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Qaagi - Book of Why

Causes

Effects

the release of superoxide anion ( O2− ) , a reactive oxygen species knownto causeoxidative stress and neuronal injury in vivo

Inflammationleadsto skeletal muscle atrophy in vivo

Starvationleadsto skeletal muscle atrophy in vivo

myospheres ... ableto contributeto injured muscle in vivo

Mutations in the pre - mRNAresultin the use of the skeletal muscle exon in vivo

The common effect of erythropoietin to promote cell viability and to provide for cell proliferation ... the neural stem cellscan ... contributeto haematopoietic and muscle tissue when transplanted in vivo

HSP60 injectedcausesneuronal and oligodendrocyte injury in the CNS in vivo through TLR4-dependent signaling

The muscular wall of the cardiac inflow tract consists of two layers - the inner smooth muscle layer and the outer myocardial sleeve - and the atrial progenitorscontributedto both cardiac and smooth muscle cells in vivo

the pathogenic mechanismsresultin thrombosis and tissue injury in vivo

This study(passive) was designedThis study

the transition to early differentiation of myogenic cells that would be reflected in lower myogenin expressionwould preventthe transition to early differentiation of myogenic cells that would be reflected in lower myogenin expression

alsoresultedalso

the tissue damage(passive) caused bythe tissue damage

a polarization shift that promotes regeneration and increases muscle strengthcreatinga polarization shift that promotes regeneration and increases muscle strength

detachment of MTOCs in proximal tubulescausesdetachment of MTOCs in proximal tubules

in 5-HT contractile hypersensitivity and increased responsiveness to other growth factorsresultingin 5-HT contractile hypersensitivity and increased responsiveness to other growth factors

apoptosis and hypoplasiacausesapoptosis and hypoplasia

in deposition of a high density interstitial extracellular matrix ( ECM ) that severely compromises pancreaticresultingin deposition of a high density interstitial extracellular matrix ( ECM ) that severely compromises pancreatic

to uncontrolled mitochondrial proliferationleadsto uncontrolled mitochondrial proliferation

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