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a dimercomposedof two fusion proteins
in order to show itself a number of biological activities , which include activation of macrophages as well as excitement from the T - cell reaction versus cancer(passive) was discoveredThe particular fusion proteins
BCR - ABL1 fusion transcriptsresultin p230 fusion proteins
the Fc domain(passive) caused bygp350 fusion proteins
ALK ) geneleadingto ALK fusion proteins
the pathwaycausesproteolysis of the fusion proteins
by chromosomal translocations in certain forms of leukemia and lymphoma(passive) caused byoncogenic fusion proteins
translocationsresultingin chimeric fusion proteins
of a plant leaf- anchoring peptide and an antimicrobial peptide for pest management(passive) are composedBifunctional fusion proteins ( BiFuProts
Mixed lineage leukemiacauseMLL ) fusion proteins
genesresultin TRK fusion proteins
by two or more head - to - tail ligation coding region that were controlled by the same regulatory sequence(passive) was composed byFusion protein
of the extracellular domain of mouse(passive) is composedfusion protein
membrane - bound complexescomposedof ZAP70 fusion proteins
by Design Expert software(passive) designed byfusion protein
for their affinity(passive) are designedTagged fusion proteins
to optimize expression to high levels , even for components that are difficult to produce as single , non - fused proteins(passive) also can be designedFusion proteins
translocation events ... the ALK geneleadto fusion proteins
by translocations in the or genes as well as(passive) caused bya fusion proteins
the role of Nups in tumorigenesis ... chromosomal rearrangementsresultin oncogenic fusion proteins
Rearrangements in ALK , ROS1 and RET ... all of whichleadto oncogenic fusion proteins
Dr. Ballanceinventedalbumin fusion proteins
translocation / rearrangement eventsleadingto fusion proteins
M - BCR breakpointsresultingin p210 fusion proteins
by the Philadelphia chromosomal translocation(passive) caused bya fusion proteins
the Philadelphia chromosomal translocation(passive) caused bya fusion proteins
This type of variation ... deletions / translocationsresultin fusion proteins
genomic eventsresultin fusion proteins
Our teamhas designedfusion proteins
by this translocation(passive) caused byThe fusion proteins
translocationsleadto fusion proteins
with an affinity sequence(passive) may be ... designedfusion proteins
Chromosomal rearrangements ... intragenic , reciprocal translocationsresultin fusion proteins
those ... Informationcould resultin fusion proteins
This reactionresultsin fusion proteins
by mll rearrangements(passive) caused bythe fusion proteins
by the difference in the junction site of(passive) caused bythe fusion proteins
cancersleadto fusion proteins
the first epitope tagdesignedfor fusion proteins
to have different configurations(passive) can be designedThe fusion proteins
PMP import(passive) caused byPMP import
to a mistargeting to improper locationscan ... leadto a mistargeting to improper locations
in the chromosomal transloca tion tresultingin the chromosomal transloca tion t
in the constitutive activation of ALK tyrosine kinaseresultin the constitutive activation of ALK tyrosine kinase
in aberrant activation of the MAP - Kinase ( MAPK ) pathwayresultin aberrant activation of the MAP - Kinase ( MAPK ) pathway
a rare type of acute leukemiacausea rare type of acute leukemia
in Hox gene activationresultingin Hox gene activation
to inhibitory signaling ( 10leadingto inhibitory signaling ( 10
for affinity purificationDeliberately designedfor affinity purification
in the present studydesignedin the present study
of human EGF ( 6 kDacomposedof human EGF ( 6 kDa
from a fusion between one of the above antibodies and a second peptide or proteinresultfrom a fusion between one of the above antibodies and a second peptide or protein
of various portions of the extracellular domains of human membrane proteins or soluble proteins ( fusion partnerscomposedof various portions of the extracellular domains of human membrane proteins or soluble proteins ( fusion partners
to inactivation of one or both parts of the fusion proteincan leadto inactivation of one or both parts of the fusion protein
from chromosomal translocation ( e.g. , E2A - HLFresultedfrom chromosomal translocation ( e.g. , E2A - HLF
in abnormal functions of cell self - renewal , proliferation , differentiation , and/or apoptosisresultin abnormal functions of cell self - renewal , proliferation , differentiation , and/or apoptosis
to tumor growthmay leadto tumor growth
of an extracellular portion that is usually derived from an antibody and intracellular signaling modules derived from T cell signaling proteinscomposedof an extracellular portion that is usually derived from an antibody and intracellular signaling modules derived from T cell signaling proteins
of a binding regioncomposedof a binding region
of murine CXCL11 or CXCL10composedof murine CXCL11 or CXCL10
in the constitutive activation of the ALK kinase domainresultingin the constitutive activation of the ALK kinase domain
of a 1 Fc areacomposedof a 1 Fc area
of the Fc region ofcomposedof the Fc region of
of a protein or polypeptide to be detected in the cell and the first binding partnercomposedof a protein or polypeptide to be detected in the cell and the first binding partner
in abnormal functions of cell self - renewal , proliferation , differentiationresultin abnormal functions of cell self - renewal , proliferation , differentiation
of target proteins and folding factorscomposedof target proteins and folding factors
of a growth factor receptor signaling domaincomposedof a growth factor receptor signaling domain
the activationresultsthe activation
of a full - length antibody and a fluorescent proteincomposedof a full - length antibody and a fluorescent protein
from chromosomal translocations found in a variety of human cancersmay also resultfrom chromosomal translocations found in a variety of human cancers
in accelerated T - cell reconstitutionresultedin accelerated T - cell reconstitution
from chromosomal translocations associated with acute myeloid leukemiaresultingfrom chromosomal translocations associated with acute myeloid leukemia
cancercausecancer
of enhanced green fluorescent protein ( EGFP ) andcomposedof enhanced green fluorescent protein ( EGFP ) and
in loss of BRAF regulation and activation of the MAP kinase pathwayresultin loss of BRAF regulation and activation of the MAP kinase pathway
to the development of acute leukaemialeadto the development of acute leukaemia
for ( direct ) transcription regulationdesignedfor ( direct ) transcription regulation
of the Fv portion of antibodies directed at receptor proteins on cancer cellscomposedof the Fv portion of antibodies directed at receptor proteins on cancer cells
a block in differentiationcausea block in differentiation
of two copies of an endogenous protein and the Fc region of human IgG1 ( Figure 2composedof two copies of an endogenous protein and the Fc region of human IgG1 ( Figure 2