high blood pressure and a narrowing of the valves of the heart(passive) is ... caused byCardiac hypertrophy
Li , Q. et al . Attenuation of microRNA-1 derepresses the cytoskeleton regulatory protein twinfilin-1to provokecardiac hypertrophy
when the heart responds to increased stresses such as high blood pressure in the body or lungs(passive) is causedCardiac hypertrophy
Select item 2057105318.Attenuation of microRNA-1 derepresses the cytoskeleton regulatory protein twinfilin-1to provokecardiac hypertrophy
View Article Takeda Y , Yoneda T , Demura M , Miyamori I , Mabuchi H. Sodium - Induced Cardiac Aldosterone SynthesisCausesCardiac Hypertrophy
An increase in mechanical load in the heart ... ( heart development , exercise and pregnancy ) or pathologically ( high blood pressure and heart - valve regurgitationcausescardiac hypertrophy
PubMed - indexed for MEDLINE ] Related citations Select item 205710538.Attenuation of microRNA-1 derepresses the cytoskeleton regulatory protein twinfilin-1to provokecardiac hypertrophy
single - point mutations in genes encoding sarcomeric proteins including ventricular myosin regulatory light chain ( RLC(passive) caused bycardiac hypertrophy
Breast Cancer Res Treat , 2013 Jan. PMID 23224145 , Free PMC Article Attenuation of microRNA-1 derepresses the cytoskeleton regulatory protein twinfilin-1to provokecardiac hypertrophy
Circulation 99 : 2694–2701 , 1999.OpenUrlAbstract / FREE Full Text ↵ Takeda Y , Yoneda T , Demura M , Miyamori I , and Mabuchi H. Sodium - induced cardiac aldosterone synthesiscausescardiac hypertrophy
hemodynamic overload , which includes pressure overload ( PO ) caused by aortic stenosis and high blood pressure , and volume overload ( VO ) caused by valvular insufficiency , chronic myocardial infarction , arteriovenous shunt , and pregnancy(passive) is caused byCardiac hypertrophy
| Hypertension Quinapril , an angiotensin converting enzyme inhibitorpreventscardiac hypertrophy
https://doi.org/10.1152/ajpheart.1992.263.1.H177 93 Takeda Y , Yoneda T , Demura M , Miyamori I , Mabuchi H : Sodium - induced cardiac aldosterone synthesiscausescardiac hypertrophy
AB - Hyperactivation of Gq signalingcausescardiac hypertrophy
famous athlete who died from steroids The greatest concerns regarding Clenbuterol side effects will revolve around the heartcan causecardiac hypertrophy
1 - 9 Hypercholesterolemia diets are considered as major sourcesto causecardiac hypertrophy
Cardiomyocyte specific deletion of PP2Acausescardiac hypertrophy
to eventual heart failureleadingto eventual heart failure
to heart failure ( 35leadsto heart failure ( 35
towards heart failureleadingtowards heart failure
to heart failure in miceleadingto heart failure in mice
in heart failure and ventricular arrhythmiascan resultin heart failure and ventricular arrhythmias
to heart failure , which has been characterized extensivelyleadingto heart failure , which has been characterized extensively
to congestive heart failure , arrhythmia , and sudden deathleadsto congestive heart failure , arrhythmia , and sudden death
to the decreased contractility of the heart in heart failurecontributesto the decreased contractility of the heart in heart failure
high blood pressure and congestive heart failurecan causehigh blood pressure and congestive heart failure
to heart failure , which is a leading cause of morbidity and mortalityeasily leadsto heart failure , which is a leading cause of morbidity and mortality
to heart failure , and is induced either by physiological stimulican leadto heart failure , and is induced either by physiological stimuli
to heart failure between 6 – 9 months of ageleadsto heart failure between 6 – 9 months of age
to heart failure between 7–9 months of ageleadsto heart failure between 7–9 months of age
to left ventricular systolic dysfunction and possibly heart failureleadingto left ventricular systolic dysfunction and possibly heart failure
in mitochondrial dysfunction , cardiomyocyte apoptosis , and heart failure ( Kang et al . , 2004 ; Bugger et al . , 2010can resultin mitochondrial dysfunction , cardiomyocyte apoptosis , and heart failure ( Kang et al . , 2004 ; Bugger et al . , 2010
from cardiac specific GLUT4 deletion in mice FEBS J.resultingfrom cardiac specific GLUT4 deletion in mice FEBS J.
to premature death in some casescan leadto premature death in some cases
an acute heart failure and at the same time is followed by decrease in effective adrenergic regulation , ionic transport , power supply and reduction of ability of myofibrils to transform chemical energy of ATP to mechanical energy of muscular contractionpreventsan acute heart failure and at the same time is followed by decrease in effective adrenergic regulation , ionic transport , power supply and reduction of ability of myofibrils to transform chemical energy of ATP to mechanical energy of muscular contraction
to heart disease and/or deathleadingto heart disease and/or death
to a heart attack and deathcan leadto a heart attack and death
from cardiac specific GLUT4 deletion in miceFEBSresultingfrom cardiac specific GLUT4 deletion in miceFEBS
to dilated cardiomyopathy , myocyte apoptosis , and deathleadsto dilated cardiomyopathy , myocyte apoptosis , and death
from chronic hypertensionresultingfrom chronic hypertension
to sudden cardiac death [ 21leadsto sudden cardiac death [ 21
to death Crazybulk 's Clenbutrolleadingto death Crazybulk 's Clenbutrol
from pressure overload and aortic hypertrophy induced by Ang II treatment.15,16resultingfrom pressure overload and aortic hypertrophy induced by Ang II treatment.15,16
heart diseases(passive) caused byheart diseases
from pressure overload , thyroxine , or anemia.10 11 12 The increased DNA synthesis in the surviving part of the ventricles after MIresultingfrom pressure overload , thyroxine , or anemia.10 11 12 The increased DNA synthesis in the surviving part of the ventricles after MI
from increased mTORC1 activation during FLCN deficiencyresultingfrom increased mTORC1 activation during FLCN deficiency
in terminal dilated cardiomyopathyresultedin terminal dilated cardiomyopathy
in a fatal dilated cardiomyopathyresultingin a fatal dilated cardiomyopathy
an increased risk to major cerebrovascular eventscontributesan increased risk to major cerebrovascular events
from a variety of diverse pathologic conditions , including myocardial infarction , hypertension , hypertrophic cardiomyopathy , and valvular regurgitationmay resultfrom a variety of diverse pathologic conditions , including myocardial infarction , hypertension , hypertrophic cardiomyopathy , and valvular regurgitation
to higher mortality risk Kamada et alcan leadto higher mortality risk Kamada et al
from alternative usage of terminal or internal exons , some of which are also developmentally regulated and may be attributable to decreased expression of Fox-1 protein in cardiac hypertrophyresultingfrom alternative usage of terminal or internal exons , some of which are also developmentally regulated and may be attributable to decreased expression of Fox-1 protein in cardiac hypertrophy
from experimental hypertension ( induced either by desoxycorticosterone acetate ( DOCA ) and sodium or by renal artery stenosisresultingfrom experimental hypertension ( induced either by desoxycorticosterone acetate ( DOCA ) and sodium or by renal artery stenosis