Loading ...
More ) Large platelets with high haemostatic activitymay leadto increased platelet aggregation
Technically they ’re attemptingto preventa rise in platelet aggregation
high levels of thromboxane a2 , a prostacyclin type hormone released by activated platelets(passive) caused byIncreased platelet aggregation
an inhibition of protein kinase C by -tocopherol(passive) caused bydecreased platelet aggregation
the activation of plateletsleadsto increased platelet aggregation
the local thrombus , endothelial injury , and probably the thrombolytic treatment itself(passive) caused byincreased platelet aggregation
diabetics ... spiky ’ plateletscan leadto increased platelet aggregation
platelet activityresultingin increased platelet aggregation
acts together with PAFto causeincreased aggregation of platelets
An activated plasmatic coagulation system together withcontributesincreased platelet aggregation
This situation , called hyperlipidemia and hypertriglyceridemiacausesan increase in platelet aggregation
Acquired conditionscausedecreased platelet aggregation
the cholesterol - enriched dietsleadto increased platelet aggregation
These same oxidative events can damage the endothelial cells lining the blood vessels ,resultingin increased platelet aggregation
as we age levels riseleadingto increasing platelet aggregation
prostaglandinscausedecreased platelet aggregation
the STA2-induced IP3productioncould contributeto the augmentation of platelet aggregation
Increased fibrinogen levelsresultin increased platelet aggregation
High levels of plasma fibrinogen after exercise result in elevated blood viscosity and this along with increased vWF binding , αIIbβ3 and P - Selectin expression allcontributeto the increased platelet aggregation
No change or an increase in EF , and no focal wall motion abnormalities in response to mental stress testing Mental stress testingresultedin increased platelet aggregation
thromboxane A2 activityleadingto increased platelet aggregation
Technically they are tryingto preventan increase in platelet aggregation
Angiotensin IIcontributesto increased platelet aggregation
TXA2 is a very strong blood platelet activator acting as a pro - aggregator and vasoconstrictor mediatorleadingto increased platelet aggregation
catecholamines released in response to pain(passive) caused byIncreased platelet aggregation
pro - inflammatory markerscontributeto increasing platelet aggregation
The resulting endothelial cell dysfunctionalso resultsin increased platelet aggregation
whether patient has failedto preventa rise in platelet aggregation
As / kg / day as sodium arsenite ) in the drinking water for 4 weeksresultedin increased platelet aggregation
Congenital disorderscausedecreased platelet aggregation
In the presence of high drug levels , such as during an infusion this is not a problem , however combined with the low trough levels with oral inhibitorscan leadto an increase in platelet aggregation
Ticarcillinmay causedecreased platelet aggregation
diabetes mellitus and disturbance of ocular circulation(passive) caused byincreased platelet aggregation activity
Both are less prominent , it has been shownto cause⢠increased platelet aggregation
20 - 6 anti - fungal drugs m. Gladwin and b. Figure 8.7 interstitial pulmonary edemaresultsin increased platelet aggregation
the prostaglandin metabolism disturbanceleadingto increased platelet aggregation
A platelet glycoprotein IIIa gene ( ITGB3 ) polymorphismresultingin increased platelet aggregation
a serotonin increase ... in turnmight provokeenhanced platelet aggregation
I HLA determinantsresultingin increased platelet aggregation
increased platelet thromboxane production (resultingin increased platelet aggregation
to the formation of arterial blood clotsleadingto the formation of arterial blood clots
to strokes , heart attacks and blood clotscan leadto strokes , heart attacks and blood clots
excessive coagulation of the bloodpreventingexcessive coagulation of the blood
to the formation of blood clots ( thrombus ) that can block blood flow , depriving tissues of nutrients and potentially causing a stroke or heart attackleadsto the formation of blood clots ( thrombus ) that can block blood flow , depriving tissues of nutrients and potentially causing a stroke or heart attack
your platelets to become less sticky , which is a good benefit if you are at high risk for a heart attack or blood clotcausesyour platelets to become less sticky , which is a good benefit if you are at high risk for a heart attack or blood clot
excessive blood clottingpreventingexcessive blood clotting
to the development of vascular complication in diabetes mellitusmight contributeto the development of vascular complication in diabetes mellitus
in leukocyte activationresultedin leukocyte activation
clots to form in arteriescan causeclots to form in arteries
invariablyresultsinvariably
a variety of effects : vasoconstriction , anoxia , ROS , atherosclerotic plaques , retinopathy , nephropathy and CV diseasecan causea variety of effects : vasoconstriction , anoxia , ROS , atherosclerotic plaques , retinopathy , nephropathy and CV disease
to vascular complicationsleadingto vascular complications
in concurrent myocardial and cerebral infarction from thrombosisresultingin concurrent myocardial and cerebral infarction from thrombosis
to increased blood clotting and increased angiotension IIleadsto increased blood clotting and increased angiotension II
in myocardial infarction and cerebral thrombosispossibly resultingin myocardial infarction and cerebral thrombosis
in a better blood lipid profile and less plaque formation ( 5resultingin a better blood lipid profile and less plaque formation ( 5
to the pathogenesis of acute myocardial infarctionmay contributeto the pathogenesis of acute myocardial infarction
to the pathogenesis of ischemic heart disease.23 ... 25may contributeto the pathogenesis of ischemic heart disease.23 ... 25
heavy bleeding that is difficult to controlcan causeheavy bleeding that is difficult to control
from vascular endothelial damageresultingfrom vascular endothelial damage
in coronary thrombosis in non - stenosed coronaryresultingin coronary thrombosis in non - stenosed coronary
in a blockageresultingin a blockage
in an adult and pediatric dosagesresultingin an adult and pediatric dosages
abnormal or prolonged bleeding during and after your dental implant surgerycan causeabnormal or prolonged bleeding during and after your dental implant surgery
a platelet plugcreatesa platelet plug
to an early “ call ” of the clot formationleadingto an early “ call ” of the clot formation
in prolonged bleeding timesresultingin prolonged bleeding times
after administration of 500 mg Aspirin and 125 mg Pycnogenolwas preventedafter administration of 500 mg Aspirin and 125 mg Pycnogenol
formation of microthrombicausingformation of microthrombi
from removal of NOresultingfrom removal of NO
in an endocrine glandresultingin an endocrine gland
to bruising in some individualsmay leadto bruising in some individuals
to micro thrombosis in blood vesselsmay leadto micro thrombosis in blood vessels
to the development of ischemic retinal lesions and thereby plays a significant role in the genesis of diabetic retinopathyleadsto the development of ischemic retinal lesions and thereby plays a significant role in the genesis of diabetic retinopathy
from increased fibrinogen , thrombospondin I and mucin [ 3 , 4 , 5 , 6 , 7 , 8resultingfrom increased fibrinogen , thrombospondin I and mucin [ 3 , 4 , 5 , 6 , 7 , 8
in increased complications such as location , activity , poor positioning or body piercing with contaminated needlesresultingin increased complications such as location , activity , poor positioning or body piercing with contaminated needles
heart attacks and strokescan triggerheart attacks and strokes
MI -asprin , plavix cox 2 enzyme inhibitormay preventMI -asprin , plavix cox 2 enzyme inhibitor
blood hypercoagulability(passive) caused byblood hypercoagulability
to microthrombi in blood vesselscan leadto microthrombi in blood vessels