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the product ... unlikelyto causeenzyme reaction inhibition
mMcausedsevere enzyme inhibition
hepatic enzyme induction and twocausehepatic enzyme inhibition
damage to proteinscausesenzyme inhibition , denaturation
Such Infectionscan leadto tetracycline enzyme inhibition
by competitive binding at an enzyme 's binding site(passive) is ... caused byEnzyme inhibition
Excess concentrations of various ions associated with salinitycan causeenzyme inhibition
ligand binding to Gi - coupled receptorscausesinhibition of the enzyme
to bind to a specific epitope on the enzymedoes ... causeenzyme inhibition
Activation of enzymes Inhibition of enzymes In therapeutic drugscausinginhibition on enzymes
whereas ligand binding to Gi - coupled receptorscausesinhibition of the enzyme
Subtle effects on proteinresultin enzyme inhibition
This bindingleadsto inhibition of the enzyme
The damage to proteinscan cause enzyme inhibition
by the levels of circulating drugs(passive) caused bythe enzyme inhibition
proteinscausesenzyme inhibition
any drugcausingENZYME INHIBITION
drugcausesthe enzyme inhibition
soluble lignin - derived compoundsmay ... causeenzyme inhibition
by rna interference(passive) caused byenzyme inhibition
1can causeenzyme inhibition
compoundsleadingto enzyme inhibition
its deficiencycausesinhibition of this enzyme
by some drugs(passive) caused byenzyme inhibition
a substance resembling substrate molecule through blocking its active site(passive) caused byEnzyme inhibition
It has been knownto causeinhibition of the [ [ enzyme
ASPIRIN ... other drugscauseENZYME INHIBITION
the substratecausinginhibition of the enzyme
structural and conformational propertiescontributeto enzyme inhibition
the compound ... effectiveto resultin enzyme inhibition
inhibitors binding to a Cys at the mouth of the active siteresultedin enzyme inhibition
Carbamate pesticidescan causeinhibition of the acetylcholinesterase enzyme
by nerve agents(passive) caused byThe enzyme inhibition
nerve agents(passive) caused byThe enzyme inhibition
strong oxidationcausesinhibition of the enzyme
the compound ... for times effectiveto resultin enzyme inhibition
A clear picture of the enzymatic events in cataractogenesis will form the basis of drug developmentto causeselective enzyme inhibition
by the amino acid(passive) provoked bythe enzyme inhibition
by the nerve agent(passive) caused bythe enzyme inhibition
this modificationresultsin enzyme inhibition
compounds ( 20b , 20fto discovercompounds ( 20b , 20f
compounds ( 20b , 20f ) with good lung retention and cell potency that could be taken forward to in - vivo studies where significant target engagement could be demonstratedto discovercompounds ( 20b , 20f ) with good lung retention and cell potency that could be taken forward to in - vivo studies where significant target engagement could be demonstrated
impaired ventilationto causeimpaired ventilation
to drug interactionsleadsto drug interactions
acetaldehyde build up , which leads to unpleasant effectscausesacetaldehyde build up , which leads to unpleasant effects
in increased histone acetylationresultedin increased histone acetylation
levelsresultedlevels
in an increase in the concentration of cAMPresultingin an increase in the concentration of cAMP
to increases in D - serine levels and a reduction in the formation of toxic D - serine oxidation productscan leadto increases in D - serine levels and a reduction in the formation of toxic D - serine oxidation products
in decreased intracellular production of cholesterolresultsin decreased intracellular production of cholesterol
reduced metabolism of drugs and their subsequent accumulation within the bodycan causereduced metabolism of drugs and their subsequent accumulation within the body
a decrease in the accumulation of sorbitol in the cellscausesa decrease in the accumulation of sorbitol in the cells
to decrease in rate of hepatic biotransformation of the drugs causing increased serum concentration andleadsto decrease in rate of hepatic biotransformation of the drugs causing increased serum concentration and
a rise in intracellular levels of cAMP which is believed to result in cell deathcausesa rise in intracellular levels of cAMP which is believed to result in cell death
to an increase in the intracellular concentration of sodium and thus ( by stimulation of sodium - calcium exchangeleadsto an increase in the intracellular concentration of sodium and thus ( by stimulation of sodium - calcium exchange
to an increase in the intracellular concentration of sodium and thus ( by stimulation of sodium - calcium exchange ) an increase in the intracellular concentration of calciumleadsto an increase in the intracellular concentration of sodium and thus ( by stimulation of sodium - calcium exchange ) an increase in the intracellular concentration of calcium
to an increase in the intracellular concentration of sodiumleadsto an increase in the intracellular concentration of sodium
in reduced drug metabolismwill resultin reduced drug metabolism
to inhibition of protein biosynthesisleadsto inhibition of protein biosynthesis
to reduced cholesterol synthesisleadsto reduced cholesterol synthesis
to an increase of hydrophilic and decrease of hydrophobic properties ... leading to a failure of the formation of the active centerleadsto an increase of hydrophilic and decrease of hydrophobic properties ... leading to a failure of the formation of the active center
in abrogation ofresultedin abrogation of
to loss of drug efficacyleadingto loss of drug efficacy
to accumulation of squalene inside the fungal cells , depletion of ergosterolleadsto accumulation of squalene inside the fungal cells , depletion of ergosterol
increased accumulation of SAHcausesincreased accumulation of SAH
in increased transcriptionresultedin increased transcription
increase in concentration , not just lengthcan ... causeincrease in concentration , not just length
growth suppression of the bacteriacausesgrowth suppression of the bacteria
to inhibition of lithium effectsleadsto inhibition of lithium effects
to lower levels of blood cholesterol , fatty acids and lipids , improved insulin sensitivity and reduced formation of glucose in the liverleadsto lower levels of blood cholesterol , fatty acids and lipids , improved insulin sensitivity and reduced formation of glucose in the liver
in increasedresultedin increased
to an increaseleadsto an increase
to toxic levels of reactive oxygen species ( ROS ) which can damage DNA , proteins and lipids inside of the cellleadsto toxic levels of reactive oxygen species ( ROS ) which can damage DNA , proteins and lipids inside of the cell
to decreasedleadsto decreased
to an enhancement in systemic drug exposure andmay leadto an enhancement in systemic drug exposure and
to an enhancement in systemic drug exposuremay leadto an enhancement in systemic drug exposure
to cell deathcould leadto cell death
to increased levels of cGMP dysfunctionleadsto increased levels of cGMP dysfunction
Drug interaction(passive) caused byDrug interaction
to the death of the parasitewould leadto the death of the parasite