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The volume overload of the left ventricleleadsto eccentric hypertrophy
In contrast , volume overload in the heart increases the end diastolic pressure and LV wall stress that leads to serial replication of sarcomeresresultingin eccentric hypertrophy
AR AR(passive) caused byLV eccentric hypertrophy
Aortic regurgitation ( ARleadsto eccentric hypertrophy
Endurance exercise training ( EET ) imparts volume loading on the left ventricle ( LVleadingto eccentric hypertrophy
cardiomyopathy : Dilatation of ventricle(s ) with impaired ability to contractleadingto eccentric hypertrophy
Pressure overloadcauseseccentric / concentric hypertrophy
These featuresleadto eccentric hypertrophy
an increase in systolic / diastolic pressure(passive) is caused byEccentric hypertrophy
aerobic exerciseto causeeccentric hypertrophy
left ventricular volumeleadingto eccentric hypertrophy
aerobic training like running marathons , etc(passive) caused byeccentric hypertrophy
typically(passive) is ... triggeredEccentric hypertrophy
high afterloadleadingto eccentric hypertrophy
a volume challenge to the heart ... tendto resultin eccentric hypertrophy
Volume loading lesionleadsto eccentric hypertrophy
mitral regurgitation(passive) caused byinduced eccentric hypertrophy
The accompanying increased preload and enlargement of the ventricleleadsto eccentric hypertrophy
venous return ( preloadcauseseccentric hypertrophy
side of the heartcauseseccentric hypertrophy
Increased diastolic wall stress in response to VO is thoughtto contributeto eccentric hypertrophy
reducing the amount of oxygen available to the cardiac muscle(passive) may also be caused byEccentric hypertrophy
large myocardial infarction , and severe cardiac systolic dysfunctiontriggerseccentric hypertrophy
sereiesleadingto eccentric hypertrophy
Factors that increase the stroke volume such as regurgitant valves , septal defects , or arterial - venous fistulascauseeccentric hypertrophy
Overexpression of Gαq in the heart and the resulting autonomous activation of downstream Gq signaling pathwayscauseseccentric hypertrophy
Chronic MRcausedeccentric LV hypertrophy
The partial loss of cardiomyocyte EGFRledto an eccentric hypertrophy
training with dynamic exerciseleadsto eccentric hypertrophy
Increased systolic wall stress causes path hypertrophy , increased venous returncauseseccentric hypertrophy
eventsleadingto eccentric hypertrophy
because pathological cardiac hypertrophy is known to lead to cardiac failure.23 Converselyresultedin eccentric hypertrophy
Cardiac - Hypertrophy by PD168 Administration Chronic PD168 treatmentresultedin eccentric hypertrophy
What type of pathologiescauseeccentric hypertrophy
The prolonged cardiac adaptation to the pregnancy statecould leadto an eccentric hypertrophy
fibrillar collagen deg- radation , myocardial remodelling and subsequent chamber dilationresultingin eccentric hypertrophy
VO ) modelscausingeccentric hypertrophy
Sarcomeres replicate mainly in seriesresultingin eccentric hypertrophy
coupling and increases cell dimension and sarcomere numbersleadingto eccentric hypertrophy
The Wnt pathwaymay influenceeccentric hypertrophy
from increased circumferential tension due to flow mediated dilation rather than the elevated pressure alone [ 14resultingfrom increased circumferential tension due to flow mediated dilation rather than the elevated pressure alone [ 14
from volume overload states such as aortic or mitral regurgitation and leads to an increase in muscle mass with comparative dilation of the LV chamberresultsfrom volume overload states such as aortic or mitral regurgitation and leads to an increase in muscle mass with comparative dilation of the LV chamber
from situations of volume overload or forms of exerciseresultsfrom situations of volume overload or forms of exercise
to overall cardiac enlargement ( due to myofibril deposition in seriesleadsto overall cardiac enlargement ( due to myofibril deposition in series
from volume overload leading to cardiac myocyte dropoutresultsfrom volume overload leading to cardiac myocyte dropout
to increased oxygen demand by the myocardium which can result in angina or ischemic symptomsleadsto increased oxygen demand by the myocardium which can result in angina or ischemic symptoms
to a decrease in ventricular wall thickness or thinning of the wall with an increase in diastolic volume and wall tensionleadsto a decrease in ventricular wall thickness or thinning of the wall with an increase in diastolic volume and wall tension
in molecular changes occuring in all regions of the heartresultingin molecular changes occuring in all regions of the heart
to an increase in left ventricular cavity volumeleadsto an increase in left ventricular cavity volume
from high output state and volume overloadresultingfrom high output state and volume overload
in left ventricular ( LV ) dilatation and a concurrent elevation in stroke volume in order to compensate for the excessive blood volume delivered to , or remaining in the LV ( Figure 4(bresultsin left ventricular ( LV ) dilatation and a concurrent elevation in stroke volume in order to compensate for the excessive blood volume delivered to , or remaining in the LV ( Figure 4(b
in a length to width ratio of ≈ 11:1can resultin a length to width ratio of ≈ 11:1