Loading ...
of two sets of Aα- , Bβ- , and γ chains(passive) is composedFibrinogen
Tirofiban hydrochloridepreventsfibrinogen
Greater understanding of the biochemical and biophysical features of FXIII , thrombin , andmay leadfibrinogen
of six polypeptide chains : ( Aα , Bβ , and γ)2(passive) is composedFibrinogen
of six subunits - two each of Aα , Bβ , and γ −(passive) is composedFibrinogen
of six polypeptide chains , two Aα , two Bβ and two γ - chains(passive) is composedFibrinogen
the low - grade inflammation after fiber inhalation(passive) caused byfibrinogen
Blocking of the GP IIb / IIIa receptors with antagonists , such as abciximab , eptifibatide , and tirofibanpreventsfibrinogen
The ebook Красная книга чукотского most cells circulate river reducing due fauna hasresultfibrinogen
organs of fibrin deposition(passive) caused byfibrinogen
of two sets of three different polypeptide chains ( 36 ) : Aα , Bβ , and γ , with A and B designating the N - terminal fibrinopeptides A ( 16 amino acids ) and B ( 14 amino acids ) in the Aα and Bβ chains , respectively(passive) is composedFibrinogen
clot compositionmay influencefibrin(ogen
the liver(passive) is also created byFibrinogen
of 2 Aα- ( purple ) , 2 Bβ- ( teal ) , and 2 γ - chains ( green ) arranged in a trinodular structure with 2 distal D domains and a central E domain(passive) is composedFibrinogen
informatics prediction(passive) designed byfibrinogen
in part(passive) is causedFibrinogen
of several pairs of polypeptide chains such as Aα , Bβ , and γ(passive) is composedFibrinogen
Circutrolcan preventfibrinogen
it needsto createfibrinogen
IIIa Inhibitorspreventsfibrinogen
The venomcausesfibrinogen
of 2Aα , 2Bβ , and 2γ - chains , which are arranged in a dimer of bilateral symmetry(passive) is composedFibrinogen
of α , β , and γ chains(passive) is composedFibrinogen
of three pairs of polypeptide chains called Aα , Bβ and γ(passive) is composedFibrinogen
adipsoe FBS(passive) caused byfibrinogen
environment interactionsinfluencedfibrinogen
of six polypeptides two copies each of three non - identical chains called Aα Bβ and γ(passive) is composedFibrinogen
AGGRASTATpreventsfibrinogen
of three different polypeptide chains or subunits(passive) is composedFibrinogen
of two pairs of polypeptide chains , including the Aα , Bβ , and γ chains , linked by disulfide bonds(passive) is composedFibrinogen
The insertion boundaryto setfibrinogen
of α , β and γ polypeptides(passive) is composedFibrinogen
of two units of three polypeptide chains : Aα Bβ and γ which are assembled with the stoichiometry ( AαBβγ)2 [ 1(passive) is composedFibrinogen
of two polypeptide trimers(passive) is composedFibrinogen
Food plancan influencefibrinogen
trauma or rom chair(passive) can be caused byFibrinogen
The presence of hirudin in the exositepreventsfibrinogen
of _ _ _ ( 1,2 , 3(passive) is composedfibrinogen
battleto leadfibrinogen
of 2 copies each of 3 non - identical chains(passive) is composedFibrinogen
to platelet aggregation 9leadsto platelet aggregation 9
in platelet aggregation.76resultingin platelet aggregation.76
to blood viscosity , platelet aggregation , and fibrin formationcontributesto blood viscosity , platelet aggregation , and fibrin formation
proliferation , vasoconstriction in the damaged sites of vessels wall , stimulation of platelets accumulation , and regulation of cell adhesion.[16causesproliferation , vasoconstriction in the damaged sites of vessels wall , stimulation of platelets accumulation , and regulation of cell adhesion.[16
to blood clots & healingcontributesto blood clots & healing
to atherosclerosis through several mechanisms : 1contributesto atherosclerosis through several mechanisms : 1
fibrin to be producedcausesfibrin to be produced
in an absence of fibrinresultsin an absence of fibrin
fibrin to be laid downcausesfibrin to be laid down
increased platelet aggregation , hyper - coagulation , and excessive blood thickeningcan causeincreased platelet aggregation , hyper - coagulation , and excessive blood thickening
the need to use plasmapheresis [ 18causesthe need to use plasmapheresis [ 18
increased platelet aggregation , hypercoagulation , and excessive blood thickeningcan causeincreased platelet aggregation , hypercoagulation , and excessive blood thickening
to arterial plaque formationcan leadto arterial plaque formation
arterial plaque and blood clotscan causearterial plaque and blood clots
an abnormal blood clotcausingan abnormal blood clot
thrombin formation , platelet aggregation , blood rheology and blood viscosityinfluencesthrombin formation , platelet aggregation , blood rheology and blood viscosity
in the cleavage of only two pairs of small fibrinopeptides , A and B. Occasionally , extrahepatic manifestations , such as cryoglobulinaemia associated with hepatitis Cresultsin the cleavage of only two pairs of small fibrinopeptides , A and B. Occasionally , extrahepatic manifestations , such as cryoglobulinaemia associated with hepatitis C
repair of neurons | Views : 1324preventsrepair of neurons | Views : 1324
to platelet aggregation and a hypercoagulable state that favors the formation of atheromatous plaques.[35contributesto platelet aggregation and a hypercoagulable state that favors the formation of atheromatous plaques.[35
to the formation of insoluble fibrinleadingto the formation of insoluble fibrin
in soluble fibrin monomersresultingin soluble fibrin monomers
to form of fibrin wallleadingto form of fibrin wall
the latter to polymerizecausesthe latter to polymerize
an abnormal arterial blood clotcausingan abnormal arterial blood clot
clot formation thus stopping the bleedingcausesclot formation thus stopping the bleeding
directly to liver regeneration after PHx in micecontributesdirectly to liver regeneration after PHx in mice
small clotsmay causesmall clots
in inhibition of fibrin network formationresultingin inhibition of fibrin network formation
fibrin threads that causes blood to clot 11 Formation of Blood Cellscreatesfibrin threads that causes blood to clot 11 Formation of Blood Cells
to products that enhance coagulationleadingto products that enhance coagulation
clotting ( walls of areacausesclotting ( walls of area
to the development of vascular dysregulation by increasing viscosity , facilitating platelet aggregation , modulating endothelial function , and promoting smooth muscle proliferation and migrationmay contributeto the development of vascular dysregulation by increasing viscosity , facilitating platelet aggregation , modulating endothelial function , and promoting smooth muscle proliferation and migration
to platelet aggregation and phospholipase A2 cleaves lecithin to lysolecithin which can weaken cell membranes and allow for efficient spread of venom.4,10Gilatoxincan leadto platelet aggregation and phospholipase A2 cleaves lecithin to lysolecithin which can weaken cell membranes and allow for efficient spread of venom.4,10Gilatoxin
blood platelets to bind together , initiating abnormal arterial blood clotscan also causeblood platelets to bind together , initiating abnormal arterial blood clots
to blood clots that cause a wide range of health problems including atherosclerosiscan leadto blood clots that cause a wide range of health problems including atherosclerosis
to coagulation being a terminal substrate in plasma clotting , which is cleaved specifically by thrombincontributesto coagulation being a terminal substrate in plasma clotting , which is cleaved specifically by thrombin
blood clotting and increasing fibrinolysiscausesblood clotting and increasing fibrinolysis
to generation of fibrin , which may favor biofilm formationleadingto generation of fibrin , which may favor biofilm formation
in fibrin strands forming that makes grading reactions difficultcan resultin fibrin strands forming that makes grading reactions difficult
to the self - assembly of fibrin molecules into a hydrogel ( 38 , 39leadsto the self - assembly of fibrin molecules into a hydrogel ( 38 , 39