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Accumulation of β2 microglobulincausesdialysis amyloidosis
sufficient permeation of β2-microglobulin ( molecular weight : 11,800 ) which is knownto causedialysis amyloidosis
subacute zinc toxicity(passive) caused byDialysis anaemia
2-microglobulin ( molecular weight : 11,800 ) which is knownto causedialysis amyloidosis
the accumulation of amyloid protein(passive) is caused byDIALYSIS AMYLOIDOSIS
chronic kidney failure(passive) caused bydialysis patients with anemia
CKD.FibroGen China(passive) caused bydialysis patients with anemia
uremic low - molecular - weight proteins(passive) is caused bydialysis amyloidosis
the factorsinfluencehemoglobin level in dialysis patients
chronic kidney disease(passive) caused bydialysis patients with anemia
from binding of glucose with hemoglobin in red blood cellsresultsfrom binding of glucose with hemoglobin in red blood cells
from glucose binding to hemoglobin in red blood cellsresultsfrom glucose binding to hemoglobin in red blood cells
in a 17 % increase in the Fischer ratio in patients with alcoholic hepatitis and a 3 % decrease in patients with resistant pruritus ( p < 0.05resultedin a 17 % increase in the Fischer ratio in patients with alcoholic hepatitis and a 3 % decrease in patients with resistant pruritus ( p < 0.05
these accumulations in patients with fulminant WDto preventthese accumulations in patients with fulminant WD
from hemodilution population of cardiac surgery patientsresultingfrom hemodilution population of cardiac surgery patients
to mortalitycould leadto mortality
the ratio of absorbance at 675 nm over 800 nm , a measure(passive) is influenced bythe ratio of absorbance at 675 nm over 800 nm , a measure
in a decrease of pruritus ( VASresultedin a decrease of pruritus ( VAS
Subsequent maxima in this peak(passive) are created bySubsequent maxima in this peak
microthrombi formation and then infarctioncould causemicrothrombi formation and then infarction