too much dopaminemay causea form of schizophrenia Parkinson disease degeneration of dopamine releasing neurons
the brainresultsin dementia Parkinson s disease degeneration of the dopamine - releasing neurons of the substantia
Environmental and genetic risk factorscontributeto Parkinson 's Disease ( PD ) pathogenesis and the associated midbrain dopamine ( mDA ) neuron loss
the pathwayleadingfrom D2 dopamine receptors to the side effects from the treatment of psychoses & Parkinson 's disease
drugsinfluencethe neurotransmitter dopamine that are routinely taken by people with Parkinson s disease
death of these neurons in SNpccausesdopamine deficiency in the striatum ... leading to Parkinson disease ( PD
Loss of dopaminergic neuronsleadsto the loss of the neurotransmitter dopamine and the onset of Parkinson 's symptoms
The observation stating that it is possibleto influencethe symptoms of Parkinson 's disease and psychosis by modifying dopamine activity
exactly whatcausesthe death of dopamine - releasing cells in Parkinson 's disease
low levels(passive) may be caused bySymptoms of Parkinson 's disease ... dopamine in the brain
Nigral iron accumulation and activation of NMDA receptors ( NRscontributeto the neurodegeneration of dopamine ( DA ) neurons in Parkinson 's disease ( PD
Severe movement disordersresultfrom the loss of striatal dopamine in patients with Parkinson 's disease
by genetic mutations in the LRRK2 gene(passive) caused bythe effects of dopamine loss in Parkinson 's disease
Oxidative StressLeadsto Dopamine Neuron Death in Parkinson 's Disease
Nigral iron accumulation and activation of NMDA receptorscontributeto the neurodegeneration of dopamine neurons in Parkinson 's disease
by a deficiency of dopamine(passive) to be caused byThe Dopamine Theory of Parkinson 's Disease ... believed
motor cortexresultingfrom dopamine depletions in animal models of Parkinson disease
While oxidative processes have been reportedto contributeto dopamine neuron loss in Parkinson 's disease
oxidative stresscontributesto the degeneration of dopamine cell in Parkinson 's disease
Toxicity Excessive exposure or intake ... a neurodegenerative disordercausesDopamine neuronal death and symptoms similar to Parkinson 's disease
an imbalance between the oxidation of dopamine and the availability of antioxidant defenses(passive) may be caused bythe selective degeneration of dopamine neurons in Parkinson 's disease
the molecular eventsleadingto the degeneration of dopamine neurons in Parkinson 's disease
The loss of these nerve cellsleadsto decreased dopamine levels resulting in Parkinson 's disease
nausea and vomitingcausedby taking dopamine agonists ( such as bromocriptine and levodopa ) in Parkinson 's disease
manganesecausesParkinson 's Disease by lowering dopamine levels
manganesecan causeParkinson 's Disease by lowering dopamine levels
manganesemay causeParkinson 's disease by lowering dopamine levels
this regionresultsin being hyperactive in Parkinsons sufferers due to loss of dopamine
too muchleadstoo much
Dopa - Responsive Dystonia ... Evidence Observed in Studies on Segawa Disease(passive) Is Caused byDopa - Responsive Dystonia ... Evidence Observed in Studies on Segawa Disease
Dopa - responsive dystonia ... evidence observed in studies on Segawa disease(passive) is caused by Dopa - responsive dystonia ... evidence observed in studies on Segawa disease
symptoms(passive) caused bysymptoms
dyskinesia(passive) caused bydyskinesia
Scientistshave discoveredScientists
the characteristic tremors and coordination problemscausingthe characteristic tremors and coordination problems
to addictive and impulsive disorders that rapidly resolve upon discontinuation of the medicationmay leadto addictive and impulsive disorders that rapidly resolve upon discontinuation of the medication
motor impairment through aberrant motor learningcausemotor impairment through aberrant motor learning
in slow movementsresultingin slow movements
a decrease in the spontaneous activity of the basal ganglionic circuitcausesa decrease in the spontaneous activity of the basal ganglionic circuit
to the muscle rigidity and tremors associated with the disorderleadingto the muscle rigidity and tremors associated with the disorder
to the development of dopamine replacement therapyledto the development of dopamine replacement therapy
movement to become more and more difficultcausingmovement to become more and more difficult
dyskinesia(passive) caused bydyskinesia
tremor , stiffness and movement slowness in patientscausestremor , stiffness and movement slowness in patients
to rigidity stiffness of movement , tremor and other symptoms and signsleadsto rigidity stiffness of movement , tremor and other symptoms and signs
to rigidity stiffness of movementleadsto rigidity stiffness of movement
to rigidityleadsto rigidity
to rigidity , tremor and other signs and signsleadsto rigidity , tremor and other signs and signs
a 200 increase in dopamine release in the brain , with an alteration in the activity of neurons and a corresponding clinical improvement in motor functionto causea 200 increase in dopamine release in the brain , with an alteration in the activity of neurons and a corresponding clinical improvement in motor function
to rigidity , tremor and different signs and signsleadsto rigidity , tremor and different signs and signs
depressioncan ... causedepression
compulsive sexual behaviormay causecompulsive sexual behavior
Nausea and vomiting(passive) caused byNausea and vomiting
Nausea and vomiting(passive) caused byNausea and vomiting
to muscular tremoursleadingto muscular tremours
to changes in learningcontributeto changes in learning
to the symptoms that it doesleadsto the symptoms that it does
in bradykinesia and tremorresultsin bradykinesia and tremor
in shaking and difficulty with walking , movement , speaking , swallowing and coordinationmay resultin shaking and difficulty with walking , movement , speaking , swallowing and coordination
to a breakdown of that balanceleadto a breakdown of that balance
zone 6 necrosis(passive) caused byzone 6 necrosis
a cerebrovascular accident(passive) caused bya cerebrovascular accident
to excessive and abnormally patterned brain cell electrical activity in both the GPi and the STNleadsto excessive and abnormally patterned brain cell electrical activity in both the GPi and the STN