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The cells ... a low oxygen atmosphere ( 0.5 % O2 , 5 % CO2 and 94.5 % N2 ) for the last 6 hours of treatmentto preventhypoxic cytotoxicity
An initial insult at 28 - 30 weeks Placental insufficiently occurring some 3 or 4 weeks before birth ,causinghypoxic ischaemia
the hydrogen content in the air increaseswill causehypoxic asphyxia
were given supplemental oxygento preventhypoxic preconditioning
As is known , puddles will cause the limited availability of oxygen in the soilcausinghypoxic conditions20
claiming a Ca2 + -mediated activation of ion channelsto causehypoxic vasodilatation
cord occlusion(passive) caused byhypoxic ischaemia
repeated coronary artery occlusions in dog heart(passive) caused byHypoxic preconditioning attenuates
decreased rate of blood flow(passive) is caused byHypoxic encephalopathy
oxygen deprivation to the brain(passive) is caused byHypoxic Encephalopathy
2004 ) Role of Akt Signaling in Mitochondrial Survival Pathway(passive) Triggered by2004 ) Role of Akt Signaling in Mitochondrial Survival Pathway
Glaucoma Tolerance | Molecular Medicine | Full Text Glaucoma - Induced Degeneration of Retinal Ganglion Cells(passive) Prevented byGlaucoma Tolerance | Molecular Medicine | Full Text Glaucoma - Induced Degeneration of Retinal Ganglion Cells
Das D. K. Role of akt signaling in mitochondrial survival pathway(passive) triggered byDas D. K. Role of akt signaling in mitochondrial survival pathway
Glaucoma - induced injury of retinal ganglion cell soma and axons(passive) prevented byGlaucoma - induced injury of retinal ganglion cell soma and axons
Axon Injury & Repair , HPAN , Publications Faculty : Jeff Gidday Glaucoma - Induced Degeneration of Retinal Ganglion Cells(passive) Prevented byAxon Injury & Repair , HPAN , Publications Faculty : Jeff Gidday Glaucoma - Induced Degeneration of Retinal Ganglion Cells
The binding of angiotensin II to its … ( More ) Role of Akt signaling in mitochondrial survival pathway(passive) triggered byThe binding of angiotensin II to its … ( More ) Role of Akt signaling in mitochondrial survival pathway
Select item 151842844.Role of Akt signaling in mitochondrial survival pathway(passive) triggered bySelect item 151842844.Role of Akt signaling in mitochondrial survival pathway
to MSC migration and survivalcontributingto MSC migration and survival
17425878 Similar articles Select item 1518428417.Role of Akt signaling in mitochondrial survival pathway(passive) triggered by17425878 Similar articles Select item 1518428417.Role of Akt signaling in mitochondrial survival pathway
long - lasting , endogenous protection in a mouse model of stroke , characterized by reductions in leukocyte - endothelial adherence , inflammation , and infarct volumescreateslong - lasting , endogenous protection in a mouse model of stroke , characterized by reductions in leukocyte - endothelial adherence , inflammation , and infarct volumes
alsopreventsalso
the intracellular modifications(passive) triggered bythe intracellular modifications
the potential paracrine mechanisms induced by CLK cellsmay influencethe potential paracrine mechanisms induced by CLK cells
P - selectin surface expression in platelets under I / R ( Figure 5F ... and this was reversed by peptide P , but not peptide C. SimilarlypreventedP - selectin surface expression in platelets under I / R ( Figure 5F ... and this was reversed by peptide P , but not peptide C. Similarly
cortical infarctionpreventscortical infarction
down - regulation of the adam17 genepreventeddown - regulation of the adam17 gene
apoptosis and enhance MSC survival [ 12–15can preventapoptosis and enhance MSC survival [ 12–15
in no tolerance if it was performed in the presence of TNF - α - neutralizing antibodyresultedin no tolerance if it was performed in the presence of TNF - α - neutralizing antibody
the induction and activation of 5-lipoxygenasepreventsthe induction and activation of 5-lipoxygenase
neuroprotective responses [ 75can triggerneuroprotective responses [ 75
the electrophysiological profile of ASCs on a population levelinfluencedthe electrophysiological profile of ASCs on a population level
in a p38 MAPK - dependent translocation of the HSP27 from the cytosol to the cytoskeleton ... 78resultedin a p38 MAPK - dependent translocation of the HSP27 from the cytosol to the cytoskeleton ... 78
further enhanced neuroprotection(passive) caused byfurther enhanced neuroprotection
the resistant capacity of hPBMNCs to oxidative stresswould ... influencethe resistant capacity of hPBMNCs to oxidative stress
to activation of the JAK / STAT3 signaling pathway ... thus promoting trophoblast cell viability and angiogenesis in PEmay contributeto activation of the JAK / STAT3 signaling pathway ... thus promoting trophoblast cell viability and angiogenesis in PE
in transcriptional changesresultedin transcriptional changes
the induction of Akt kinase in the ventricles during the period of delayed cardioprotection ( 1 - 3 days after preconditioningcausesthe induction of Akt kinase in the ventricles during the period of delayed cardioprotection ( 1 - 3 days after preconditioning
in the stimulation of VEGF , the most potent angiogenic factor known to dateresultedin the stimulation of VEGF , the most potent angiogenic factor known to date
in a lower overall mean oxygen level in the tumour tissueresultedin a lower overall mean oxygen level in the tumour tissue
mitochondrial survival pathways(passive) triggered bymitochondrial survival pathways
the development of ischemic tolerance(passive) triggered bythe development of ischemic tolerance