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medicationcausinghyperglycemia , hyperuricemia
Removal of graft - bearing intestinal segmentsledtorecurrence of hyperglycemia
also(passive) may ... be discoveredMild hyperglycemia
by diabetes(passive) caused byhyperglycemia ... mellitus
by impaired insulin secretion(passive) caused byT2D. Hyperglycemia
a lack of suppression of glucagoncausespostprandial hyperglycemia
glucocorticoidcauseshyperglycemia from gluconeogenesis
by insulin resistance(passive) caused byPostprandial hyperglycemia
Loss of first - phase insulin responsecan causepostprandial hyperglycemia
both fat and proteincan causepostprandial hyperglycemia
by dietary carbohydrates(passive) caused bypostprandial hyperglycemia
a decrease in additional insulin secretioncausespostprandial hyperglycemia
by early disorder in insulin secretion(passive) caused bypostprandial hyperglycemia
the impairment of(passive) is caused primarily byPostprandial hyperglycemia
As a result of either insulin deficiency or the body 's resistance to insulin , the level of glucose in the bloodstream increasescausinghyperglycemia
either abnormal control of insulin secretion in pancreatic cells or the deterioration of the action of insulin in peripheral tissues(passive) caused byhyperglycemia
by either a lack of insulin ( due to autoimmune destruction of islet cells ) or insulin resistance(passive) caused byhyperglycemia
either a lack of insulin due to autoimmune destruction of islet cells or insulin resistance(passive) caused byhyperglycemia
by excessive hepatic glucose production , a deficiency in insulin secretion(passive) caused byhyperglycemia
having too much glucose and/or not enough insulin in the body(passive) is caused byHyperglycemia
the absence of insufficient production of or deficiency of response to insulincauseshyperglycemia
the absence of insufficient production of or deficiency of response to insulinwill causehyperglycemia
by excessive hepatic glucose production and peripheral insulin resistance(passive) caused byhyperglycemia
excessive hepatic glucose production and peripheral insulin resistance(passive) caused byhyperglycemia
patients with diabetes mellitus , the absence or insufficient production of insulincauseshyperglycemia
but it can not be used for sugar metabolism due to insulin resistant cells in liver , fat and musclesalso causeshyperglycemia
When beta cells fail to produce enough insulin , glucose builds up in the bloodcausinghyperglycemia
the absence of inadequate production of or deficiency of response to insulincauseshyperglycemia
Either impaired glucose utilization or elevated hepatic glucose productioncan causehyperglycemia
an absence of insufficient production or lack of response to insulincauseshyperglycemia
the absence of insufficient production of or lack of response to insulincauseshyperglycemia
the absence of insufficient production or lack of response to insulincauseshyperglycemia
of insufficient production of or lack of response to insulincauseshyperglycemia
the lack of response to insulin or insufficient production of the samecan causehyperglycemia
insufficient production of or lack of response to insulincauseshyperglycemia
insulin deficiency or insulin resistance(passive) caused byhyperglycemia
diabetes , which results from the body being unable to produce enough insulin or unable to use the insulin it produces properly(passive) is caused byHyperglycemia
the absence of insufficient production of or not enough response to insulinwill causehyperglycemia
by relative or absolute deficiency of insulin or peripheral resistance to insulin(passive) caused byhyperglycemia
by either a lack of insulin or insulin resistance(passive) caused byHyperglycemia
to DKAcan leadto DKA
osmotic diuresis ... leading to excessive losses of water , sodium and potassiumcausesosmotic diuresis ... leading to excessive losses of water , sodium and potassium
to microvascular retinal diseaseleadsto microvascular retinal disease
alsocausesalso
from defects in insulin secretion , insulin action , or both diabetic ketoacidosis ( DKAresultingfrom defects in insulin secretion , insulin action , or both diabetic ketoacidosis ( DKA
diabetic complicationscausesdiabetic complications
from defects in insulin secretion , insulin action , or both impaired fasting glucose or impaired glucoseresultingfrom defects in insulin secretion , insulin action , or both impaired fasting glucose or impaired glucose
to overall glucose controlcontributesto overall glucose control
significantly to overall glucose controlcontributessignificantly to overall glucose control
to symptoms or risk of acute hyperglycemic complications ( such as diabetic ketoacidosis and hyperglycemic nonketotic comaleadingto symptoms or risk of acute hyperglycemic complications ( such as diabetic ketoacidosis and hyperglycemic nonketotic coma
from impaired insulin secretion , defects in insulin actionresultingfrom impaired insulin secretion , defects in insulin action
long - term microvascular and macrovascular complications , such as nephropathy , neuropathy , retinopathy , and peripheral vascular diseasemay causelong - term microvascular and macrovascular complications , such as nephropathy , neuropathy , retinopathy , and peripheral vascular disease
to acute metabolic complications , such as diabetic ketoacidosis ( DKAmay leadto acute metabolic complications , such as diabetic ketoacidosis ( DKA
in acute metabolic complications such as : Diabetic Ketoacidosis ( DKAmay resultin acute metabolic complications such as : Diabetic Ketoacidosis ( DKA
in acute metabolic complications such as diabetic ketoacidosis ( DKA ) andmay resultin acute metabolic complications such as diabetic ketoacidosis ( DKA ) and
to poor glycemic controlcan contributeto poor glycemic control
in acute metabolic complications such as diabetic ketoacidosis ( DKA ) and hyperglycemic hyperosmolar nonketotic syndromemay resultin acute metabolic complications such as diabetic ketoacidosis ( DKA ) and hyperglycemic hyperosmolar nonketotic syndrome
long term microvascular and macrovascular complications , such as nephropathy , neuropathy , retinopathy , and atherosclerosismay causelong term microvascular and macrovascular complications , such as nephropathy , neuropathy , retinopathy , and atherosclerosis
long - term microvascular and macrovascular complications , such as nephropathy , neuropathy , retinopathy and atherosclerosiscan causelong - term microvascular and macrovascular complications , such as nephropathy , neuropathy , retinopathy and atherosclerosis
from defects in insulin secretion and insulin actionresultingfrom defects in insulin secretion and insulin action
from defects in insulin secretion or insulin actionresultingfrom defects in insulin secretion or insulin action
in defects in insulin secretion , insulin action , or bothresultingin defects in insulin secretion , insulin action , or both
from defects in insulin action , insulin secretion or bothresultingfrom defects in insulin action , insulin secretion or both
from defects in insulin secretion , insulin action , or bothResultingfrom defects in insulin secretion , insulin action , or both
from defects in insulin secretion , insulin action or bothresultsfrom defects in insulin secretion , insulin action or both
from defects in insulin secretion , insulin action , or eachresultingfrom defects in insulin secretion , insulin action , or each
from defects in insulin secretion , insulin action , orresultingfrom defects in insulin secretion , insulin action , or
from defects in insulin secretion , insulin action , or both A.resultingfrom defects in insulin secretion , insulin action , or both A.
from defects of insulin action insulin secretion or bothresultingfrom defects of insulin action insulin secretion or both
from defects in insulin secretion , insulin actionresultingfrom defects in insulin secretion , insulin action
from the defects of insulin secretion , insulin action or bothresultingfrom the defects of insulin secretion , insulin action or both
oxidative stress via excessive reactive oxygen species ( ROS ) production in the endothelium , which leads to cellular dysfunction and the development of diabetic vascular diseasescausesoxidative stress via excessive reactive oxygen species ( ROS ) production in the endothelium , which leads to cellular dysfunction and the development of diabetic vascular diseases
to the development of cardiovascular diseases and type 2 diabetes [ 1 , 2contributesto the development of cardiovascular diseases and type 2 diabetes [ 1 , 2
to overstimulation of insulinmay leadto overstimulation of insulin
from defects in insulin secretion and insulin action or both [ 1resultingfrom defects in insulin secretion and insulin action or both [ 1
from defects in insulin secretion , insulin action or both [ 1resultingfrom defects in insulin secretion , insulin action or both [ 1
from defects of insulin action , insulin secretion or both [ 1resultingfrom defects of insulin action , insulin secretion or both [ 1
from defects in insulin secretion , insulin action or both [ 3resultingfrom defects in insulin secretion , insulin action or both [ 3
from defects in insulin secretion , insulin action or both [ 1resultsfrom defects in insulin secretion , insulin action or both [ 1
from a defect in insulin action or deficiency in insulin secretion or both leading to alterations in carbohydrate , lipid and protein metabolismresultingfrom a defect in insulin action or deficiency in insulin secretion or both leading to alterations in carbohydrate , lipid and protein metabolism