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the insertion(passive) caused byan MLL / LAF4 fusion gene
by t(1;19)(q23;p13(passive) caused byE2A - PBX1 fusion gene
A translocationcausinga fusion of the C8orf34 gene
mutation ... <causinggene fusion " EXACT
by t(1;19)(q23;p13(passive) caused byPBX1 fusion gene
by a reciprocal translocation between chromosomes 9 and 22 ( the Philadelphia ( Ph ) chromosome(passive) caused bythe fusion gene
Drs(passive) was designed byThe GCaMP6f fusion gene
the t(9;22)(q34;q11 ) chromosomal translocation(passive) caused bythe fusion gene
by a reciprocal translocation between chromosomes 9 and 22(passive) caused bythe fusion gene
the balanced translocation t(9;22)(q34;q11resultsin the fusion gene
in NSCLC(passive) was discoveredEML4-ALK ) fusion gene
a t(12;22 ) translocation(passive) caused bygene fusion
a little inversion in chromosome 2leadsto a fusion gene
common rearrangementsresultingfrom gene fusion
the genescomposingthe fusion gene
a novel toxin genesresultingfrom gene fusion
a translocationleadingto the fusion gene
The translocationleadsto the ETV6-PDGFRB fusion gene
a chromosomal event called chromothripsis(passive) caused bygene fusion
by a chromosomal event called chromothripsis(passive) caused bya gene fusion
by a chromosomal event called chromothripsis(passive) caused bygene fusion
mutationcausinggene fusion
A mutationcausinggene fusion
DNA damage which results in parts from two separate genes to merge together(passive) is caused byA fusion gene
The translocation involves the PML and RARa ( retinoic acid receptor ) genesresultingin a PMLRARa fusion gene
ABL1 genesleadingto a fusion gene Fusion gene
and ' ' ABL1 ' ' genesleadingto a [ [ fusion gene
by t(9;22)(q34;q11)is(passive) caused bya fusion gene
by a t(7;17)(p15;q21 ) translocation(passive) caused bythe fusion gene
patients and their medical teamsdiscoverthe NTRK - fusion gene
a viral insertion rather thancausesthe gene fusion
Eventsleadingto a fusion gene
by t(9;22)(q34;q11)is thought to have a cooperative effect with EVI1 during the progression of AML and CML.[20(passive) caused bya fusion gene
Numerous studiesdiscoveredgene fusion
by t(9;22)(q34;q11)is thought to have a cooperative effect with EVI1 during the progression of AML and CML(passive) caused bya fusion gene
t(9;22)(q34;q11)is thought to have a cooperative effect with EVI1 during the progression of AML and CML(passive) caused bya fusion gene
co -(passive) was ... discoveredThe fusion gene
by t(9;22)(q34;q11)is thought to have a cooperative effect with EVI1 during the progression of AML and CML.[24(passive) caused bya fusion gene
the MLL rearrangementleadingto the fusion gene
Strand NGS between ALK and EML4 genes(passive) discovered byGene Fusion
from t ( 6;22)(p21;q12resultingfrom t ( 6;22)(p21;q12
to improved clinical outcomes in patients with lung cancerledto improved clinical outcomes in patients with lung cancer
from a mutationresultingfrom a mutation
the production of TRK , a family of proteins that drives tumour growthcausesthe production of TRK , a family of proteins that drives tumour growth
in dominant oncogenic activityresultingin dominant oncogenic activity
the production of TRK , a familycausesthe production of TRK , a family
to the development of solid tumorscan leadto the development of solid tumors
to the development of solid tumors in a variety of tissue typescan leadto the development of solid tumors in a variety of tissue types
from the Philadelphia chromosome rearrangement tresultingfrom the Philadelphia chromosome rearrangement t
to ERG over expressionleadingto ERG over expression
problems in the cell leading to cancercausesproblems in the cell leading to cancer
in measurable fusion protein productiondid ... resultin measurable fusion protein production
to ERG overexpressionleadingto ERG overexpression
ERG overexpression precedes chromosome copy number changes in prostate carcinomascausingERG overexpression precedes chromosome copy number changes in prostate carcinomas
ERG overexpression precedes chromosome copy number changes in prostate carcinomas andcausingERG overexpression precedes chromosome copy number changes in prostate carcinomas and
to ERGleadingto ERG
from the head - to - tail fusion of the Bcr and Abl genes[1resultsfrom the head - to - tail fusion of the Bcr and Abl genes[1
to over - expression of full length ERGleadingto over - expression of full length ERG
Erg Overexpression PrecedesCausingErg Overexpression Precedes
to changes in kinase activityleadingto changes in kinase activity
of a resistance and reporter genecomposedof a resistance and reporter gene
of HSP70 gene and an antigen protein genecomposedof HSP70 gene and an antigen protein gene
to tumor formationcan contributeto tumor formation
of native genetic sequence from two different genescomposedof native genetic sequence from two different genes
to an active enzyme with conserved type IB enzyme functionleadsto an active enzyme with conserved type IB enzyme function
constitutive activation of the tyrosine kinase , which is implicated in uncontrolled cell growth and proliferationcausesconstitutive activation of the tyrosine kinase , which is implicated in uncontrolled cell growth and proliferation
from a specific chromosomal translocationresultingfrom a specific chromosomal translocation
of a 5 sequence from the BCR region and 3 sequence from ABLcomposedof a 5 sequence from the BCR region and 3 sequence from ABL
to constitutive activation of the tyrosine kinaseleadsto constitutive activation of the tyrosine kinase
of a 5 sequence from the BCR regioncomposedof a 5 sequence from the BCR region
from a chromosomal translocation between chromosomes 9 and 12 observed in human leukemiaresultingfrom a chromosomal translocation between chromosomes 9 and 12 observed in human leukemia
from rearrangement of chromosome 4 that has been identified in many cancers including those of the urinary bladderresultingfrom rearrangement of chromosome 4 that has been identified in many cancers including those of the urinary bladder
to deletion ofleadingto deletion of
leukemia in humanscausesleukemia in humans
of human MIS transcriptional regulatory sequences linked to the SV40 T - antigen genecomposedof human MIS transcriptional regulatory sequences linked to the SV40 T - antigen gene
from genomic rearrangements of protein kinasesresultingfrom genomic rearrangements of protein kinases
of parts of two different genescomposedof parts of two different genes
cancers(passive) caused bycancers
in effective expression , processing and stability of messenger RNAresultsin effective expression , processing and stability of messenger RNA
the same pathway [ 1leadsthe same pathway [ 1