he foundcausednecrosis of the islet tissue of the pancreas
Glucagoncauseshyperglycaemia within the pancreatic islets
Use of both dietresultedin serious pancreatic islet hyperplasia
Furthermore , at least three additional pathways / interaction networks altered in the adrenal medulla following recurrent hypoglycemia were identified ,may contributeto the impaired epinephrine secretion in HAAF
DPP-4 inhibitors block the degradation of molecules like glucagon - like peptide-1leadingto augmented pancreatic secretion of insulin
A decreased response of the beta cells to gut incretin hormones is thoughtto contributeto the pancreatic endocrine secretory dysfunction
cellsresultingfrom hyperplasia of pancreatic islet
Inflammation of the hypothalamusleadsto defective pancreatic islet
Intraislet endothelial cellscontributeto revascularization of transplanted pancreatic islet
also(passive) was ... influencedEpinephrine - stimulated lipolysis
an enzyme cascadecausesthe hydrolysis of epinephrine
of endocrine cells producing and releasing hormones from secretory granules(passive) is composedThe pancreatic islet of Langerhans
of endocrine cells producing and releasing hormones from secretory granules in response to various stimuli for maintenance of blood(passive) is composedThe pancreatic islet of langerhans
well knownto originatein the pancreatic Islets of Langerhans
of endocrine cells producing and releasing hormones from secretory granules in response to various stimuli for maintenance of blood glucose homeostasis(passive) is composedThe pancreatic islet of Langerhans
Epinephrine ... the adrenal glandresultsin hypersecretion of endogenous epinephrine
Body weight has been foundto influenceepinephrine pharmacokinetics
the researchersdiscoveredthat pancreatic islet
of several cell types(passive) is composedA pancreatic islet
Using animal modelsdiscoveredthat pancreatic islet
of three types of cells of neural crest origin , each with distinct histochemical and electromicroscopic features(passive) is composedThe pancreatic islet
to a diagnosis ofleadsto a diagnosis of
a fall in mean and diastolic pressure with little or no effectcausesa fall in mean and diastolic pressure with little or no effect
a fallcausesa fall
a fall in meancausesa fall in mean
a fall in mean and diastolic pressure with little or no effect on systolic pressurecausesa fall in mean and diastolic pressure with little or no effect on systolic pressure
to hypoglycemia and hyperglycemialeadingto hypoglycemia and hyperglycemia
in severely low blood glucoseresultsin severely low blood glucose
to type 1 diabetesleadingto type 1 diabetes
in severely low bloodresultsin severely low blood
to impaired insulin secretion and type 2 diabetesDoes ... contributeto impaired insulin secretion and type 2 diabetes
to impaired insulin secretionDoes ... contributeto impaired insulin secretion
from the embryonic pancreatic mesenchymeoriginatefrom the embryonic pancreatic mesenchyme
to islet dysfunction in subjects with T2D.contributesto islet dysfunction in subjects with T2D.
tumors to formcausestumors to form
to type 2 diabetesleadto type 2 diabetes
high blood sugar levels in people with Type 2 diabetescauseshigh blood sugar levels in people with Type 2 diabetes
in severely low blood glucose resultsin severely low blood glucose
to the onset of T2DMleadsto the onset of T2DM
problems in how you digest foodcausesproblems in how you digest food
cellsresults cells
to the activation of inflammatory mediatorsleadsto the activation of inflammatory mediators
to impaired insulin secretion and type 2 diabetescontributeto impaired insulin secretion and type 2 diabetes
to cardiovascular actionmight contributeto cardiovascular action
to sensitization of nociceptorscontributesto sensitization of nociceptors
to sensitization of nociceptors after injurycontributesto sensitization of nociceptors after injury
to hepatic glucose productioncontributesto hepatic glucose production
as a primary disease or secondaryresultingas a primary disease or secondary
to loss of insulin secretion and death of insulin - secreting beta cellscontributesto loss of insulin secretion and death of insulin - secreting beta cells
diabetescausediabetes
from endocrine progenitorsoriginatefrom endocrine progenitors
physiological changes in the body , such as constriction of blood vessels , dilation of pupils , increased heart and respiratory rate , and the metabolism of glucosecausesphysiological changes in the body , such as constriction of blood vessels , dilation of pupils , increased heart and respiratory rate , and the metabolism of glucose
immediate reduction in airway inflammationtypically causesimmediate reduction in airway inflammation