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Smart Reasoning:

C&E

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Qaagi - Book of Why

Causes

Effects

microglial cellscausingneuronal injury in microglial - neuronal co - cultures and probably also in vivo

oligodendrocyte - specific MCT1 depletioncausedsevere axonal injury and motor neuron death in vivo ( 43

Neutrophilscontributeto ischemia / reperfusion injury in rat liver in vivo injury

oxidative stresscontributingto in vivo pulmonary injury and inflammation

The radiationcould resultin leg contracture and mitochondrial injury of the muscles

glutamate toxicity ... the synapseresultsin a die - back phenomenon in axons of motor neurons supplying the damaged skeletal muscles

p38 MAPK signalingcan contributesto amphotericin B and cyclosporine - induced renal injury in vivo

the overexpression ofcausesin vivo toxicity by disrupting neuromuscular junctions ( NMJs ) and inducing apoptosis in motor neurons

inhibition of lincRNA - p21resultedin neointimal hyperplasia in vivo in a carotid artery injury model

Antioxidant properties of magnesium lithospermate Bcontributeto the cardioprotection against myocardial ischemia / reperfusion injury in vivo and in vitro

the body ... blood flowresultsin muscle tension and susceptibility to musculoskeletal injury

embryonic stem ( ES ) cellscontributingto the process of muscle regeneration and their in vivo immunogenicity

The mechanismsleadingto placental and fetal injury in vivo

a novel hybrid promoterdesignedto express the GAA protein in skeletal and cardiac muscle

zinc toxicitycould contributeto neuronal injury in vivo

the primary triggerleadsto intracellular calcium fluxes and cardiac muscle contraction

a potentialto causeproximal tubule injury in vivo

the heart muscleresultingin injury and eventual death of muscle cells

Duchenne muscular dystrophyleadsto progressive deterioration in skeletal and cardiac muscle function

hypoxic ( low oxygen ) conditions(passive) caused byIschemic injury in skeletal muscle

NOX1 and NOX2contributein vivo to hyperoxia - induced acute lung injury

Plasmid constructsresultedin long - term in vivo gene expression in skeletal muscle

overexpression of this proteincausesmuscle degeneration in vivo

oxidative stressleadsto mitochondrial uncoupling in skeletal muscle in vivo

HCV nucleocapsid ( HCV - Cp ) , devoid of envelope proteins ... ableto causecryoglobulin - mediated tissue injury in vivo

Starvationleadsto skeletal muscle atrophy in vivo

the TWEAK - Fn14 systemcausesskeletal muscle wasting both in vitro and in vivo

This study(passive) was designedThis study

tenderness and painthereby causingtenderness and pain

contractioncausescontraction

a clinical syndrome(passive) caused bya clinical syndrome

general aches , pain , and tendernesscausinggeneral aches , pain , and tenderness

train - of - four fade in micecausestrain - of - four fade in mice

weakness(passive) caused byweakness

the defects(passive) caused bythe defects

a condition(passive) caused bya condition

myocardial apoptosis(passive) caused bymyocardial apoptosis

myocardial apoptosis(passive) caused bymyocardial apoptosis

to shockleadingto shock

during reperfusionmay be causedduring reperfusion

a condition(passive) caused bya condition

in weakness and loss of ambulationresultingin weakness and loss of ambulation

in weakness and loss of ambulationresultin weakness and loss of ambulation

efficiency of transfer and stability of expressioninfluencingefficiency of transfer and stability of expression

free serum myoglobin(passive) caused byfree serum myoglobin

This exercise(passive) is designedThis exercise

alsoresultedalso

creatinine phosphokinase and free serum myoglobin(passive) caused bycreatinine phosphokinase and free serum myoglobin

alsoresultedalso

increased mitochondrial ironcausesincreased mitochondrial iron

to the uncontrolled growth leading to tumormight contributeto the uncontrolled growth leading to tumor

in DNA damagemay resultin DNA damage

in increased myocardial damage and worsened heart functionresultingin increased myocardial damage and worsened heart function

Often , it?s(passive) caused byOften , it?s

to fibrosis and fatty tissue accumulationleadingto fibrosis and fatty tissue accumulation

the head and neck to suddenly and rapidly move forward , backward , and sidewaysmay causethe head and neck to suddenly and rapidly move forward , backward , and sideways

contraction of the target muscleto causecontraction of the target muscle

to the significant problem of age - related loss of muscle mass and may have implications for onset and progression of ALSmight contributeto the significant problem of age - related loss of muscle mass and may have implications for onset and progression of ALS

the filaments to disassemblecausingthe filaments to disassemble

in fluctuating weaknessresultingin fluctuating weakness

the Techniques(passive) is caused bythe Techniques

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Smart Reasoning:

C&E

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