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However , AA ( ornithine ) can chelate Zntherefore preventinginhibition of CPS1
Midazolammay causeinhibition of levodopa
B. The non - selective TRP channel antagonist Ruthenium redcausedinhibition of mechanosensitivity
rather targets a fraction of Mei2to preventinhibition of Mmi1
Spred1-TESK1 bindingcausesinhibition of TESK1
Azasterols have been shownto causeinhibition of 24-SMT
in altered expression patterns of the fiber - specific proteins , alpha - crystallin , filensin , phakinin and MIP(passive) signaling resultedInhibition of TGFbeta
Activation of GCN2 ... in turncausesinhibition of eIF2B.
Addition of the cyclin - dependent kinase ( CDK ) inhibitor roscovitine after S - phase entry reduced MVM replication , suggesting that CDK activity was critical for continued viral replication and virus - induced reduction of p21 may thus be necessaryto preventinhibition of CDK
The virus infects erythroid progenitor cellscausinginhibition of erythropoiesis
application of the test formulation ... particularly its potent antioxidant vitamin Ccausesinhibition of melanogenesis.9
Three patients ... which is knownto causeinhibition of CYP3A4
The fact that Ang II and caffeine induced an increase in [ Ca2+]i that preceded membrane depolarization ... an initial increase in [ Ca2+]icausesinhibition of IK(dr
Jason W Brooks , Gareth Pryce , +9 authors David Baker Activation of cannabinoid receptorscausesinhibition of spasticity
co - expression of GSK-3beta2 but not by co - expression of GSK-3alpha or GSK-3beta1(passive) was prevented byInhibition of neurite outgrowth
Parvovirus B19 is a single - stranded DNA virus that is cytotoxic to erythroid progenitor cellscausinginhibition of erythropoiesis
Brown P. , Bridges D. , Ledent C. , Bifulco M. , Rice AS . , Di Marzo V. , Baker D. Activation of cannabinoid receptorscausesinhibition of spasticity
Eur J Pharmacol 2002 Mar 29;439(1 - 3):83 - 92 Activation of cannabinoid receptorscausesinhibition of spasticity
The failure to decrease CHOP-10 under some experimental conditionscausesinhibition of adipogenesis
Addition of acetylcholine to the culture medium at concentrations of 100 , 10 and 1 microMcausedinhibition of neurite outgrowth
Brooks JW , Pryce G , Bisogno T , Jaggar SI , Hankey DJ , Brown P , Bridges D , Ledent C , Bifulco M , Rice AS , Di Marzo V , Baker D.Pain Research Group , Department of Anaesthetics , Faculty of Medicine , Imperial College , Chelsea and Westminster Hospital Campus , London , UK.Activation of cannabinoid receptorscausesinhibition of spasticity
hydrogen peroxide , which can inhibit the activity of tyrosinase , but also Inhibition of dopachrome and DHICA oxidase enzyme activity(passive) caused byinhibition of hemolysis
63 Text - Based Restudy 64 TGF - beta receptor type I 2012preventsInhibition of sumoylation
0104causedinhibition of SHP2
TSC2 knockoutpreventedinhibition of mTOR
Coexpression of masGRK3ctpreventedinhibition of CaV2.2
targeting dSesndid preventinhibition of dTOR
Active S6K(Pcausesinhibition of IRS1
the highest dilutioncausinginhibition of haemagglutination
the artificial substrate GTN and/or ROScauseinhibition of ALDH-2
chondroitin sulfate proteoglycans(passive) caused byinhibition of neurite outgrowth
excess TIMP-2leadsinhibition of MMP-2
Prostaglandin F2 alphacausedinhibition of mitoses
Agonist stimulation of TLR8 in cultured cortical neuronscausesinhibition of neurite outgrowth
Experiments blocking NgR from interacting with MAGpreventedinhibition of neurite outgrowth
View at Google Scholar S. Agarwal , S. Loder , C. Brownley et al .preventsInhibition of Hif1α
Gallery of Irs Form Trk1 MEK Inhibitor PD09059PreventsInhibition Of CDK
Overexpression of the nicotinamidase , Pnc1ppreventsinhibition of Sir2p
var grifdata = [ { ' pmid ' : 28977798 , ' textresultedInhibition of Beclin-1
Cell swelling , which may occur during activation of CFTR or upon permeabilization ... unlikelyto causeinhibition of ENaC
to decreased malondialdehyde - acetaldehyde adduct formation in VL-17A cells under chronic alcohol exposure " , abstract =leadsto decreased malondialdehyde - acetaldehyde adduct formation in VL-17A cells under chronic alcohol exposure " , abstract =
to reduced AICD signalling and neurogenesis as well as enhanced Mtap2 expressionleadsto reduced AICD signalling and neurogenesis as well as enhanced Mtap2 expression
apoptosis induced by high glucose and palmitate [ 7 , 8preventedapoptosis induced by high glucose and palmitate [ 7 , 8
inhibition of glucose reabsorption in renal proximal tubular cellscausesinhibition of glucose reabsorption in renal proximal tubular cells
to inversion of metastatic characteristics and restoration of paclitaxel sensitivity of chronic chemoresistant ovarian carcinoma cellsleadsto inversion of metastatic characteristics and restoration of paclitaxel sensitivity of chronic chemoresistant ovarian carcinoma cells
to decreased malondialdehyde - acetaldehyde adduct formation in VL-17A cells under chronic alcohol exposure — Research Nebraska Kavitha Swaminathan , Dahn L Clemensleadsto decreased malondialdehyde - acetaldehyde adduct formation in VL-17A cells under chronic alcohol exposure — Research Nebraska Kavitha Swaminathan , Dahn L Clemens
the inactivation of nuclear CRLs and hampers EBV DNA replicationpreventsthe inactivation of nuclear CRLs and hampers EBV DNA replication
muscle weakness and disruption of sarcomere structure during hindlimb suspension by Jay J Salazar and Daniel E Michele and Susan V Brooks Journal of Applied Physiology , ISSN 8750 - 7587 , 01/2010 , Volume 108 , Issue 1preventsmuscle weakness and disruption of sarcomere structure during hindlimb suspension by Jay J Salazar and Daniel E Michele and Susan V Brooks Journal of Applied Physiology , ISSN 8750 - 7587 , 01/2010 , Volume 108 , Issue 1
to downregulation of Nanog in ES cells.(Aledto downregulation of Nanog in ES cells.(A
to lower hepatic lipid concentrations and improved glycemic control in mice given a HFD , which works with the additional exploration of NaCT inhibitors for the treating metabolic diseasesledto lower hepatic lipid concentrations and improved glycemic control in mice given a HFD , which works with the additional exploration of NaCT inhibitors for the treating metabolic diseases
to downregulation of Nanog in mouse ES cellsleadsto downregulation of Nanog in mouse ES cells
KA - induced truncation of Dyrk1A , 3R - Tau expression , and Tau Thr212 phosphorylationpreventsKA - induced truncation of Dyrk1A , 3R - Tau expression , and Tau Thr212 phosphorylation
to ∼10 fold NEK8 induction of transcript , but not protein , while miR-149 * mimic transfection reduced NEK8 transcript expression below the level of the control ( Figure 4ledto ∼10 fold NEK8 induction of transcript , but not protein , while miR-149 * mimic transfection reduced NEK8 transcript expression below the level of the control ( Figure 4
tau degradation and cell deathpreventstau degradation and cell death
to significantly decreased populations of Foxp3 +signaling ledto significantly decreased populations of Foxp3 +
amphetamine?s effect on dendritic spine densitypreventedamphetamine?s effect on dendritic spine density
in protection against curcumin - induced cell deathresultedin protection against curcumin - induced cell death
cholangiocarcinoma progression through regulating cell proliferation and motility ... arresting cell cycle and reducing Erk activationpreventscholangiocarcinoma progression through regulating cell proliferation and motility ... arresting cell cycle and reducing Erk activation
in enhanced DENV-2-induced cell deathresultedin enhanced DENV-2-induced cell death
the morphological characteristics of pyroptosis , including death ( 27preventsthe morphological characteristics of pyroptosis , including death ( 27
the cleavage of pro - caspase-8 to active caspase-8preventsthe cleavage of pro - caspase-8 to active caspase-8
a compensatory increase in caspase-8 activity ... resulting in no effect of caspase-1 inhibition on cell viability as seen in Hmeso cells ( Fig 5Amay causea compensatory increase in caspase-8 activity ... resulting in no effect of caspase-1 inhibition on cell viability as seen in Hmeso cells ( Fig 5A
to decreased advanced glycated end product formation in high glucoseleadsto decreased advanced glycated end product formation in high glucose
to decreased phagocyte production of endotoxin - induced proinflammatory cytokines [ 32ledto decreased phagocyte production of endotoxin - induced proinflammatory cytokines [ 32
the proteolytic activation of IL-1β ... resulting in decreased secretion of the mature form from microgliapreventsthe proteolytic activation of IL-1β ... resulting in decreased secretion of the mature form from microglia
NMDA - induced cell death and beta - amyloid - induced synaptic dysfunction in hippocampal slice culturespreventsNMDA - induced cell death and beta - amyloid - induced synaptic dysfunction in hippocampal slice cultures
NMDA - induced cell death and β - amyloid - induced synaptic dysfunction in hippocampal slice culturespreventsNMDA - induced cell death and β - amyloid - induced synaptic dysfunction in hippocampal slice cultures
xenograft tumor growth of MDA - MB-231 breast cancer cells and MYC - dependent neuroblastoma cells [ 33,34preventsxenograft tumor growth of MDA - MB-231 breast cancer cells and MYC - dependent neuroblastoma cells [ 33,34
decreased cell viability ( Figure 5B ) , migratory and invasive capability ( Figure 5C – D ) of T24 cancer cellscauseddecreased cell viability ( Figure 5B ) , migratory and invasive capability ( Figure 5C – D ) of T24 cancer cells
in increased strength normalized to muscle size ( sPo ) in EDL muscles from male ( P<0.0001 , main effect GLM ANOVA ) andresultedin increased strength normalized to muscle size ( sPo ) in EDL muscles from male ( P<0.0001 , main effect GLM ANOVA ) and
to strongly reduced viral titers in primary human airway cultures and also limited viral replication in a mouse modelledto strongly reduced viral titers in primary human airway cultures and also limited viral replication in a mouse model
in decreased tumor necrosis factor ( TNF ) alpha release ... suggesting that enhanced TGFbeta expression in the plaque macrophages contributes to the proinflammatory effects in the vessel wallresultedin decreased tumor necrosis factor ( TNF ) alpha release ... suggesting that enhanced TGFbeta expression in the plaque macrophages contributes to the proinflammatory effects in the vessel wall
to increased glucose excretion , which is also seen in patients with genetic mutations in the kidney 's specific SGLT2 isoform , a disease which is termed renal glycosuria.69 Interestinglyleadsto increased glucose excretion , which is also seen in patients with genetic mutations in the kidney 's specific SGLT2 isoform , a disease which is termed renal glycosuria.69 Interestingly
apoptosis and improves angiogenesis in endothelial cells under different metabolic stresses , including high glucose and high palmitate alone or in combination ... and in diabetic hearts ( Figs 3d , e and 4 and ESM Figs 5 , 7 and 8)preventsapoptosis and improves angiogenesis in endothelial cells under different metabolic stresses , including high glucose and high palmitate alone or in combination ... and in diabetic hearts ( Figs 3d , e and 4 and ESM Figs 5 , 7 and 8)
in SIRT1 accumulation in the ApoE−/− mouse aorta and ApoE−/−/caspase-1−/− mice had attenuated early atherosclerosis , decreased aortic expression of proinflammatory cytokines , and reduced aortic monocyte recruitment , as well as decreased endothelial activation [ 59resultedin SIRT1 accumulation in the ApoE−/− mouse aorta and ApoE−/−/caspase-1−/− mice had attenuated early atherosclerosis , decreased aortic expression of proinflammatory cytokines , and reduced aortic monocyte recruitment , as well as decreased endothelial activation [ 59
reabsorption of the glucosepreventsreabsorption of the glucose
in improved plasma glucose control for patients living with diabetes mellitus type 2 ( DMT2can resultin improved plasma glucose control for patients living with diabetes mellitus type 2 ( DMT2
to reduced growth in glioma cell lines ( Gangemi et al , 2009leadsto reduced growth in glioma cell lines ( Gangemi et al , 2009
the self - renewal abilitypreventedthe self - renewal ability
growth arrest in T. bruceicausesgrowth arrest in T. brucei