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the HER2 expressionresultingfrom ERBB2 gene amplification
activated ( greatly increased ) N - myc expressionresultingfrom gene amplification
0003 ] Overexpression of ErbB-2usually resultingfrom erbB-2 gene amplification
where a recurrent rearrangement ,resultingin gene amplification
as a mechanism by which some tumor cell lines can acquire resistance to growth - inhibiting drugs.86(passive) was first discoveredGene amplification
The primersetfor gene amplification
One possible mechanism for PSCA overexpression ismay resultfrom PSCA gene amplification
that tumor promoters tendto causegene amplification
PMID 26733678 , Free PMC Article Increased expression of Solute carrier family 12 member 5 viacontributesgene amplification
expression , or sequencescauseamplification of the gene
According to Werneck et al.[20 ] , increased TOP2A expression does not appearto result solelyfrom gene amplification
increased levels of dihydrofolic acid reductaseresultingfrom gene amplification
Akt1 overexpression andcausedAkt2 gene amplification
S. K. Deng , T. D. Petes , , Mre11-Sae2 and RPA collaborateto preventpalindromic gene amplification
new york Genetic duplications pottsvilleresultingfrom gene amplification
mRNA overexpression of these genesmay resultfrom gene amplification
Overexpresion of the HER2 protein generally ( > 95 %resultsfrom HER2 gene amplification
Deng , S. K. , Yin , Y. , Petes , T. D. & Symington , L. S. Mre11-Sae2 and RPA collaborateto preventpalindromic gene amplification
mutations that occur after a person is born(passive) to be caused byGene amplification events
Site - specific DNA replication at the chorion loci in Drosophila follicle cellsleadsto extensive gene amplification
random overreplication of regions of genomic DNA(passive) caused byGene amplification
an abnormal increase in the copy number of a proto- oncogene i. Ex(passive) Caused byGene Amplification a.
rounds of exposure to a selective agentresultsin gene amplification
the persistence of unrepaired DSBs ... eventsrequiredto triggergene amplification
The lack of increase in the frequency of amplified mutants in the double - deficient cells ... requiredto triggergene amplification
the primary mechanismscausinggene amplification
11 - 6 Alteration of DNA Gene Dosage forexsetGene Amplification
Increased telomerase activitymay resultfrom gene amplification
Some mutants in developmentally expressed genes of plantsresultfrom heritable gene amplification
the multiple recombinational eventsleadingto extensive gene amplification
the repeated initiations be subsequently abortedto preventgene amplification
Selection for fast growth adaptively imprints genomes ,resultingin gene amplification
two potential mechanismsmay have contributedto the gene amplification
PMID 28461336 , Free PMC Article Increased expression of EHF viacontributesgene amplification
promiscuous recombination eventstriggeringgene amplification
Excess Aurora A activityresultingfrom gene amplification
Serum - free subclones were subjected to further selective pressure in glutamine - free media and with increasing concentrations of MSX , an inhibitor of GSresultingin gene amplification
Failure to inhibit re - replicationcan resultin gene amplification
the process of genetic instabilitycausedEGFR gene amplification
the conditionto causegene amplification
ERBB2 overexpression(passive) caused byERBB2 overexpression
in mRNA overexpressionresultedin mRNA overexpression
PIK3CA overexpression(passive) caused byPIK3CA overexpression
in E6AP overexpressionresultingin E6AP overexpression
the overexpression.[13causesthe overexpression.[13
the overexpression.[11causesthe overexpression.[11
an overexpression of PDGFRα(passive) caused byan overexpression of PDGFRα
TMEM16A overexpression(passive) can be caused byTMEM16A overexpression
Overexpression of GLO1(passive) commonly caused byOverexpression of GLO1
to massive overexpressionleadsto massive overexpression
PSCA overexpression(passive) is caused byPSCA overexpression
Overexpression of ErbB2(passive) most often caused byOverexpression of ErbB2
to the overexpression of the geneleadingto the overexpression of the gene
most c - erbB-2 overexpression(passive) is caused bymost c - erbB-2 overexpression
the majority of cases of overexpression(passive) are caused bythe majority of cases of overexpression
Overexpression of Aur - A is thought(passive) to be caused byOverexpression of Aur - A is thought
to HER2 protein overexpressionleadingto HER2 protein overexpression
to the overexpression of the EZH2 geneleadingto the overexpression of the EZH2 gene
the abnormal overexpression of MDM2(passive) caused bythe abnormal overexpression of MDM2
DHFR overexpression [ 8causingDHFR overexpression [ 8
members.1–2 HER2 overexpression(passive) mainly caused bymembers.1–2 HER2 overexpression
that overexpression of MAEL ( show MAEL Antibodies ) ,(passive) caused bythat overexpression of MAEL ( show MAEL Antibodies ) ,
that overexpression of MAEL ( Montrer MAEL Anticorps ) ,(passive) caused bythat overexpression of MAEL ( Montrer MAEL Anticorps ) ,
to the overexpression of FBI-1 mRNAcontributesto the overexpression of FBI-1 mRNA
to overexpression of iASPP in ovarian canercontributesto overexpression of iASPP in ovarian caner
to PXN overexpression in lung tumorscould contributeto PXN overexpression in lung tumors
to overexpression of dihydrofolate reductase ( DHFRleadsto overexpression of dihydrofolate reductase ( DHFR
Approximately 90 % HER2 overexpression breast cancers(passive) are caused byApproximately 90 % HER2 overexpression breast cancers
receptor overexpression and activation of downstream pathwayscausingreceptor overexpression and activation of downstream pathways
to overexpression of this receptor in 20–25 % of breast cancersleadsto overexpression of this receptor in 20–25 % of breast cancers
to overexpression of oncogenes that drive cancer progressioncan leadto overexpression of oncogenes that drive cancer progression
in overexpression ( e.g. , N - Mycresultingin overexpression ( e.g. , N - Myc
to overexpression of the Her2 growth factor receptorleadsto overexpression of the Her2 growth factor receptor
to protein overexpression ... and gain of activity in RGNTsleadsto protein overexpression ... and gain of activity in RGNTs
the overexpression of many mitogens and their particular receptorscausesthe overexpression of many mitogens and their particular receptors
to AR protein overexpression ... and subsequently to increased AR signalingmay leadto AR protein overexpression ... and subsequently to increased AR signaling
to increased mRNA expression and subsequently protein overexpressionleadingto increased mRNA expression and subsequently protein overexpression
to overexpression of HER2 protein on the surface of breast cancer cellsleadsto overexpression of HER2 protein on the surface of breast cancer cells
from a copy number increase associated with overexpression of the amplified generesultsfrom a copy number increase associated with overexpression of the amplified gene
in overexpression and constitutive activation of the encoded receptor tyrosine kinaseresultingin overexpression and constitutive activation of the encoded receptor tyrosine kinase