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insulin resistancemay contributeto the pathogenesis of polycystic ovary syndrome ( PCOS
Retinol - binding protein 4 ( RPB4 ) , a 21-kDa peptide ... a recently identified adipokinemay contributeto the pathogenesis of polycystic ovary syndrome ( PCOS
Chazenbalk G1 , Trivax BS , Yildiz BO , Bertolotto C , Mathur R , Heneidi S , Azziz R. Adipose tissue dysfunction associated with low - grade chronic inflammation and dysregulation of adipokine secretionmight significantly contributeto the pathogenesis of polycystic ovary syndrome ( PCOS
Several serious medical conditionscan causeoligomenorrhea , including polycystic ovary syndrome ( PCOS
Factorscontributingto potential iron overload in polycystic ovary syndrome ( PCOS
a new brain - body programdesignedspecifically for Polycystic Ovary Syndrome ( PCOS
glycation end - products ( AGEsmay contributeto the pathogenesis of polycystic ovary syndrome ( PCOS
from high testosterone levels in women , ovarian cysts , and high sugar levels high glucose , HA1C or insulinresultsfrom high testosterone levels in women , ovarian cysts , and high sugar levels high glucose , HA1C or insulin
issues like too much testosterone and insulincausesissues like too much testosterone and insulin
from lignans found in flaxseedmay result partlyfrom lignans found in flaxseed
infertilitycan causeinfertility
from adiponectin system dysfunctionmay resultfrom adiponectin system dysfunction
irregular ovulationcausesirregular ovulation
in improved androgen levelsresultedin improved androgen levels
to her excessive weight gainledto her excessive weight gain
her body from releasing eggs on schedulepreventedher body from releasing eggs on schedule