rapid , irreversible ATP depletion(passive) is triggered byExcitotoxic necrosis
immune -mediated vascular damage(passive) is caused byFibrinoid necrosis
Bouvier R , et al Cytomegalovirus infection myasthenia gravis cymbaltacauseureteral necrosis
Ram , Said MH , Bouvier Reductin , et al Cytomegalovirus infectionmay causeureteral necrosis
vascular insufficiency or increasedurinary pressures caused by obstruction(passive) caused byureteral necrosis
Paulo César Koch - Nogueira Cytomegalovirus infectionmay causeureteral necrosis
Mononuclear cells , eosinophils and polymorphonuclear neutrophil leukocytes occur around the vessels or within the vessel wallcausingfibrinoid necrosis
the presence of fiber - like protein substances in arterial walls(passive) is caused byFibrinoid necrosis
Neutrophils are seen surrounding blood vessels and their debris within vessel wallscausingfibrinoid necrosis
the insudation and accumulation of plasma proteins in vessel walls(passive) caused byFibrinoid necrosis
Endothelial injury results in leakage of serum proteins and coagulation factorscausingfibrinoid necrosis
which may affect subchondral bonesto causeosseous necrosis
factors external to the cell or tissue , such as infection , toxins , or trauma which result in the unregulated digestion of cell components(passive) is caused bysciencemag.org ) Necrosis
Visceral fistula as a complication of endoscopic treatment of esophageal and gastric varices using isobutyl-2-cyanoacrylate : report of two casescauses08/01/1986 - " Necrosis
Sterile , granular pink vegetations that are destructivecausingfibrinoid necrosis •
progressing after injury with insertion of a fine needle in muscle(passive) was causedSegmental necrosis
mechanical trauma , damage in blood vessels , ischemia and thermal effects like extremely high or low temperature(passive) is caused byfibrinoid necrosis Necrosis
directly ... macrophage death and likely results from postapoptotic necrosis of these cells in the absence of phagocytic clearance(passive) is caused ... byLesional necrosis ,
an immune response to something happening in the blood vessels(passive) is usually caused byFibrinoid necrosis
while decreasing BPto preventfibrinoid necrosis
the malignant increased blood pressure(passive) Caused byGlomeruli fibrinoid necrosis
a critical imbalance between oxygen supply and demand of the ... Overview(passive) caused byyocardial necrosis
to immediate graft lossleadto immediate graft loss
to the formation of microbleedscontributesto the formation of microbleeds
when there is enough destruction to the vessel walls to allow plasma constituents , including the coagulation factors , to seep into vessel walls or the perivascular tissueresultswhen there is enough destruction to the vessel walls to allow plasma constituents , including the coagulation factors , to seep into vessel walls or the perivascular tissue
to aneurysm formationleadingto aneurysm formation
to perforation in 2 patients aged 10 and 27 yearsleadingto perforation in 2 patients aged 10 and 27 years
to perforation and pericholecystic abscessmay leadto perforation and pericholecystic abscess
to perforation , which may be contained as a localized abscess or progress to diffuse peritonitisleadsto perforation , which may be contained as a localized abscess or progress to diffuse peritonitis
in dark raised lesionsresultingin dark raised lesions
liver damagecausesliver damage
to a cascade of LV remodelling and further deterioration of regional wall motion towards dyskinesis ... implying regional LV remodellingleadsto a cascade of LV remodelling and further deterioration of regional wall motion towards dyskinesis ... implying regional LV remodelling
dryness andto preventdryness and
to plan antimalarials and bubbly brown pusmay leadto plan antimalarials and bubbly brown pus
perilobular fibrosis and interlobular duct distortion with resulting stenosis of the ducts and a classic picture of CP [ 16 , 17 ] ; the stenosis is then thought to be responsible for the difficult outflow of pancreatic secretions with protein precipitation and subsequent calcificationmay causeperilobular fibrosis and interlobular duct distortion with resulting stenosis of the ducts and a classic picture of CP [ 16 , 17 ] ; the stenosis is then thought to be responsible for the difficult outflow of pancreatic secretions with protein precipitation and subsequent calcification