to smooth muscle relaxation and bronchodilationleadsto smooth muscle relaxation and bronchodilation

to smooth muscle relaxation and vasodilation Metabolism One way or anotherleadsto smooth muscle relaxation and vasodilation Metabolism One way or another

to smooth muscle relaxation and consequently vasodilatationleadsto smooth muscle relaxation and consequently vasodilatation

to contraction and migrationleadingto contraction and migration

to contraction of the circumferential actomyosin ringleadingto contraction of the circumferential actomyosin ring

smooth muscle contraction Tolerancepreventssmooth muscle contraction Tolerance

to an inhibition of collagen gel contraction ( 7leadingto an inhibition of collagen gel contraction ( 7

myosin light chain phosphorylation and muscle contraction ( 17 , 23causesmyosin light chain phosphorylation and muscle contraction ( 17 , 23

inhibition of smooth muscle contraction and thus vasodilationcausesinhibition of smooth muscle contraction and thus vasodilation

actin - myosin contraction and paracellular hyperpermeability.9thereby triggeringactin - myosin contraction and paracellular hyperpermeability.9

interaction between myosin and actin taking to contraction of the smooth musculuscausesinteraction between myosin and actin taking to contraction of the smooth musculus

interaction between myosin and actin leading to contraction of the smooth musclecausesinteraction between myosin and actin leading to contraction of the smooth muscle

to attenuation of MLC phosphorylation [ 162leadingto attenuation of MLC phosphorylation [ 162

an actin - myosin - based contraction that may lead to a centripetal retraction of endothelial cellscausesan actin - myosin - based contraction that may lead to a centripetal retraction of endothelial cells

to premature contraction of actin - myosin cables in the margin and separation of the blastomeres from the yolk cellledto premature contraction of actin - myosin cables in the margin and separation of the blastomeres from the yolk cell

to show the calcium ion status in vivodesignedto show the calcium ion status in vivo

contraction - pKG also activates myosin light chain phosphate see which phosphorylates myosincausescontraction - pKG also activates myosin light chain phosphate see which phosphorylates myosin

to steady force development ... and maintained constriction ( ‘ myogenic toneleadsto steady force development ... and maintained constriction ( ‘ myogenic tone

to contraction of an actin ring that , like a purse string , closes off the denuded region and restores barrier function [ 5leadingto contraction of an actin ring that , like a purse string , closes off the denuded region and restores barrier function [ 5

to myosin phosphorylation , myosin ATPase activation ... and ultimately stimulates actin and myosin cross - bridge cycling ( 5leadsto myosin phosphorylation , myosin ATPase activation ... and ultimately stimulates actin and myosin cross - bridge cycling ( 5

contraction.[9 ] Activation of Rho - kinase is a calcium independent pathway mediating VSMC contraction [ Figure 4].[10causingcontraction.[9 ] Activation of Rho - kinase is a calcium independent pathway mediating VSMC contraction [ Figure 4].[10

to DE phosphorylation of actomyosin phosphate complex thus causing relaxation of smooth musclesleadingto DE phosphorylation of actomyosin phosphate complex thus causing relaxation of smooth muscles

to a decrease in cell contractility as a result of the lack of myosin light chain phosphorylation by myosin light chain kinaseleadingto a decrease in cell contractility as a result of the lack of myosin light chain phosphorylation by myosin light chain kinase

tosmooth muscle relaxation and vasodilation.[51leadstosmooth muscle relaxation and vasodilation.[51

Cell retraction and gap formation(passive) were also prevented byCell retraction and gap formation

both MLC phosphorylation and SGLT1-initiated decreases in TERpreventsboth MLC phosphorylation and SGLT1-initiated decreases in TER

cell - shrinkage - induced activation of NKCC1preventscell - shrinkage - induced activation of NKCC1

in clearance of GFP - actin from the equatorial regionresultedin clearance of GFP - actin from the equatorial region

thrombin - induced changes in actin morphology and increased endothelial permeability ( 14preventsthrombin - induced changes in actin morphology and increased endothelial permeability ( 14

the abnormal accumulation of cortical microfilaments by CPI-17preventedthe abnormal accumulation of cortical microfilaments by CPI-17

to failure of cytokinesis and centrosomal amplificationleadingto failure of cytokinesis and centrosomal amplification

ASC of resting cells to collapse and most myosin IIB , F - actin , and ezrin to dissociate from ASCcausedASC of resting cells to collapse and most myosin IIB , F - actin , and ezrin to dissociate from ASC

dilation in vascular smooth musclecausingdilation in vascular smooth muscle

in the reducing the cross - bridge formation of actin - myosinresultingin the reducing the cross - bridge formation of actin - myosin

Na+-glucose cotransport - dependent tight junction regulation in natural mucosapreventsNa+-glucose cotransport - dependent tight junction regulation in natural mucosa

lamellar extension in all directions in neutrophils.23causedlamellar extension in all directions in neutrophils.23

howevermay ... resulthowever

increases in paracellular permeabilitypreventedincreases in paracellular permeability

Its contractile activity(passive) triggered byIts contractile activity

the TNF - induced change in actin stress fiberspreventsthe TNF - induced change in actin stress fibers