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missense mutant myostatincauseshyperplasia
HGH and IGF-1 ... the only substancecan triggerhyperplasia
too much estrogen(passive) to be caused byHyperplasia
unopposed estrogen(passive) caused byhyperplasia
chronic inflammation(passive) may be caused byhyperplasia
tissue inflammation(passive) caused byhyperplasia
Treatment with progestins ( see Hormone Therapy for Endometrial Cancer ) and a dilation and curettage ( D&C ) or hysterectomy ( removing the uteruscan preventhyperplasia
gastrointestinal reflux disease ( GERD ) -- a condition in which stomach contents back up into the esophagus -- or a similar condition called laryngopharyngeal reflux ( LPR(passive) may be caused byadenoid hyperplasia
Biliary Epithelial Hyperplasia Inhibition of the apoptotic or senescent process in the biliary epitheliamay causehyperplasia
a proliferation of melanocytes within the nail matrix or nail bed epithelium(passive) caused byMelanocytic hyperplasia
Chronic hypertrophy / inflammationcauseshyperplasia
estrogen treatment during fetal or neonatal life(passive) caused byhyperplasia
the IGF-1 peptide 's abilityto sparkhyperplasia
due to excess estrogen in the uterus(passive) is causedHyperplasia
covered metallic stentto preventhyperplasia
that HGH is alreadycauseshyperplasia
use of test , HGH , or IGF(passive) can be caused byHyperplasia
IGF levelscausinghyperplasia
anti - inflammation drugs and cytotoxic drugs , which are usedto preventhyperplasia
The primary role of progestin in postmenopausal estrogen therapy ... endometrial protectionto preventhyperplasia
prosthesis and its treatment(passive) caused byHyperplasia
In Drosophila neural stem cell ( NSC ) lineages , excessive Notch signalling results in supernumerary NSCscausinghyperplasia
Exogenous estrogen taken without progesteronewill frequently causehyperplasia
The severity of breast cancer is increased by estrogen ,causehyperplasia
With a combined contraceptive , progestin works against estrogento preventhyperplasia
Satellite cellscausinghyperplasia
fat cellsare created( hyperplasia
gastroesophageal reflux disease ( GERD , the regurgitation and backing up of stomach contents into the esophagus ) and laryngopharyngeal reflux ... LPR , a condition similar to GERD , sometimes called " silent reflux(passive) may be caused byadenoid hyperplasia
What This Relationship Means Because of IGF-1 DES 1 , 3 ’s abilityto createhyperplasia
form the cells it needs to bind with directlyto causehyperplasia
Hgh Canada Genevatropincan causehyperplasia
papilloma virus ( Cheng , U.S. Pat(passive) caused byhyperplasia
GH , and IGF-1 bothcausehyperplasia
as estrogen use with intact uterus requires protective progesteroneto preventhyperplasia
supernumerary stem cellscausinghyperplasia
any woman taking estrogen , who also had a uterus , needed to take progesterone as well ,to preventhyperplasia
stomach acid producing cellscausinghyperplasia
45 years is also known as stomatitis nicotina , smoker ’s palate and inflammationcausinghyperplasia
The condition is also known as stomatitis nicotina , smoker ’s keratosis , smoker ’s palate and inflammationcausinghyperplasia
inflammatory processes that stimulate B - cells(passive) Caused byFollicular hyperplasia •
László F , Durszt F , Julesz J , Petri G. Surgical treatment of Cushing 's disease(passive) caused byLászló F , Durszt F , Julesz J , Petri G. Surgical treatment of Cushing 's disease
to endometrial cancermay leadto endometrial cancer
in the gross enlargement of an organmay resultin the gross enlargement of an organ
new muscle cells and fiberscreatesnew muscle cells and fibers
joint destruction ( 6causingjoint destruction ( 6
in narrowing of the vessel lumen within 3–6 months after angioplastycan resultin narrowing of the vessel lumen within 3–6 months after angioplasty
in the formation of a benign tumormay resultin the formation of a benign tumor
to a higher risk of cancer of the uteruscan leadto a higher risk of cancer of the uterus
obstructive symptoms as judged by continuous mouth breathingcausingobstructive symptoms as judged by continuous mouth breathing
in - stent restenosiscausingin - stent restenosis
in a re - narrowing of the blood vessel , or restenosismay resultin a re - narrowing of the blood vessel , or restenosis
vessel enlargement(passive) was caused byvessel enlargement
from estrogen - alone treatment according to Pfizermay resultfrom estrogen - alone treatment according to Pfizer
a mediastinal hemorrhage due to secondary hyperparathyroidismcauseda mediastinal hemorrhage due to secondary hyperparathyroidism
to chronic airway obstruction resulting in sleep apneamay leadto chronic airway obstruction resulting in sleep apnea
from increased physiologic demands or hormonal stimulationUsually resultsfrom increased physiologic demands or hormonal stimulation
in - stent femoropopliteal restenosis(passive) caused byin - stent femoropopliteal restenosis
of TAg positive cells prior to appearance of advanced tumor stagescomposedof TAg positive cells prior to appearance of advanced tumor stages
a goiter ( McCance & Heuther , 2014causinga goiter ( McCance & Heuther , 2014
to restenosis ( 5 ) , osteo- clast - mediated bone resorption ( 6,7 ) , and retinal neovascularization ( 8)leadingto restenosis ( 5 ) , osteo- clast - mediated bone resorption ( 6,7 ) , and retinal neovascularization ( 8)
to diabetes mellitus , obesity and arterial hypertensioncontributesto diabetes mellitus , obesity and arterial hypertension
to vascular endothelium ... and also related to cholesterol , ergosterolcan ... contributeto vascular endothelium ... and also related to cholesterol , ergosterol
to increased production of mucus and worsening of the symptoms of asthmaleadsto increased production of mucus and worsening of the symptoms of asthma
from decreased apoptosis rather than increased mitosis » and also proposed that « histological assessment of the « apoptotic index » of a tumour several days after the commencement of therapy might , in some cases , provide a useful measure of its response » [ 1might ... resultfrom decreased apoptosis rather than increased mitosis » and also proposed that « histological assessment of the « apoptotic index » of a tumour several days after the commencement of therapy might , in some cases , provide a useful measure of its response » [ 1
from smooth muscle cell proliferation , which may not be the primary mechanism by which restenosis occursresultingfrom smooth muscle cell proliferation , which may not be the primary mechanism by which restenosis occurs
from excessive cell proliferation of smooth muscle cells in the neointimal layer of the blood vesselresultingfrom excessive cell proliferation of smooth muscle cells in the neointimal layer of the blood vessel
in ( D ) clinical sequelae including ( E ) cytokine - induced constitutional symptoms associated with myelofibrosisresultingin ( D ) clinical sequelae including ( E ) cytokine - induced constitutional symptoms associated with myelofibrosis
primary aldosteronism ranges from glomerulosa - like ( idiopathic hyperplasia of the adrenals ) to fasciculata - like ( glucocorticoid - suppressible hyperaldosteronismcausingprimary aldosteronism ranges from glomerulosa - like ( idiopathic hyperplasia of the adrenals ) to fasciculata - like ( glucocorticoid - suppressible hyperaldosteronism
to prostate enlargement that interferes with urination flow Drugs used to treat symptoms slow prostate growth and relax the bladder smooth muscle to make it easier for urine to flow from the bladder through the urethra 15leadingto prostate enlargement that interferes with urination flow Drugs used to treat symptoms slow prostate growth and relax the bladder smooth muscle to make it easier for urine to flow from the bladder through the urethra 15
an increase in the size of a tissue or organcausesan increase in the size of a tissue or organ
to tissue hypoxia , which induces upregulated expression of HIF-1αleadsto tissue hypoxia , which induces upregulated expression of HIF-1α
to increase in left ventricular masscontributesto increase in left ventricular mass
to hypergastrinemialeadsto hypergastrinemia
airway hyperresponsiveness ( AHRcausingairway hyperresponsiveness ( AHR
from smooth muscle cell proliferation , migration , and extracellular matrix depositionresultingfrom smooth muscle cell proliferation , migration , and extracellular matrix deposition
to the growth of the tissueoften leadsto the growth of the tissue
to hypoxia and invasion of immune cellsleadingto hypoxia and invasion of immune cells
an enlargement of these glands , resulting in increased calcium levelscausesan enlargement of these glands , resulting in increased calcium levels
to oedema and hypertrophyhas ledto oedema and hypertrophy
workoutscausingworkouts