DnA Reactive inter - mediatesmay causeenzyme inactivation
a biologically - relevant oxidative post - translational modificationcausesenzyme inactivation
the polymer products(passive) is mainly caused byEnzyme inactivation
Reactive inter - mediatesmay causeenzyme inactivation
alexander flemingdiscoveredenzyme lysozyme
by the ionic liquid(passive) caused byenzyme inactivation
The peroxynitrite - induced nitration of manganese superoxide dismutase ( MnSOD ) tyrosine residuecausesenzyme inactivation
in 1957(passive) was discoveredprevious!This enzyme
h2 light , high temperature and humiditywill causeenzyme inactivation
When aldehyde dehydrogenase reacts with disulfiram ( a drug used in alcoholism therapy ) a thiol group ( A ) is initially modifiedcausingenzyme inactivation
to break down the chemical glue , which binds dead skin cells together(passive) are designedEnzyme exfoliators
changes in the shape of the active sitecan ... causedenaturation of an enzyme
not so highto causedenaturation of the enzyme
the rate of reactioncan causedenaturation of the enzyme
too high of temperaturecan ... causedenaturation of the enzyme
high temperaturecan causedenaturation of the enzyme
Changes in temperature or phcan causedenaturation of the enzyme
the 3-dimensional shape of the enzymecausingenzyme denaturation
the active sitecan ... causedenaturation of an enzyme
techniquescausedenaturation of the enzyme
Oils with high acidity values ( greater than 4 %can causedenaturation of the enzyme
as high temperatures and very low pHcan causedenaturation of an enzyme
clottingdiscoversenzyme thrombin
Others need a neutral pH. Incorrectcan ... leadto the enzyme denaturation
acetone(passive) caused byenzyme denaturation
Factorsinfluencingextracellular enzyme
The ability of ATPto causeenzyme uncoupling
Temperatures outside of this rangecan causedenaturation of the enzyme
The SAPA repeatscauseenzyme oligomerization
an enzyme changescan causedenaturing of the enzyme
of four distinct domains , an N - terminal domain with residues(passive) is composedEnzyme
HHP of more than 300 MPa was reportedto causedenaturation of enzymes
Mutations to the proteasecan resultenzyme
the type of [ [ mutationcausesenzyme
Exogenous addition of high - molecular - weight enzyme orleadenzyme
evolution Scientistsdiscoverenzyme
mutations(passive) may be caused byEnzyme
the sugar part to be broken offcausesthe sugar part to be broken off
to inflammationleadsto inflammation
a loss of its catalytic activitywill causea loss of its catalytic activity
to a deficiency in the lipidleadingto a deficiency in the lipid
its ability to induce mutations in cells due to a size limitation imposed by the dynamic transcription bubbleinfluencesits ability to induce mutations in cells due to a size limitation imposed by the dynamic transcription bubble
in increased susceptibilityresultingin increased susceptibility
production of fats from carbohydratescausesproduction of fats from carbohydrates
to the development of the first electronic bloodleadingto the development of the first electronic blood
to inhibition of DNA synthesis as well as RNA processingleadingto inhibition of DNA synthesis as well as RNA processing
to vasodilationleadsto vasodilation
in decreased enzyme activityresultsin decreased enzyme activity
Silent Reflux(passive) is causedSilent Reflux
duecauseddue
from an N370S mutationresultingfrom an N370S mutation
the fruit & vegetable processing enzymes marketledthe fruit & vegetable processing enzymes market
to inflammationleadsto inflammation
cells to release stored glucose into the bloodstream _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ Digestive enzyme that helps break down proteinscausescells to release stored glucose into the bloodstream _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ _ Digestive enzyme that helps break down proteins
to glycolytic fluxcontributesto glycolytic flux
to the development of the first electronic blood glucose measuring system ( commercialised by Abbott Diagnosticsleadingto the development of the first electronic blood glucose measuring system ( commercialised by Abbott Diagnostics
in a cost acceleration of approximately 106-resultsin a cost acceleration of approximately 106-
to the toxic accumulation of glycogen in the lysosomesleadingto the toxic accumulation of glycogen in the lysosomes
in decreased metabolism and increasedresultingin decreased metabolism and increased
to new treatments for breast , pancreatic cancercould potentially leadto new treatments for breast , pancreatic cancer
to new treatments for breastcould ... leadto new treatments for breast
in the loss of catalytic activityresultingin the loss of catalytic activity
to excessive accumulation of glycogenleadsto excessive accumulation of glycogen
to pro - inflammatory prostaglandins which results in reduced inflammation and its associated painleadsto pro - inflammatory prostaglandins which results in reduced inflammation and its associated pain
Increased uric acid production(passive) is generally caused byIncreased uric acid production
to digest a food resulting in symptomsdesignedto digest a food resulting in symptoms
to inflammation and rednessleadsto inflammation and redness
its substrate absorption and metabolisminfluenceits substrate absorption and metabolism
syndromic intellectual disability with neuromuscular defectscausessyndromic intellectual disability with neuromuscular defects
an accumulation of the enzyme in the ER ) and healthycausesan accumulation of the enzyme in the ER ) and healthy
inability of their body to break down the lactose content in milk productscausesinability of their body to break down the lactose content in milk products
to 30 - 70 % decrease in the activity of enzymeleadsto 30 - 70 % decrease in the activity of enzyme
to the loss of functional conformation of enzymeleadingto the loss of functional conformation of enzyme
to excess dopamine and toxic reactions in the brain and nervous systemleadingto excess dopamine and toxic reactions in the brain and nervous system
to increased NAD+ levels and higher SIRT1 activityleadingto increased NAD+ levels and higher SIRT1 activity
to a pathological excess of acetylcholine in the bodyleadto a pathological excess of acetylcholine in the body
to an accumulation of abnormal glycogen called limit dextrin in affected tissuesleadsto an accumulation of abnormal glycogen called limit dextrin in affected tissues