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Activation of cannabinoid receptorscausesinhibition of spasticity
hydrogen peroxide , which can inhibit the activity of tyrosinase , but also Inhibition of dopachrome and DHICA oxidase enzyme activity(passive) caused byinhibition of hemolysis
by dual inhibition of PDGFR - beta and VEGFR-2 kinases(passive) is caused byinhibition of angiogenesis
the binding of plasminogen ... fibrin - plasminogen interactioncausesinhibition of fibrinolysis
Hydrocodone Description : Hydrocodone is a phenanthrene derivative opiate agonist that binds to opioid receptors in the CNScausinginhibition of ascendin
by chemical irritants(passive) provoked byinhibition of bronchospasms
Extracellular secretion of PGK1 by tumor cells increases angiostatin activitycausinginhibition of angiogenesis
a metabolic signal from liver to peripheral fatcausinginhibition of lipolysis
a 10 Hz burstcausedinhibition of SPNs
Serum withdrawalcausedinhibition of AKT
Midazolammay causeinhibition of levodopa
Insulin deficiencycausesinhibition of lipogenesis
depletion of Nanog - likecausedinhibition of Sox17expression
vaccinecausinginhibition of erythropoiesis
Crcausedinhibition of U46619
Diabetescausesinhibition of glucose-6-phosphate
Both low and high humiditycan causeinhibition of methanogenesis
the extensive dissolution of LCFAs in the reactorcausedinhibition of methanogenesis
Aminophyllinecausesinhibition of phosphodiesterase
Methylxanthine therapy ( aminophyllinecausesinhibition of phosphodiesterase
LeeleadsInhibition of hexokinase
a resonance - suppported positive charge or radical(passive) may be influenced byinhibition of cox-2
the extensive dissolution of LCFAs in the reactor brothcausedinhibition of methanogenesis
HES1 , ... 3-kinasecausinginhibition of phosphoinositide
transcriptional upregulation of the human ortholog of zebrafish Her6 , HES1causinginhibition of phosphoinositide
to useful activity in such chronic diseasesmay leadto useful activity in such chronic diseases
suppression of endogenous glucose production independent of changes in insulincausessuppression of endogenous glucose production independent of changes in insulin
FFA concentration in circulation to decreasecausesFFA concentration in circulation to decrease
to decreases in cell proliferation , migration and invasion in gastric cancer cells via suppression of cyclin D1 and EMT progressionleadsto decreases in cell proliferation , migration and invasion in gastric cancer cells via suppression of cyclin D1 and EMT progression
to disruption of the membrane and cell deathleadsto disruption of the membrane and cell death
to an increase in H3K27me3 levelswould leadto an increase in H3K27me3 levels
cardiac dysfunction during pressure overloadcausescardiac dysfunction during pressure overload
to an accumulation of cyclic adenosineleadingto an accumulation of cyclic adenosine
to selective repression of these genesleadsto selective repression of these genes
to NOTCH1 degradationledto NOTCH1 degradation
alsoresultedalso
to increased levels of its neurotoxic substrate quinolinic acid ( QUINmight leadto increased levels of its neurotoxic substrate quinolinic acid ( QUIN
an increase in CGMP in smooth muscles causing vasodilation in blood vesselscausesan increase in CGMP in smooth muscles causing vasodilation in blood vessels
to increased free erythrocyte protoporphyrin ( FEP ) in the blood which can then bind to zinc to yield zinc protoporphyrinleadsto increased free erythrocyte protoporphyrin ( FEP ) in the blood which can then bind to zinc to yield zinc protoporphyrin
to cell deathleadingto cell death
to the changes of the p53 gene expressionledto the changes of the p53 gene expression
to reduced VEGF and iNOS expressionsledto reduced VEGF and iNOS expressions
to increase free erythrocyte protoporphyrin ( FEP ) in the blood which can then bind to zinc to yield zinc protoporphyrin ( ZPPleadsto increase free erythrocyte protoporphyrin ( FEP ) in the blood which can then bind to zinc to yield zinc protoporphyrin ( ZPP
in increased glioblastoma propagationresultedin increased glioblastoma propagation
in induction of Smad signallingresultedin induction of Smad signalling
a phenotype reminiscent of the cell - biological changes elicited by modulation of the expression levels of either TaP63 or Dicer itselfresulteda phenotype reminiscent of the cell - biological changes elicited by modulation of the expression levels of either TaP63 or Dicer itself
a reduction in frequency of alloreactive effector T cells activated by allogeneic DCscauseda reduction in frequency of alloreactive effector T cells activated by allogeneic DCs
to an increase in ABCA1 transporter activity and the depletion of barrier cholesterol levels in keratinocytesleadsto an increase in ABCA1 transporter activity and the depletion of barrier cholesterol levels in keratinocytes
in commitment to the neuroectodermal lineageresultedin commitment to the neuroectodermal lineage
to accumulation of progeny virions in the nucleus of infected cellsleadsto accumulation of progeny virions in the nucleus of infected cells
the Cubitus interruptus ( Ci ) , Fused , and Cos protein complex attached to microtubules to remain intactcausesthe Cubitus interruptus ( Ci ) , Fused , and Cos protein complex attached to microtubules to remain intact