cholinergic projection pathways ( GABA - ergic mechanismsinfluencescholinergic projection pathways ( GABA - ergic mechanisms

interneurons within the spinal cordinfluencesinterneurons within the spinal cord

from Ca(2+)-permeable AMPA receptor - mediated excitotoxic death of interneuronsresultingfrom Ca(2+)-permeable AMPA receptor - mediated excitotoxic death of interneurons

Bursting(passive) is prevented byBursting

the degradation of GABApreventsthe degradation of GABA

voltage - gatedcalciumchannelsfromopeninginsynaptic knob and presynaptic neuron releases less or no neurotransmitter neural coding the way in which the nervous system converts information to a meaningful pattern of APspreventsvoltage - gatedcalciumchannelsfromopeninginsynaptic knob and presynaptic neuron releases less or no neurotransmitter neural coding the way in which the nervous system converts information to a meaningful pattern of APs

secondary disinhibition ofdopaminergic neuronscausingsecondary disinhibition ofdopaminergic neurons

the development of local competition between right- and left - eye populations in V1 ( Hensch and Stryker , 2004influencesthe development of local competition between right- and left - eye populations in V1 ( Hensch and Stryker , 2004

to the difference in absolute sensitivity and receptive field size at low light levels ... while the connexin36-mediated gap junction coupling plays a minor rolecontributesto the difference in absolute sensitivity and receptive field size at low light levels ... while the connexin36-mediated gap junction coupling plays a minor role

increased extracellular concentrations of 5-HT(passive) caused byincreased extracellular concentrations of 5-HT

in spatially confined inhibition of Ca(2 + ) transients shortly after bAPresultsin spatially confined inhibition of Ca(2 + ) transients shortly after bAP

to the second of a pair of stimulicontributingto the second of a pair of stimuli

effectivelypreventseffectively

in focal dystonia , such as in the case of writer 's cramp.46 This decrease in the amount of GABA is thought to encourage plasticity and perhaps lead to excessive plasticity and functional impairment.47 Mood disordersmay resultin focal dystonia , such as in the case of writer 's cramp.46 This decrease in the amount of GABA is thought to encourage plasticity and perhaps lead to excessive plasticity and functional impairment.47 Mood disorders

to impaired cognition and memorymay leadto impaired cognition and memory

to decrease of the signal during early and persistent phase of LTP , as demonstrated in ( fleadsto decrease of the signal during early and persistent phase of LTP , as demonstrated in ( f

a deficiency in the neurotransmitter level over timecreatesa deficiency in the neurotransmitter level over time

in synaptic GABA accumulationresultsin synaptic GABA accumulation

Both AMPA and NMDA PPF(passive) were influenced byBoth AMPA and NMDA PPF

the proliferative response of human fibroblasts to PDGF without affecting these major signal transduction pathways [ 13preventsthe proliferative response of human fibroblasts to PDGF without affecting these major signal transduction pathways [ 13

in the anticonvulsant activityresultedin the anticonvulsant activity

to the cortical effects since only the drugs with GABA antagonistic activity were able to restore binocularitymay contributeto the cortical effects since only the drugs with GABA antagonistic activity were able to restore binocularity

to the enhanced responses to baclofen observed in hypertensioncontributesto the enhanced responses to baclofen observed in hypertension

the anxietycould causethe anxiety

and resolve pulmonary edema in pre - clinical heart failure animal modelscan preventand resolve pulmonary edema in pre - clinical heart failure animal models

from either a modification of presynaptic mechanisms that regulate neurotransmitter release during SE similar to that observed in models of chronic temporal lobe epilepsy ( Hirsch et al .resultingfrom either a modification of presynaptic mechanisms that regulate neurotransmitter release during SE similar to that observed in models of chronic temporal lobe epilepsy ( Hirsch et al .

to the brain not being able to regulate itself and possible overexcitementwill leadto the brain not being able to regulate itself and possible overexcitement

to increased Pcl and hyperpolarization of neuronal membranesleadingto increased Pcl and hyperpolarization of neuronal membranes

to anxious thoughts , racing mind , and insomniacan leadto anxious thoughts , racing mind , and insomnia

Serotonin agonism(passive) can be prevented bySerotonin agonism

to muscle relaxation and tension relief CBGcould leadto muscle relaxation and tension relief CBG

powerfullyinfluencespowerfully

to alterations in intemeuron - dependent network synchrony in schizophrenia , which may then lead to impaired neuroplasticitymay contributeto alterations in intemeuron - dependent network synchrony in schizophrenia , which may then lead to impaired neuroplasticity

to decreased long - term potentiation ( LTPleadsto decreased long - term potentiation ( LTP

to dreadful diseases like hepatitis , cirrhosisleadsto dreadful diseases like hepatitis , cirrhosis

to a certain specific set of neuronal changes that induce hallucinations in 5 ht 2a receptors or increased activity in certain cortical areas triggering I guess the vague term is " memories " that would be a mixure of experiences combined with current perception which may or may not be related to acetylcholine effectsmay leadto a certain specific set of neuronal changes that induce hallucinations in 5 ht 2a receptors or increased activity in certain cortical areas triggering I guess the vague term is " memories " that would be a mixure of experiences combined with current perception which may or may not be related to acetylcholine effects

coherence and synchrony with time - gatescreatescoherence and synchrony with time - gates

the misaligned spindle(passive) caused bythe misaligned spindle

to its analgesic efficacycontributeto its analgesic efficacy

the hearing and motor skill problems linked to autism spectrum disorder or the emotional imbalance and diminished cognitive function of schizophreniamay createthe hearing and motor skill problems linked to autism spectrum disorder or the emotional imbalance and diminished cognitive function of schizophrenia