinginhibition of sporulation

vaccinecausinginhibition of erythropoiesis

Crcausedinhibition of U46619

Diabetescausesinhibition of glucose-6-phosphate

LeeleadsInhibition of hexokinase

to a decrease in adhesion 19,26may ... leadto a decrease in adhesion 19,26

to reduced cAMP production and proteinleadingto reduced cAMP production and protein

in G2 / M phase arrest and tumor cell apoptosismay resultin G2 / M phase arrest and tumor cell apoptosis

alsomay ... leadalso

in both prevention of A / E lesion formation and decreased invasionresultedin both prevention of A / E lesion formation and decreased invasion

to the hemolytic syndromes that occur in patients with acute poisoning by thesemay contributeto the hemolytic syndromes that occur in patients with acute poisoning by these

a tumor disease(passive) can be influenceda tumor disease

in cell deformation , displacement , and detachmentresultedin cell deformation , displacement , and detachment

to a decrease of its transcriptional activityleadsto a decrease of its transcriptional activity

to a reduction in TLR - enhanced bacterial internalizationleadsto a reduction in TLR - enhanced bacterial internalization

Inhibition of apoptosiscan ... can resultInhibition of apoptosis

to activation of the p53 transcription factor resulting in cell cycle arrest or apoptosis following DNA damage and or oncogenic activationleadsto activation of the p53 transcription factor resulting in cell cycle arrest or apoptosis following DNA damage and or oncogenic activation

in a number of cancers , inflammatory diseases , and viral infectionscan resultin a number of cancers , inflammatory diseases , and viral infections

in a number of cancers , autoimmune diseases , inflammatory diseases , and viral infectionscan resultin a number of cancers , autoimmune diseases , inflammatory diseases , and viral infections

a rise in type II collagen and glycosaminoglycan creationcausesa rise in type II collagen and glycosaminoglycan creation

the concentration of acetylcholine to increase in the braincausesthe concentration of acetylcholine to increase in the brain

cell - cycle arrest and subsequent apoptosis of the target cellcausescell - cycle arrest and subsequent apoptosis of the target cell

accumulation of acetylcholinecausesaccumulation of acetylcholine

to accumulation of acetylcholinemay leadto accumulation of acetylcholine

in apoptosisresultsin apoptosis

the total protein leveldoes ... influencethe total protein level

the adhesion interface(passive) where ... is causedthe adhesion interface

acetylcholine to accumulate , leading to overstimulation of muscles and organs , resulting in convulsions , paralysis and deathcausesacetylcholine to accumulate , leading to overstimulation of muscles and organs , resulting in convulsions , paralysis and death

in the UV polymerizationis ... causedin the UV polymerization

a Trp53-dependent reduction in zygote developmentcauseda Trp53-dependent reduction in zygote development

in increased gut motilityresultsin increased gut motility

to cell cycle arrestleadsto cell cycle arrest

to enhancement of long - term memory formation ( McQuown et al . , 2011leadsto enhancement of long - term memory formation ( McQuown et al . , 2011

to allergic reactionscontributesto allergic reactions

persistent firing of nerve pulses leading to nerve dysfunctioncausespersistent firing of nerve pulses leading to nerve dysfunction

to temporary or permanent decolorizationmay leadto temporary or permanent decolorization

hereditary axon degenerationcauseshereditary axon degeneration

to an increase in the total amount of putrescine and spermidine taken up from the extracellular mediumledto an increase in the total amount of putrescine and spermidine taken up from the extracellular medium

in producing a form of HDL that is not cardioprotectivemight resultin producing a form of HDL that is not cardioprotective

to microtubule arrestleadingto microtubule arrest

to increased free erythrocyte protoporphyrin ( FEP ) in the blood which can then bind to zinc to yield zinc protoporphyrinleadsto increased free erythrocyte protoporphyrin ( FEP ) in the blood which can then bind to zinc to yield zinc protoporphyrin

to cell deathleadsto cell death

T cell anergycausesT cell anergy

to the hemolytic syndromes that occur in patients with acute poisoning by these metalsmay contributeto the hemolytic syndromes that occur in patients with acute poisoning by these metals

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