chromosomal translocations to stimulate transcriptional elongation of HIV-1 and MLL - target genes , respectively(passive) created byfusion proteins
chromosomal translocations affecting the MLL gene at the 11q23 chromosome locus.22 Aberrant fusion proteins involving the MLL histone methyltransferase ( HMT(passive) created byMLL fusion proteins
GST is commonly used as a tagto createfusion proteins
combining two different genes ( or recipes ) to produce one final product(passive) are created byFusion proteins
orderto createLacZ - dockerin fusion proteins
the peptide SIGYPLP and the positive control peptide KKKKKKK upstream of the S11e single - chain Fv ( “ adenobody ” ) directed against the knob domain of the adenovirusto createfusion proteins
inserting the full - length Cul3 sequence into pECFP - C1 or pEYFP - C1 plasmids ( Clontech , Mountain View , CA(passive) were created byCul3 fusion proteins
fusing a soluble TWEAKR to an Fc polypeptide(passive) created bytwo fusion proteins
two genes — originally coding for separate proteins — joining together(passive) are created byFusion proteins
with a cytokine N - terminal domain and the encephalitogenic peptide 69 - 88 of guinea pig myelin basic protein ( GP69 - 88 ) as the C - terminal domain(passive) were designedFusion proteins
containing aEF1 alpha promoter and the myc leader peptideto createfusion proteins
e.g. , translocationsresultingin TRK fusion proteins
to lack thenatural glycine – glycine motif that forms the isopeptidebond with the target lysine side chain , and thus cannotbe cleaved by SUMO peptidases(passive) were designedThe fusion proteins
The ORF of AtPAP1 ( GenBank Accession Number NM_104541.3 ... then fused to the GAL4 activation domain in vector pGADT7-Rec2 ( Clontechto createthe fusion proteins
For example , the expression vector pUR278 ( Ruther et al . , EMBO J. , 2:1791 , 1983 ) can be usedto createlacZ fusion proteins
the Philadelphia chromosomal translocation(passive) caused bya fusion proteins
hadler@u.washington.eduAbstractOne of the most common chromosomal abnormalities in acute leukemia is a reciprocal translocation involving the HRX gene at chromosome locus 11q23resultingin HRX fusion proteins
joined genes(passive) are created byFusion proteins
these rearranged genes(passive) created byThe fusion proteins
chromosomal translocation such as Bcr - Abl(passive) created byfusion proteins
to self - assemble on the cellulose surface , leading into the submonolayer of proteins having the HFBI pointing away from the surface(passive) are designedThe fusion proteins
BioBricks ... being usedto createfusion proteins
Two fragments of the su1 cDNA are usedto createfusion proteins
acidic conditions ... raising the temperature at neutral(passive) are triggered byfusion proteins
through the genetic engineering of two or more genes that originated from separate proteins and impart properties from each of the parent proteins(passive) are createdFusion proteins
The pET28a plasmid was usedto createfusion proteins CBM3a
The pCMV - GH vector ( Laboratory of Dr. Stephen A. Johnston , Department of Biochemistry , The University of Texas , Dallas , Tex . ) containing the human growth hormoneto createfusion proteins
The majority of CML patients ( 95 % ) display M - BCR breakpointsresultingin p210 fusion proteins
merging the ORF of two proteins and removing the stop codon between them(passive) can be created byFusion proteins
the cyan fluorescent protein ... can be usedto createfusion proteins
Recombinant technology is widely usedto createfusion proteins
from chromosomal translocations in cancerresultingfrom chromosomal translocations in cancer
from aberrant chromosomal translocationsresultingfrom aberrant chromosomal translocations
from oncogenic chromosomal translocationsresultingfrom oncogenic chromosomal translocations
from internal deletions or chromosomal translocationsresultingfrom internal deletions or chromosomal translocations
from chromosomal translocations such as BCR - ABLresultingfrom chromosomal translocations such as BCR - ABL
from chromosomal translocations in pediatric AMLsresultingfrom chromosomal translocations in pediatric AMLs
from these variant translocationsresultingfrom these variant translocations
from oncogenic translocationsresultingfrom oncogenic translocations
from chromosomal translocations ( e.g. , Bcr - Ablresultingfrom chromosomal translocations ( e.g. , Bcr - Abl
from chromosomal translocations associated with acute myeloid leukemia [ 3resultingfrom chromosomal translocations associated with acute myeloid leukemia [ 3
from chromosomal translocations associated with acute myeloid leukemia [ 19resultingfrom chromosomal translocations associated with acute myeloid leukemia [ 19
an axon guidance switch that marks the difference between EWSR1 and TAF15 translocated extraskeletal myxoid chondrosarcomas ' , Journal of Pathologytriggeran axon guidance switch that marks the difference between EWSR1 and TAF15 translocated extraskeletal myxoid chondrosarcomas ' , Journal of Pathology
from leukemia - related translocationsresultingfrom leukemia - related translocations
upon translocations at the human MOZ locuscreatedupon translocations at the human MOZ locus
in the present studydesignedin the present study
of N copies of the linked polypeptidescomposedof N copies of the linked polypeptides
from translocations , or products of normal but silent genes ( 1resultingfrom translocations , or products of normal but silent genes ( 1
of the GAD and mutagenized Sml1composedof the GAD and mutagenized Sml1
of codons 234 to 365 of human MEF2Bcomposedof codons 234 to 365 of human MEF2B
in the cytosoloriginatingin the cytosol
from translocations , such as the DNAJB1-PRKACA chimaeric transcript in fibrolamellar HCC.95resultingfrom translocations , such as the DNAJB1-PRKACA chimaeric transcript in fibrolamellar HCC.95
of the GAL4 DNA - binding domain and random segments of E. 88composedof the GAL4 DNA - binding domain and random segments of E. 88
from the chromosomal rearrangement of RETresultingfrom the chromosomal rearrangement of RET
of the terminal 48 or 50 amino acids of mature betacellulin and a binding defective form of Pseudomonas exotoxin ( BTC - TX48 and BTC - TX50 , respectivelycomposedof the terminal 48 or 50 amino acids of mature betacellulin and a binding defective form of Pseudomonas exotoxin ( BTC - TX48 and BTC - TX50 , respectively
of glutathione transferase ( GSTcomposedof glutathione transferase ( GST
in free cytosolic eYFPresultingin free cytosolic eYFP
of segments of nuclear - encoded organelle polypeptides and an enzymatic reportercomposedof segments of nuclear - encoded organelle polypeptides and an enzymatic reporter
of ELPs and Caveolin 1composedof ELPs and Caveolin 1
of Fc regions of IgG1 fused with another moleculecomposedof Fc regions of IgG1 fused with another molecule
of the human being placental isozyme of alkaline phosphatase ( AP ) in the N - terminus and murinecomposedof the human being placental isozyme of alkaline phosphatase ( AP ) in the N - terminus and murine
of two or more fused proteins – proteins that originally functioned independentlyare composedof two or more fused proteins – proteins that originally functioned independently
of three functional parts : secretion signal peptide ( Usp45composedof three functional parts : secretion signal peptide ( Usp45
of a PRLR antagonist ( G129R ) and anti - tumor domains were developedcomposedof a PRLR antagonist ( G129R ) and anti - tumor domains were developed
to sustained T cell activation , and long - term survival of mice with established tumorsleadsto sustained T cell activation , and long - term survival of mice with established tumors
of the A(1)-adenosine receptor and G protein alpha - subunitscomposedof the A(1)-adenosine receptor and G protein alpha - subunits
an axon guidance switch that marks the difference between EWSR1 and TAF15 translocated extraskeletal myxoid chondrosarcomas — Italian Ministry of Health Monica Brenca , Silvia Stacchiotti , Kelly Fassetta , Marta Sbaraglia , Milijana Janjusevic , Dominga Racanelli , Maurizio Polano , Sabrina Rossi , Silvia Brich , Gian Paolo Dagrada , Paola Collini , Chiara Colombo , Alessandro Gronchi , Annalisa Astolfi , Valentina Indio , Maria Abbondanza Pantaleo , Piero Picci , Paolo G Casali , Angelo Paolo Dei Tos , Silvana Pilotti & 1 otherstriggeran axon guidance switch that marks the difference between EWSR1 and TAF15 translocated extraskeletal myxoid chondrosarcomas — Italian Ministry of Health Monica Brenca , Silvia Stacchiotti , Kelly Fassetta , Marta Sbaraglia , Milijana Janjusevic , Dominga Racanelli , Maurizio Polano , Sabrina Rossi , Silvia Brich , Gian Paolo Dagrada , Paola Collini , Chiara Colombo , Alessandro Gronchi , Annalisa Astolfi , Valentina Indio , Maria Abbondanza Pantaleo , Piero Picci , Paolo G Casali , Angelo Paolo Dei Tos , Silvana Pilotti & 1 others
of the NH2-terminal and hydrophobic domain of ten - m1 attached to the alkaline phosphatase reporter genecomposedof the NH2-terminal and hydrophobic domain of ten - m1 attached to the alkaline phosphatase reporter gene
of lactaptin and peptides specific for human tumorscomposedof lactaptin and peptides specific for human tumors
of the A1-Adenosine Receptor and G Protein { alpha}-SubunitsAbstractFulltextPDFWeb of ScienceComposedof the A1-Adenosine Receptor and G Protein { alpha}-SubunitsAbstractFulltextPDFWeb of Science
of canine gnrh and t helper ( th ) cell epitope p35composedof canine gnrh and t helper ( th ) cell epitope p35