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Qaagi - Book of Why

Causes

Effects

a bronchodilator inhalerinfluencesthe rate of fatigue development during cycling exercise in moderate to very severe COPD patients

As such , the inclusion of resistance training is now accepted practicewhen designingan exercise programme for patients with HF.33 Training

a bronchodilator inhalerinfluencespulmonary function , gas exchange and exercise tolerance in moderate to severe COPD patients

five items ... activitiescauseshortness of breath in some patients with COPD as previously described.[18

the most important mechanismsleadingto muscle dysfunction and atrophy in COPD patients

the increase in arterial partial pressure of carbon dioxide ( PaCO2resultsfrom use of oxygen - driven nebulisers in patients with stable COPD

Other mechanismscould contributeto the abnormal cardiovascular response to the upright posture in patients with POTS

oxygen therapycan leadto an improvement in exercise endurance in patients with COPD

the skinmay resultin mild to moderate COPD patients : a cross - sectional study

impairmentsinfluencinggait velocity and asymmetry of hemiplegic patients after mild to moderate stroke

Group ( Acomposedof 15 patients who received aerobic exercise in form of treadmill

Factorscontributingto muscle wasting and dysfunction in COPD patients

2 ] Factorscontributingto muscle wasting and dysfunction in COPD patients

these transcriptome dataoriginatedfrom the mild or moderate stage of COPD patients

the transcriptome dataoriginatedfrom the mild or moderate stage of COPD patients

It remains unknown , howeveris contributingto the increased cardiovascular risk in mild - to - moderate COPD patients

2.1 % of casesresultedin moderate paravalvular aortic regurgitation ( with mild regurgitation in 12.5 % of patients

cardiac factorscontributeto exercise limitation in patients with mild to moderate COPD

A black box warning cautionsmay causeacute bronchospasm in patients who have chronic obstructive pulmonary disorder ( COPD

Severe acetylsalicylic acid overdosecausesrespiratory failure in patients with a transition to pulmonary edema

This hyperinflationsignificantly contributesto the shortness of breath experienced by patients with emphysema

Wust R and Degens H. Factorscontributingto muscle wasting and dysfunction in COPD patients

H1N1causingAFOP in a patient with bilateral lung transplantation

lung disease(passive) caused bypatients with mild to moderate hypoxia

to muscleleadingto muscle

to a fast and significant deterioration of the function and symptoms and to increased exacerbationsledto a fast and significant deterioration of the function and symptoms and to increased exacerbations

to superior training outcomes , including improved peak work ratemight leadto superior training outcomes , including improved peak work rate

this program(passive) was designedthis program

the fatality rate to rise very drasticallycausethe fatality rate to rise very drastically

in modest decreases in impairment ... with functional improvement and improved QOLresultedin modest decreases in impairment ... with functional improvement and improved QOL

to the onset of chronic hypoxemia , where the blood contains reduced levels of oxygencould leadto the onset of chronic hypoxemia , where the blood contains reduced levels of oxygen

physical trainingdesignedphysical training

in improvement in exercise endurance , muscle strength , activity levels and quality of lifewould resultin improvement in exercise endurance , muscle strength , activity levels and quality of life

hypercapniacan causehypercapnia

in improvement in exercise endurance , muscle strength , activity levels and quality of life compared with control group having only nutritional counselingwould resultin improvement in exercise endurance , muscle strength , activity levels and quality of life compared with control group having only nutritional counseling

alternative ways to use existing bone or resort to augmenting the patient with autogenous or alloplastic bone materialsto discoveralternative ways to use existing bone or resort to augmenting the patient with autogenous or alloplastic bone materials

to fewer adverse consequences after ischemic strokemight contributeto fewer adverse consequences after ischemic stroke

in modest decreases in impairment ... with functional improvement and improved quality of liferesultedin modest decreases in impairment ... with functional improvement and improved quality of life

in deconditioning of multiple organ systems , including the heart and musclesresultingin deconditioning of multiple organ systems , including the heart and muscles

in deconditioning of multiple organ systems , including the heart and musclesresultingin deconditioning of multiple organ systems , including the heart and muscles

the sensation of severe breathlessness ( dyspnea(passive) caused bythe sensation of severe breathlessness ( dyspnea

a worsening , possibly a lengthening of the Achilles tendon , a condition that is very difficult to treatcan causea worsening , possibly a lengthening of the Achilles tendon , a condition that is very difficult to treat

to get your heart pumpingis designedto get your heart pumping

from abnormal right - handed AsAo helical flowresultingfrom abnormal right - handed AsAo helical flow

alternative ways to use existing bone or resort to augmenting the patient with autogenous or alloplastic bone materialsto discoveralternative ways to use existing bone or resort to augmenting the patient with autogenous or alloplastic bone materials

to an improvement of up to 15pc in mental testsledto an improvement of up to 15pc in mental tests

a spiral of deconditioning due to persistent inactivitycausesa spiral of deconditioning due to persistent inactivity

to a significant enhancement of CPCs migration capacity after the three weeks lasting training program from 113ledto a significant enhancement of CPCs migration capacity after the three weeks lasting training program from 113

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