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a chromosomal translocation and a solid tumor(passive) created bya fusion gene
a chromosomal translocation and a solid tumour(passive) created bya fusion gene
the CML translocation(passive) created bythe fusion gene
the Ph translocation(passive) created bythe fusion gene
a t(12;22 ) translocation(passive) caused bygene fusion
a chromosomal translocation involving the ALK gene(passive) caused bya gene fusion
most cases of chronic myelogenous leukemia ( CML ... a chromosomal translocationcreatesa fusion gene
or , alternatively , a chromosome translocationcreatesthe PCM1-JAK2 fusion gene
the second translocation shown in D.(passive) created bya fusion gene
The translocation t(1;19)(q23;p13causingTCF3-PBX1 fusion gene
when the translocation joins two otherwise separated genes , an event which is common in cancer(passive) may be createdA fusion gene
when the translocation joins two otherwise - separated genes , the occurrence of which is common in cancer(passive) may be createdA gene fusion
chromosomal translocation , mutated or overexpressedoncogenes , oraberrantgenesidentrfied mgenettcdrseases , such as Huntmgtons drsease ) or foreign genes ( vu - al genes(passive) created byfusion genes
when the translocation joins two otherwise separa(passive) may be createdA fusion gene
translocation of the EML4 and ALK genes(passive) is created bythe EML4-ALK fusion gene
Clonal hypereosinophilia ... a chromosome translocationcreatesthe PCM1-JAK2 fusion gene
a four - way variant translocationoriginatingthe RBM15-MKL1 fusion gene
It involves a chromosome 3p25 and 2q13 translocationcreatinga fusion gene
a four - way varia nt translocationoriginatingthe RBM15-MKL1 fusion gene
The t(14;18 ) translocation ... follicular lymphomacreatesan IGH / Bcl-2 fusion gene
The Philadelphia chromosome outcomes from chromosomal translocation between your gene on chromosome 9 as well as the gene on chromosome 22to createthe fusion gene
the Philadelphia translocation , which occurs in 95 % of CML cases(passive) created bythe gene fusion
Secretory breast carcinomas have a characteristic balanced translocation , t(12;15createsa ETV6-NTRK3 gene fusion
Incorrect : follicular lymphoma ... a t(14;18 ) translocationcreatesa BCL2-IgH fusion gene
In ALL is commonly involved in translocation t(12;21)(p13;q22leadsto the fusion gene
The cryptic translocation t(5;11)(q35;p15.5createsa NSD1-NUP98 fusion gene
when the translocation joins two separate genes ( e.g. , as seen in some cancers(passive) may be createdA gene fusion
the gold standard for LGFMS diagnosis : a balanced translocation t(7;16)(q32–34;p11creatinga fusion gene
a translocation between chromosomes 9 and 22 ... resulting in an abnormal chromosome 22 ( Philadelphia chromosome(passive) is caused byThe BCR‐ABL1 fusion gene
creating the PAX3-FOXO1 fusion gene or less frequently , a translocation between chromosome 1 and 13creatingthe PAX7-FOXO1 fusion gene
an interstitial deletion on chromosome 4q12(passive) is created byThe FIP1L1-PDGFRA fusion gene
unequal crossing - over between a misaligned HBD – HBB gene pair ( fig . 3(passive) was created bythe fusion gene
a chromosomal event called chromothripsis(passive) caused bygene fusion
the ABL1 gene on chromosome 9 to the BCR gene on chromosome 22 ) 2(passive) created bya fusion gene
PDGFRB - associated chronic eosinophilic leukemia , a type of cancer of blood - forming cells , can be caused by a genetic rearrangement known as a translocation that brings together part of the ETV6 gene and part of another gene called PDGFRBcreatingthe ETV6-PDGFRB fusion gene
t(9;22)(q34;q11)is thought to have a cooperative effect with EVI1 during the progression of AML and CML(passive) caused bya fusion gene
identified multimodular proteinsresultingfrom gene fusion
KI - JK | Creative Bioarray CSC - C0676 Established in 1989 from the pleural effusion of a 15-year - old boy with Ki-1 positive lymphoma ( anaplastic large cell lymphoma ; ALCL ) ; carries the translocation t(2;5leadingto NPM1-ALK fusion gene
In this case the reporter gene is directly attached to the gene of interestto createa gene fusion
juxtapositioning the Abl1 gene on chromosome 9 to a part of the BCR ( " breakpoint cluster region " ) gene on chromosome 22(passive) is created bya fusion gene
from a specific chromosomal translocationresultingfrom a specific chromosomal translocation
from chromosomal translocation t ( 9,22resultingfrom chromosomal translocation t ( 9,22
from chromosomal translocation t ( 1,19resultingfrom chromosomal translocation t ( 1,19
from a chromosomal translocation t(11;22 , q24;q12resultingfrom a chromosomal translocation t(11;22 , q24;q12
from the t(9;22)(q34;q11 ) chromosomal translocationresultingfrom the t(9;22)(q34;q11 ) chromosomal translocation
from the chromosomal translocation t ( 4;11resultingfrom the chromosomal translocation t ( 4;11
from a non - random chromosomal translocationresultingfrom a non - random chromosomal translocation
from chromosomal translocation t ( 12,21resultingfrom chromosomal translocation t ( 12,21
from a reciprocal chromosomal translocation ( RCTresultingfrom a reciprocal chromosomal translocation ( RCT
from a f(11;22 ) translocationresultingfrom a f(11;22 ) translocation
from the ( 2;5)(p23;q35 ) chromosomal translocation.2 - 4resultingfrom the ( 2;5)(p23;q35 ) chromosomal translocation.2 - 4
from a t(8;9)(p22;p24 ) chromosomal translocationresultingfrom a t(8;9)(p22;p24 ) chromosomal translocation
from a t(6;20)(q15;q11.2 ) chromosomal translocation in the lymphoma cell line BLUE-1resultingfrom a t(6;20)(q15;q11.2 ) chromosomal translocation in the lymphoma cell line BLUE-1
from the chromosomal translocation t(9;11)(p22;q23resultingfrom the chromosomal translocation t(9;11)(p22;q23
of the BCR and ABL genescomposedof the BCR and ABL genes
from the chromosomal translocation t(X;18)(p11;q11resultingfrom the chromosomal translocation t(X;18)(p11;q11
ERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesionsNeoplasiaYearcausingERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesionsNeoplasiaYear
ERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesionscausingERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesions
lung cancer in 2006could causelung cancer in 2006
from the chromosome inversion inv(2)(p21;p23resultingfrom the chromosome inversion inv(2)(p21;p23
non - small cell lung cancer AURORA , Colo.causednon - small cell lung cancer AURORA , Colo.
to overexpress the 3-hydroxy-3-methylglutaryl coenzyme A ( HMG - CoA ) reductase gene ( hmgTk ) with the glutamate dehydrogenase promoterdesignedto overexpress the 3-hydroxy-3-methylglutaryl coenzyme A ( HMG - CoA ) reductase gene ( hmgTk ) with the glutamate dehydrogenase promoter
as a consequence of the t(9:22)(q34;q11resultsas a consequence of the t(9:22)(q34;q11
from a t(5;9)(q14.1;p24.1resultingfrom a t(5;9)(q14.1;p24.1
to overexpress the 3-hydroxy-3-methylglutaryl coenzyme A ( HMG - CoA ) reductase gene ( hmg(Tk ) ) with the glutamate dehydrogenase promoterdesignedto overexpress the 3-hydroxy-3-methylglutaryl coenzyme A ( HMG - CoA ) reductase gene ( hmg(Tk ) ) with the glutamate dehydrogenase promoter
to prostate cancer Researchers in the U.S.leadsto prostate cancer Researchers in the U.S.
skin cancercausesskin cancer
from a reciprocal translocation between chromosome 9 and 22resultingfrom a reciprocal translocation between chromosome 9 and 22
of the pituitary expression - specific promoter region of the POMC genecomposedof the pituitary expression - specific promoter region of the POMC gene
of exon 1 - 2 of MKLN1 as the 5′-portion and exon 2–21 of MET as the 3′-portion ( Figwas composedof exon 1 - 2 of MKLN1 as the 5′-portion and exon 2–21 of MET as the 3′-portion ( Fig
from a t(1;9)(q24;q34 ) translocationresultingfrom a t(1;9)(q24;q34 ) translocation
from a t(6;20)(q15;q112 ) chromosomal translocation in the lymphoma cell line BLUE-1 PubMed | Institute For Medizinischeresultingfrom a t(6;20)(q15;q112 ) chromosomal translocation in the lymphoma cell line BLUE-1 PubMed | Institute For Medizinische
from the translocation between chromosomes 9 and 22 that underlies the Philadelphia chromosome of chronic myelogenous leukemia ( CMLresultingfrom the translocation between chromosomes 9 and 22 that underlies the Philadelphia chromosome of chronic myelogenous leukemia ( CML
to improved prostate cancer diagnosis “ The prostate cancer gene fusions , and proteins they producemay leadto improved prostate cancer diagnosis “ The prostate cancer gene fusions , and proteins they produce
of CYP57B3 from Aspergillus oryzae andcomposedof CYP57B3 from Aspergillus oryzae and
to ERG over expressionleadingto ERG over expression
from the translocation between chromosomes 9 and 22 that characterizes the Philadelphia chromosome ( Ph ) of chronic myelogenous leukemia ( CMLresultingfrom the translocation between chromosomes 9 and 22 that characterizes the Philadelphia chromosome ( Ph ) of chronic myelogenous leukemia ( CML
of the CYP57B3 gene from Aspergillus oryzae and a cytochrome reductase gene from Saccharomyces cerevisiaecomposedof the CYP57B3 gene from Aspergillus oryzae and a cytochrome reductase gene from Saccharomyces cerevisiae
to over - expression of full length ERG [ 2 ] , [ 3 ] , [ 4leadingto over - expression of full length ERG [ 2 ] , [ 3 ] , [ 4
by juxtaposing the ABL1 gene on chromosome 9 region q34 to a part of the BCR breakpoint cluster region gene on chromosome 22 region q A multi - faceted promoter of DNA mutation in chronic myelogeneous leukemiais createdby juxtaposing the ABL1 gene on chromosome 9 region q34 to a part of the BCR breakpoint cluster region gene on chromosome 22 region q A multi - faceted promoter of DNA mutation in chronic myelogeneous leukemia