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a 20-fold increase in tPA being released from the allograft and congested viscera , which overwhelms the activity of the plasminogen activator inhibitor ( Virji et al . 1989 ; Porte et al . 1989(passive) is caused byFibrinolysis
Soybean Trypsin Inhibitor is neededto preventfibrinolysis
the A2 proteincould ... influencefibrinolysis
Factor XIIIinfluencesfibrinolysis
by control of PAI-1preventsfibrinolysis
plasmin activitycausingfibrinolysis
the enzyme plasmin(passive) normally caused byfibrinolysis
plasmin formationpreventingFibrinolysis
plasmin generationcausingfibrinolysis
contact with caterpillars : preliminary communication(passive) caused byFibrinolysis
the amount of PAI-1 available ,thereby influencingfibrinolysis
PAI-1 , a prothrombotic enzymepreventsfibrinolysis
elevated levels of PAI-1(passive) caused byfibrinolysis
high PAI-1 in obesity(passive) caused byfibrinolysis
a role ... the main proteincausesfibrinolysis
the circulating thrombin ... to plasmincausingfibrinolysis
Coronory artery • PlasmincausesFibrinolysis
small blood clots drugscausingfibrinolysis
ischemic injury to the donor liver(passive) caused byfibrinolysis
anticoagulant and fibrinolytic drugs , hemophilia , etc(passive) triggered byfibrinolysis
Aprotinin ... the first days in culture after the BAM engineeringto preventfibrinolysis
XP12B , an oral competitive plasmin inhibitorpreventsfibrinolysis
plasminogen - activator inhibitor type-1 ( PAI-1 ) expressionpreventingfibrinolysis
An additional plasma factor correlated with coronary riskmay influencefibrinolysis
it has the unique action of the indirect and direct inhibition of plasminthus preventingfibrinolysis
then this complex can catalyse the conversion of unbound plasminogen to plasminto causefibrinolysis
the quantity and functioning of platelets or coagulation factors ... medical proceduresmay causefibrinolysis
that inhibits the binding of lysine residues on fibrin to plasmin or plasminogenthus preventingfibrinolysis
plasmin ( a serine protease that degrades fibrin ) and the effects of aprotinin ( a serine protease inhibitor ) and bromelain ( an extract from pineapple ) on fibrin clot breakdown(passive) caused byfibrinolysis
supplemented with 200 U / ml aprotinin ( Gerot Pharmaceutica , Vienna , Austriato preventfibrinolysis
the expression of various oncogenes that result in the release of TF and plasminogen activator inhibitor-1 ( PAI-1preventsfibrinolysis
cardio - pulmonary bypass and shed mediastinal blood retransfusion Fibrinolysis caused by cardio - pulmonary bypass and shed mediastinal blood retransfusion Dietrich , W. ; Baranky , A. ; Wendt , P. ; Sternberger , A. ; Blümel , G. ; Spannagl , Michael ; Jochum , Marianne ; Richter , J. A. Dietrich , W. ; Baranky , A. ; Wendt , P. ; Sternberger , A. ; Blümel , G. ; Spannagl , Michael ; Jochum , Marianne ; Richter , J. A. Minimize authors Hörl , Walter H. ; Heidland , A. http://epub.ub.uni-muenchen.de/9336/1/9336.pdf ; Dietrich , W. und Baranky , A. und Wendt , P. und Sternberger , A. und Blümel , G. und Spannagl , Michael und Jochum , Marianne und Richter , J. A. ( 1988(passive) caused byFibrinolysis
the factor XIIa given along with the factor VIIa might not have been completely neutralized andmight have triggeredfibrinolysis
The site in plasminogen where fibrin binds is occupied by TXApreventingfibrinolysis
Other malignancies may enhance the expression of various oncogenes that result in the release of TF and plasminogen activator inhibitor-1 PAI-1whichpreventsfibrinolysis
Recombinant alteplase ... whose releasetriggersfibrinolysis
in particular inhibits urokinase plasminogen activator and tissue plasminogen activatorthereby preventingfibrinolysis
binding of plasminogen and TPA(passive) is triggered byFibrinolysis
Systemic antifibrinolytic agents are widely used in major surgeryto preventfibrinolysis
the enzyme plasminogen ,triggersfibrinolysis
to a decrease in blood viscosity and an improvement in its rheological propertiesleadsto a decrease in blood viscosity and an improvement in its rheological properties
promyelocytes and severe bleeding(passive) caused mainly bypromyelocytes and severe bleeding
to coagulopathy and bleedingcan ... contributeto coagulopathy and bleeding
extensive thrombosis similarcausingextensive thrombosis similar
an excess of fibrin degradation products ( FDPs ) , which initiates excessive bleedingcausesan excess of fibrin degradation products ( FDPs ) , which initiates excessive bleeding
to blood clot dissolutionleadingto blood clot dissolution
in actual clot dissolutionresultsin actual clot dissolution
to the breakdown of blood clotsleadsto the breakdown of blood clots
to the separation of blood clotscan leadto the separation of blood clots
to an increase in D - dimer levels [ 72leadingto an increase in D - dimer levels [ 72
to disseminated intravascular coagulation ( DICleadingto disseminated intravascular coagulation ( DIC
to a shift of hemostatic mechanisms in favor of thrombosisleadingto a shift of hemostatic mechanisms in favor of thrombosis
to production of fibrin degradation products ( FDPs ) which contribute to coagulation by inhibiting fibrin productionleadingto production of fibrin degradation products ( FDPs ) which contribute to coagulation by inhibiting fibrin production
from mediastinal hemorrhageresultingfrom mediastinal hemorrhage
to a hypercoagulable state ( 36–39leadingto a hypercoagulable state ( 36–39
to bleeding tendencyleadingto bleeding tendency
to aggravation of bleedingmay ... leadto aggravation of bleeding
to the breakdown of fibrin clots ( blood clotsleadsto the breakdown of fibrin clots ( blood clots
to a survival mechanism for the parasite in its intravascular environmentcan leadto a survival mechanism for the parasite in its intravascular environment
in severe hemorrhage or hypercoagulation that can result in venous thrombosis and massive pulmonary embolism with cardiovascular collapseresultingin severe hemorrhage or hypercoagulation that can result in venous thrombosis and massive pulmonary embolism with cardiovascular collapse
in a rapid resolution of thrombusresultingin a rapid resolution of thrombus
trauma - induced coagulopathy(passive) caused bytrauma - induced coagulopathy
various bleeding symptoms and allergies(passive) caused byvarious bleeding symptoms and allergies
various bleeding symptoms , and allergy(passive) caused byvarious bleeding symptoms , and allergy
to dissolution of blood clots to form complexes with the plasma , fibrinogen and epinephrine , which have anticoagulant and fibrinolytic actioncontributesto dissolution of blood clots to form complexes with the plasma , fibrinogen and epinephrine , which have anticoagulant and fibrinolytic action
serious bleedings and even deaths due to hemorrhagemay causeserious bleedings and even deaths due to hemorrhage
activation of platelet coagulability leading to vascular occlusion followed by a series of cardiovascular eventscausesactivation of platelet coagulability leading to vascular occlusion followed by a series of cardiovascular events
to decreased consumption of clotting factors causing decreased bleedingleadingto decreased consumption of clotting factors causing decreased bleeding
used in the prevention andtreatment of acute pancreatitis , hemorrhage and disseminated intravascularcoagulation(passive) caused byused in the prevention andtreatment of acute pancreatitis , hemorrhage and disseminated intravascularcoagulation
bloods clots not to form when the person is likely already suffering from internal bleedingis causesbloods clots not to form when the person is likely already suffering from internal bleeding
to an improved hemostasis , an exploratory analysis that examined the effect of TXA on death due to bleeding according to the time to treatmentleadingto an improved hemostasis , an exploratory analysis that examined the effect of TXA on death due to bleeding according to the time to treatment
fibrinogenolysis.• Ancrod is a thrombin - like enzyme that acts directly on fibrinogen , releasing only fibrinopeptide A and fibrin monomers that formcausingfibrinogenolysis.• Ancrod is a thrombin - like enzyme that acts directly on fibrinogen , releasing only fibrinopeptide A and fibrin monomers that form
to hemorrhage by clot lysis , plasmin - mediated cleavage of coagulation factors , and the anticoagulant effect of FDPs , inhibiting platelet function and fibrin polymerizationcontributesto hemorrhage by clot lysis , plasmin - mediated cleavage of coagulation factors , and the anticoagulant effect of FDPs , inhibiting platelet function and fibrin polymerization
unnecessary fibrinogen consumption(passive) caused byunnecessary fibrinogen consumption
in a gradual enzymatic cleavage of fibrin to soluble fragmentsresultsin a gradual enzymatic cleavage of fibrin to soluble fragments
to the degradation of a blood clot resulting in the formation of a fibrin in the blood clotcan leadto the degradation of a blood clot resulting in the formation of a fibrin in the blood clot
to reduced loss of bloodleadingto reduced loss of blood
in a low fibrinogen which can be replaced by one pool of cryoprecipitatewill usually resultin a low fibrinogen which can be replaced by one pool of cryoprecipitate
to degradation products containing the D - dimer antigenleadsto degradation products containing the D - dimer antigen
in ‘ fibrinolytic shutdown ’ [ 10resultingin ‘ fibrinolytic shutdown ’ [ 10