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these interactionscan causeneurodegeneration in vivo
I. Cloned Th2 cells modified with DNA methylation inhibitors in vitrocauseautoimmunity in vivo
the passively acquired ABO antibodieswill causein vivo hemolysis
likelyto causein vivo hemolysis
the factcausedby in vivo hemolysis
The phenomenaleadingin vivo hemolysis
The phenomenaleadingto in vivo hemolysis
it is not clearcauseshemolysis in vivo
in vitro * melanoma cell invasioncould influencein vivo ... metastasis
because they are reactive at room temperature but not body temperature and , thereforecausehemolysis in vivo
that , in turn , release biologically active VEGF - Aresultingin angiogenesis in vivo
VEGFcausedin vivo neovascularization
Researchmay resultin vivo v
a certain potentialto influenceosseointegration in vivo
multiple physiological effects , as well as antioxidant activityresultin radioprotection in vivo
by RNAi overexpression(passive) caused byin vivo toxicities
Factorsinfluencingin vivo transduction
eventsresultsin vivo myocyte
by mutations on the surface of the peptide binding pocket , the same mutations that also influenced the in vitro activation of PP2Ai(passive) is influencedin vivo
by mutations on the surface of the peptide binding pocket , the same mutations that also influenced the in vitro activation of PP2Ai(passive) is influenced byin vivo
mutations on the surface of the peptide binding pocket , the same mutations that also influenced the in vitro activation of PP2Ai(passive) is influenced byin vivo
The combination of in vitro , ex vivo , andhas ... contributedin vivo
Studies in the mouse model using both in vitro andhave resultedin vivo
resistant cells generated in vitro ... ableto originatein vivo
Pharmacological inhibition and conditional deletion of Cpt1a in vitro andleadsin vivo
the certain effects of lipid and protein componentsmay be discoveredin vivo
the ability of cellsto causein vivo
Enhanced bone regeneration with a synthetic cell - binding peptideresultsin vivo
cells ... sufficientto causein vivo
Activation of the MAPK pathway via activating EGFR mutationscausesin vivo
The murine cells treated with TNF - in vitro andcausein vivo
Studies involving ectopic expression of SHH in vitro andresultin vivo
the precise effects of lipid and protein componentsmay be discoveredin vivo
the variations ... " using imaging methodsbeing discoveredin vivo
the ability of MAIT cellsto contributein vivo
this method of cell ablation on its owndoes ... causein vivo
these cellsto causein vivo
using ... techniquesdesignedin vivo
Our study ... sufficientto causein vivo
study ... sufficientto causein vivo
their releasecausestheir release
from acquired , hereditary , or iatrogenic conditionscan originatefrom acquired , hereditary , or iatrogenic conditions
from acquired , hereditary , or iatrogenic conditions and is not technique dependentcan originatefrom acquired , hereditary , or iatrogenic conditions and is not technique dependent
alsowill ... causealso
MSC / cartilage interactionsinfluencesMSC / cartilage interactions
MEG2 suppression(passive) caused byMEG2 suppression
to a hepatocellular carcinoma cell line with enhanced tumorigenicity and EpCAM expression , hallmark characteristics of tumor initiating cells ( TIChas ledto a hepatocellular carcinoma cell line with enhanced tumorigenicity and EpCAM expression , hallmark characteristics of tumor initiating cells ( TIC
the periprosthetic tissue gene expression profiles by diminishing the mRNA expression of TNF , IL-1 , CPK and RANKLinfluencedthe periprosthetic tissue gene expression profiles by diminishing the mRNA expression of TNF , IL-1 , CPK and RANKL
to increased melanoma primary tumor growth due to the recruitment of mesenchymal stromal cells as well as the enhanced metastasisleadsto increased melanoma primary tumor growth due to the recruitment of mesenchymal stromal cells as well as the enhanced metastasis
to the improved TIL function and subsequent tumor regression observedcontributedto the improved TIL function and subsequent tumor regression observed
to enhanced antigen - specific T - lymphocyte responsesleadingto enhanced antigen - specific T - lymphocyte responses
neuronal function indirectly as a result of its expression in non - neuronal cell types resulting in cortical dysplasia due to aberrant neuronal migrationmay influenceneuronal function indirectly as a result of its expression in non - neuronal cell types resulting in cortical dysplasia due to aberrant neuronal migration
in enhanced antitumor killing and complete tumor regressionresultedin enhanced antitumor killing and complete tumor regression
to the improved TIL function and subsequent tumor regressioncontributedto the improved TIL function and subsequent tumor regression
subsequent tumor developmentcould influencesubsequent tumor development
to a reduction in tumor growth in immune compromised miceledto a reduction in tumor growth in immune compromised mice
to an immediate and dose dependent early activation of T cellsleadsto an immediate and dose dependent early activation of T cells
to the controlled releaseleadingto the controlled release
to a change in the response of specific substrate receptorswill ... leadto a change in the response of specific substrate receptors
to a tissue - specific induction in DNA repaircan leadto a tissue - specific induction in DNA repair
to a transgene - specific T cellmight ... leadto a transgene - specific T cell
to strong inhibition of NF - B transcriptional activityledto strong inhibition of NF - B transcriptional activity
to elevated plasma homocysteineleadsto elevated plasma homocysteine
the drug response in the hostmay influencethe drug response in the host
dose - dependent inhibition of tumor growthcausesdose - dependent inhibition of tumor growth
T cells(passive) is influencedT cells
to the observed difference in neuronal lossmay contributeto the observed difference in neuronal loss
NO production , which was found to be increased in lupus T cells ( Fig . S1did ... influenceNO production , which was found to be increased in lupus T cells ( Fig . S1
in an up to 86 % reduced tumor volumeresultedin an up to 86 % reduced tumor volume
to a selective elevated expression of TNF in inflamed tissues ( skin , in the present study , or liver , as shown in Kaplan et alleadsto a selective elevated expression of TNF in inflamed tissues ( skin , in the present study , or liver , as shown in Kaplan et al
in decreased complement - dependent cytotoxicityresultingin decreased complement - dependent cytotoxicity
to tissue - specific vascular leakage after immune recognitioncould contributeto tissue - specific vascular leakage after immune recognition
to improved tumour control with adjuvant treatmentmay contributeto improved tumour control with adjuvant treatment
in greater anti - tumor activityresultedin greater anti - tumor activity
This study(passive) was designedThis study
to increased tumor cell growthleadsto increased tumor cell growth
to altered NO productionleadsto altered NO production
better suits the analysis of cytokine production and the effects of inflammation on epileptogenesissettingbetter suits the analysis of cytokine production and the effects of inflammation on epileptogenesis
from mutation or genetic recombinationcan resultfrom mutation or genetic recombination
in extended cellular apoptosisresultsin extended cellular apoptosis