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Activation of cannabinoid receptorscausesinhibition of spasticity
hydrogen peroxide , which can inhibit the activity of tyrosinase , but also Inhibition of dopachrome and DHICA oxidase enzyme activity(passive) caused byinhibition of hemolysis
by dual inhibition of PDGFR - beta and VEGFR-2 kinases(passive) is caused byinhibition of angiogenesis
Hydrocodone Description : Hydrocodone is a phenanthrene derivative opiate agonist that binds to opioid receptors in the CNScausinginhibition of ascendin
Extracellular secretion of PGK1 by tumor cells increases angiostatin activitycausinginhibition of angiogenesis
zone 2 ) paracrine - mediated receptor autophosphorylation at Tyr1213may causeinhibition of metastasis
Binding by tacrolimuscausesinhibition of calcineurin
AMPK activationcausesinhibition of mTOR
a 10 Hz burstcausedinhibition of SPNs
Phosphorylation of raptor by AMPKthen causesinhibition of mTOR
Midazolammay causeinhibition of levodopa
Insulin deficiencycausesinhibition of lipogenesis
depletion of Nanog - likecausedinhibition of Sox17expression
the potential for adrenergic agoniststo causeinhibition of contractility
singlet oxygen produced either by weakly coupled chlorophyll molecules or by or iron - sulfur centers(passive) is caused byInhibition of PSII
transition metalscausinginhibition of sporulation
vaccinecausinginhibition of erythropoiesis
Crcausedinhibition of U46619
singlet oxygen produced either by weakly coupled chlorophyll molecules or by cytochromes or iron - sulfur centers(passive) is caused byInhibition of PSII
Injection of curare in theventral lymph saccausedinhibition of muscletwitches
Diabetescausesinhibition of glucose-6-phosphate
Both low and high humiditycan causeinhibition of methanogenesis
Aminophyllinecausesinhibition of phosphodiesterase
Methylxanthine therapy ( aminophyllinecausesinhibition of phosphodiesterase
LeeleadsInhibition of hexokinase
the extensive dissolution of LCFAs in the reactor brothcausedinhibition of methanogenesis
HES1 , ... 3-kinasecausinginhibition of phosphoinositide
in increased urinary excretion of glucose and reduced plasma glucose concentrationsresultsin increased urinary excretion of glucose and reduced plasma glucose concentrations
in an increase of urinary glucose excretionresultsin an increase of urinary glucose excretion
in reductions of plasma lipoprotein ( a ) levelsalso resultsin reductions of plasma lipoprotein ( a ) levels
in reductions of plasma Lp(a ) levelsalso resultsin reductions of plasma Lp(a ) levels
to a reduction in seizuresleadsto a reduction in seizures
in improved response of PDAC cells to ionizing radiation and gemcitabineresultedin improved response of PDAC cells to ionizing radiation and gemcitabine
to an accumulation of cyclic adenosineleadingto an accumulation of cyclic adenosine
thusleadsthus
in reduced reabsorption of filtered glucoseresultsin reduced reabsorption of filtered glucose
to the decrease in blood glucoseleadsto the decrease in blood glucose
in such resultsmay resultin such results
to cell cycle arrestleadsto cell cycle arrest
to improved glycemic controlleadsto improved glycemic control
to excess blood glucose D.would leadto excess blood glucose D.
to excess blood glucosewould leadto excess blood glucose
in an increased apoptosis and decrease in invasion and colony formation ability of HNSCC cell linesresultedin an increased apoptosis and decrease in invasion and colony formation ability of HNSCC cell lines
an increase in CGMP in smooth muscles causing vasodilation in blood vesselscausesan increase in CGMP in smooth muscles causing vasodilation in blood vessels
sometimesresultssometimes
these side effects(passive) are caused bythese side effects
enzyme methionine synthaseresultsenzyme methionine synthase
growth arrest in T. bruceicausesgrowth arrest in T. brucei
accumulation of ROMcausedaccumulation of ROM
in induction of Smad signallingresultedin induction of Smad signalling
a phenotype reminiscent of the cell - biological changes elicited by modulation of the expression levels of either TaP63 or Dicer itselfresulteda phenotype reminiscent of the cell - biological changes elicited by modulation of the expression levels of either TaP63 or Dicer itself
to an increase in ABCA1 transporter activity and the depletion of barrier cholesterol levels in keratinocytesleadsto an increase in ABCA1 transporter activity and the depletion of barrier cholesterol levels in keratinocytes
in complete loss of Prox1-positive horizontal cellsresultedin complete loss of Prox1-positive horizontal cells
to a 68 % reversible reduction in thyroid vasculature thus damaging thyroid structure and leading to alteration of the thyroid functionledto a 68 % reversible reduction in thyroid vasculature thus damaging thyroid structure and leading to alteration of the thyroid function
to lower expression of apoptotic genes in AMD cybrids ... supporting our hypothesis that modulation of this microRNA may improve cellular health and provide therapeutic benefits in RPE cellsleadsto lower expression of apoptotic genes in AMD cybrids ... supporting our hypothesis that modulation of this microRNA may improve cellular health and provide therapeutic benefits in RPE cells
to a marked increase of Nanog and LIN28 , which play essential functional roles in the enhancement of reprogramming ( 37early in the reprogramming process ledto a marked increase of Nanog and LIN28 , which play essential functional roles in the enhancement of reprogramming ( 37