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Certain medications that treat type 2 diabetescan also causehigh levels of C - peptide
due to a certain class of diabetes drugs known as sulfonylureas(passive) may also be causedHigh levels of c - peptide
ableto causehigh levels of C - peptide
when the body makes insulin(passive) is createdC - peptide
on regular human blood samples(passive) can subsequently be discoveredC - peptide
SMC proliferation and pharmacological inhibition of these pathwayspreventedC - peptide
infusionsetissues | elevated C - peptide
from proinsulin , which is synthesized in the beta - cells of the Islets of Langerhans and cleaved enzymatically releasing insulin and C - peptide(passive) is originatedC - peptide
in 1967(passive) was discoveredC - peptide
Residual -cell function and microvascular complicationsinfluencedbasal C - peptide
insulin resistance and residual β - cell function(passive) influenced byC - peptide (
from a dietary interventionresultingfrom a dietary intervention
from increased β - cell activity observed in hyperinsulinism , from renal insufficiencymay resultfrom increased β - cell activity observed in hyperinsulinism , from renal insufficiency
likelycontributeslikely
high insulin levels and low glucose ( hypoglycemiawill causehigh insulin levels and low glucose ( hypoglycemia
a reduction of type I collagen expression and decreases ... in combination with insulin , receptor activator of nuclear factor - κB ( RANKL ) levelspreventsa reduction of type I collagen expression and decreases ... in combination with insulin , receptor activator of nuclear factor - κB ( RANKL ) levels
from the increased β - cell activity observed in insulinomas ( 3 , 6 , 9may resultfrom the increased β - cell activity observed in insulinomas ( 3 , 6 , 9
to pathogenesis in some FAD casescould contributeto pathogenesis in some FAD cases
proliferation of rat aortic smoothmuscle cells cultured in high - glucose conditionspreventsproliferation of rat aortic smoothmuscle cells cultured in high - glucose conditions
from the increased beta - cell activity observed in insulinomasmay resultfrom the increased beta - cell activity observed in insulinomas
AD office(passive) created byAD office