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What conditionscausedownbeat nystagmus
lithium toxicity(passive) caused bydownbeat nystagmus
a paramedian ponto - medullary lesion Migration and multiple sclerosis in Alaskan military veterans(passive) caused byDownbeat nystagmus
a paramedian ponto - medullary lesion | springermedizin.de Judith Wagner , Nadine Lehnen , Stefan Glasauer , Nicole Rettinger , Ulrich Büttner , Thomas Brandt(passive) caused byDownbeat nystagmus
The hypometabolism might reflect reduced inhibition or even disinhibition of the circuits to the vestibular nucleithus causingdownbeat nystagmus
lesion of cerebellar flocculus , which in turn resulting in disinhibition of SVN - VTT pathway , followed by relative hyperactivity which drive the upward slow - phase(passive) is usually caused byDownbeat nystagmus
hypomagnesemia , Wernicke ’s encephalopathy , or structural brain lesions(passive) may be caused byDownbeat nystagmus
a bilateral floccular deficiencycausesdownbeat nystagmus ( DBN
a central vestibular imbalance(passive) is caused byDownbeat nystagmus
thiamine deficiency(passive) caused byDownbeat nystagmus
Toxic effects related to medicationmay also causedownbeat nystagmus
imbalance of the activities of the anterior and posterior canals(passive) is caused byDownbeat nystagmus
Some structural lesionsmay causedownbeat nystagmus
cerebellar lesions(passive) caused bydownbeat ' nystagmus
Asymmetry in the vertical smooth pursuit pathway or , imbalance in the neural tone of the central connections of the vertical semicircular canals and otolithscan causedownbeat nystagmus
focal lesion of the brainstem(passive) caused bydownbeat nystagmus
otolithic finast 5 mg lexapro factorsinfluencingdownbeat nystagmus ( DBN
What neurosurgical emergencycausesdownbeat nystagmus
convergence Description(passive) provoked bydownbeat nystagmus
otolithic factorsinfluencingdownbeat nystagmus ( DBN
lesions at the cervicomedullary junction(passive) is caused byDownbeat nystagmus
lesions in sites other than the cerebellar ( para-)floccular lobe(passive) is rarely caused byDownbeat nystagmus ( DBN
Some considered the vertical pursuit asymmetryto causedownbeat nystagmus ( 1284,1287,1288
otolith stimulation(passive) influenced bydownbeat nystagmus
Other diseasescausingdownbeat nystagmus
the rapid eye movements(passive) caused bythe rapid eye movements
odd , involuntary eye movementscausesodd , involuntary eye movements
vision problemscan causevision problems
the eyes to move uncontrollably , usually from side to side , but sometimes up and downcausesthe eyes to move uncontrollably , usually from side to side , but sometimes up and down
involuntary rhythmic shaking of the eyescausesinvoluntary rhythmic shaking of the eyes
from defective axogenesis , dendritogenesis , and neuronal guidance in the areas of the brain which control eye movementsmay ... therefore ... resultfrom defective axogenesis , dendritogenesis , and neuronal guidance in the areas of the brain which control eye movements
my eyes to shift rapidly from side to side in order to find a focal pointcausesmy eyes to shift rapidly from side to side in order to find a focal point
from a cerebellar or cervico - medullary lesion that disrupts projections from the posterior semicircular canal , resulting in tonic upward deviation of the eyes with fast downward corrective movementsoften resultsfrom a cerebellar or cervico - medullary lesion that disrupts projections from the posterior semicircular canal , resulting in tonic upward deviation of the eyes with fast downward corrective movements
you to become dizzycan causeyou to become dizzy
hypoalbuminemiacan also causehypoalbuminemia
The involuntary movements(passive) caused byThe involuntary movements
involuntary eye oscillations(passive) caused byinvoluntary eye oscillations
a reduction in vision , depth perception , coordination and balancecan causea reduction in vision , depth perception , coordination and balance
alsocan ... causealso
The fixation instability(passive) caused byThe fixation instability
severe nausea and vomitingcausessevere nausea and vomiting
Most cases of spasmus nutans begin between age 4 months and 1 yearCausesMost cases of spasmus nutans begin between age 4 months and 1 year
reduced visual acuity or other visual symptomscausesreduced visual acuity or other visual symptoms
vestibular nystagmusartificially causedvestibular nystagmus
The patient ’s(passive) is causedThe patient ’s
a wide range of symptoms including : loss of vision , reduced reading ability , reduced depth perception , difficulty climbing stairs or moving on uneven surfaces , reduced confidence , lack of eye contact , reduced mobility and the inability to drivecausesa wide range of symptoms including : loss of vision , reduced reading ability , reduced depth perception , difficulty climbing stairs or moving on uneven surfaces , reduced confidence , lack of eye contact , reduced mobility and the inability to drive
the visual deficits(passive) caused bythe visual deficits
uncontrolled repetitive rapid oscillation of the eyecausesuncontrolled repetitive rapid oscillation of the eye
the reduction in visual acuity(passive) caused bythe reduction in visual acuity
pronounced eye movement , which in turn causes his head to move involuntarily to compensatecausespronounced eye movement , which in turn causes his head to move involuntarily to compensate
from changes in position of the head or head motionsresultingfrom changes in position of the head or head motions
from unilateral vestibular excitationresultingfrom unilateral vestibular excitation
vision to vary during the daymay causevision to vary during the day
the visual defects(passive) caused bythe visual defects
from damageresultingfrom damage
from a medical condition , such as head traumacan resultfrom a medical condition , such as head trauma
your physiotherapist to the suspicion that the vestibular system is the culpritleadsyour physiotherapist to the suspicion that the vestibular system is the culprit
his damaged optic nerves(passive) caused byhis damaged optic nerves
balance issuescan causebalance issues
uncontrollable eye movementcausesuncontrollable eye movement
from a relative excess of output from the anterior canals ( purple ) , which can be a consequence of decreased cerebellar flocculus inhibitory output ( dark red ) , or decreased posterior canal output ( orange ) ( e.g. from a cervicomedullary lesioncan resultfrom a relative excess of output from the anterior canals ( purple ) , which can be a consequence of decreased cerebellar flocculus inhibitory output ( dark red ) , or decreased posterior canal output ( orange ) ( e.g. from a cervicomedullary lesion
from unilateral vestibular inhibitionresultingfrom unilateral vestibular inhibition
from a UVP hyperactivity ( secondary to a floccular lesion ) and the notion that this pathway is physiologically predominant compared to the downward pathway , maybe due to gravity , is developedcould resultfrom a UVP hyperactivity ( secondary to a floccular lesion ) and the notion that this pathway is physiologically predominant compared to the downward pathway , maybe due to gravity , is developed
the difficulties(passive) caused bythe difficulties
due to poor vision for any reason since childhoodis ... causeddue to poor vision for any reason since childhood