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Qaagi - Book of Why

Causes

Effects

Stimulation of human hematopoietic growth factor responsive cell lines with stem cell factor , interleukin-3 , andleadsgranulocyte - macrophage colony - stimulating factor

Continuous culture of these hematopoietic cells in the presence of the cytokines interleukin-3 , c - kit ligandresultedgranulocyte colony - stimulating factor

a gene translocationresultingin overexpression of colony - stimulating factor ( CSF)1

Professor Don Metcalf at the Walter & Eliza Hall Institute of Medical Research(passive) discovered bygranulocyte colony stimulating factor

Mouse ) Mouse Gaudreau S , Guindi C , Menard M , Besin G , Dupuis GpreventsAmrani A Granulocyte - macrophage colony - stimulating factor

Publications ] Itoh , T.provokesGranulocyte - macrophage colony - stimulating factor

photometric parameters of lightresultingfrom LUMINESCENCE.Receptors , Colony - Stimulating Factor

of ligand - specific alpha - subunits and a common beta - subunit ( betac ) , the major signaling entity(passive) are composedgranulocyte macrophage colony - stimulating factor

Benavides - Garcia , Roberto ; Joachim , Rose ; Pina , Nancy , A ; Mutoji , Kazadi , N ; Reilly , Matthew , A ; Hermann , Brian , P , 2015preventsGranulocyte colony - stimulating factor

for the prevention of clinical recurrences in high risk , disease - free breast cancer patients(passive) is designedgranulocyte - macrophage colony - stimulating factor

dose - limiting neutropenia in non - Hodgkin 's lymphomato preventdose - limiting neutropenia in non - Hodgkin 's lymphoma

in significant improvement of myocardial perfusion 1 week and 1 month after PCIresultedin significant improvement of myocardial perfusion 1 week and 1 month after PCI

to an increment of neutophils via stimulation of bone marrow granulocytic cells [ 19leadsto an increment of neutophils via stimulation of bone marrow granulocytic cells [ 19

reperfusion injurypreventsreperfusion injury

the progression of systemic nonresponsiveness in systemic inflammatory response syndrome and sepsis " , BLOOD , 93(2to preventthe progression of systemic nonresponsiveness in systemic inflammatory response syndrome and sepsis " , BLOOD , 93(2

selective mobilization of bone marrow hematopoietic stem cells into the blood after M phase of the cell cyclecausesselective mobilization of bone marrow hematopoietic stem cells into the blood after M phase of the cell cycle

dose - limiting neutropenia in non - Hodgkin ’s lymphomato preventdose - limiting neutropenia in non - Hodgkin ’s lymphoma

for the treatment of chemotherapy - induced neutropeniaspecially designedfor the treatment of chemotherapy - induced neutropenia

dose - limitng neutropenia in non - Hodgkins lymphomato preventdose - limitng neutropenia in non - Hodgkins lymphoma

Febrile Neutropenia and associated infectionin patients who have received bone marrow – depressing antineoplastics for the treatment of nonmyeloid malignanciesTo preventFebrile Neutropenia and associated infectionin patients who have received bone marrow – depressing antineoplastics for the treatment of nonmyeloid malignancies

cells from the bone marrow to become mature and activatedcausescells from the bone marrow to become mature and activated

increased neutrophil proliferation andcausesincreased neutrophil proliferation and

neutrophiliacan triggerneutrophilia

bacterial infectionsmay preventbacterial infections

focal marrow activitymay causefocal marrow activity

neutrophil recovery and duration of hospitalisation in bone marrow transplantationsignificantly influencesneutrophil recovery and duration of hospitalisation in bone marrow transplantation

kidney infarctionpreventskidney infarction

the build up of Beta - Amyloid plaques in the brain which would be relevant in Alzheimer ’s Diseasemight preventthe build up of Beta - Amyloid plaques in the brain which would be relevant in Alzheimer ’s Disease

the progression of systemic non responsiveness in systemic inflammatory response syndrome and sepsisto preventthe progression of systemic non responsiveness in systemic inflammatory response syndrome and sepsis

to the formation of these cellsmight contributeto the formation of these cells

in mobilization of endothelial progenitor cell populationsresultedin mobilization of endothelial progenitor cell populations

in proliferation and maturation of eosinophilsresultsin proliferation and maturation of eosinophils

in a significant increase of myocardial perfusion within 1 month after PCIresultedin a significant increase of myocardial perfusion within 1 month after PCI

progression of monocrotaline - induced pulmonary arterial hypertension in ratspreventsprogression of monocrotaline - induced pulmonary arterial hypertension in rats

the development of hepatic steatosis in ratspreventsthe development of hepatic steatosis in rats

selective mobilizacausesselective mobiliza

to this condition [ 3leadsto this condition [ 3

marrow hypercellularity which is evident on diffusioncan causemarrow hypercellularity which is evident on diffusion

to increased infiltration of the brain by hematopoietic stem cellledto increased infiltration of the brain by hematopoietic stem cell

to the release of secondary cytokines , including tumor necrosis factor ( TNF ) andleadsto the release of secondary cytokines , including tumor necrosis factor ( TNF ) and

severe infections in patients with severe congenital neutropenias [ 164preventssevere infections in patients with severe congenital neutropenias [ 164

et al A case report of interstitial pneumonia(passive) caused byet al A case report of interstitial pneumonia

loss of spermatogenesispreventsloss of spermatogenesis

to accelerated heart valve deterioration similar to that observed in nondecellularized xenogeneic cardiac bioprosthesesledto accelerated heart valve deterioration similar to that observed in nondecellularized xenogeneic cardiac bioprostheses

to acceleratedheart valve deterioration similar to that observed in nondecellularized xenogeneiccardiac bioprosthesesledto acceleratedheart valve deterioration similar to that observed in nondecellularized xenogeneiccardiac bioprostheses

in tenfold expansionresultedin tenfold expansion

24 hoursresulted24 hours

Capillary leak syndrome ( CLS ) induced by G - CSF : 3 cases report and literature review ] . " 01/01/2011 - " Systemic capillary leak syndrome(passive) caused byCapillary leak syndrome ( CLS ) induced by G - CSF : 3 cases report and literature review ] . " 01/01/2011 - " Systemic capillary leak syndrome

gastric lesions from forming ... suggesting no relationship between MPO activity and the ulcerogenic response to indomethacinpreventedgastric lesions from forming ... suggesting no relationship between MPO activity and the ulcerogenic response to indomethacin

acute arterial occlusioncan causeacute arterial occlusion

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