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breaking the previous record of 2.32 inchessetin1995
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to fatal shock in hypertensive epistaxiscontributingto fatal shock in hypertensive epistaxis
bronchoconstrictioncausedbronchoconstriction
This protection(passive) was also prevented byThis protection
an abrupt blood pressure decrease of 31 ± 4 and 35 ± 4 mmHg , respectivelycausedan abrupt blood pressure decrease of 31 ± 4 and 35 ± 4 mmHg , respectively
significant inhibition of > 50 % of the responsescausedsignificant inhibition of > 50 % of the responses
significantly less hypotension during control conditions ... than in the nitrate - tolerant state ( 99.2±2.9 versus 70.8±1.8 mmcausedsignificantly less hypotension during control conditions ... than in the nitrate - tolerant state ( 99.2±2.9 versus 70.8±1.8 mm
an abrupt 74 ± 14 beat /causedan abrupt 74 ± 14 beat /
both the effects of distal vagal nerve stimulation and carotid occlusion by blocking transmission at the parasympathetic and sympathetic gangliawould preventboth the effects of distal vagal nerve stimulation and carotid occlusion by blocking transmission at the parasympathetic and sympathetic ganglia
in a significant depression of the heart rate , MAP , and RPPresultedin a significant depression of the heart rate , MAP , and RPP
What type of neurological blockade ... hexamethoniumwould ... createWhat type of neurological blockade ... hexamethonium
arterioles and increased flow(passive) caused byarterioles and increased flow
a significant reduction in MBPcauseda significant reduction in MBP
no significant change in subsequent [ 3H]thymidine uptake in response to 2.5 % RScausedno significant change in subsequent [ 3H]thymidine uptake in response to 2.5 % RS
the chemical EPSP(passive) caused bythe chemical EPSP
the effect(passive) being prevented bythe effect
the change in ChBFpreventedthe change in ChBF
alsopreventedalso
lung damage(passive) caused bylung damage