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Qaagi - Book of Why

Causes

Effects

Shorter inspiratory timecontributesto dead space ventilation

Cardiovascular insufficiency , like shock or CPRleadsto dead space ventilation

may enlarge Zone 1thus leadingto dead space ventilation

peak flow of 240 LPM for effective respiratory support in NIV Proximal flow sensorpreventsdead space ventilation

the length of tubing in a breathing apparatus ( Carlione , 2019(passive) caused bydead space ventilation

The first portion of the exhaled breathis composedof dead - space ventilation

Tachypnoea developscausingdead - space ventilation and alveolar hypoventilation

Obstruction of the pulmonary arteriescreatesdead space ventilation

Obstruction of the pulmonary flowcreatesdead space ventilation

rapid , shallow breathing(passive) caused bydead space ventilation

the pig ... the MR scannercreateddead space in the ventilation system

vascular obstructionleadingto dead space ventilation

continuous ventilationmight leadto dead - space ventilation

a process associated with the dissipation of heat gas circulation through the tubing is requiredto preventdead space ventilation

alveolar PCO2 to fall to ~0 mm Hg in downstream alveolicausesalveolar PCO2 to fall to ~0 mm Hg in downstream alveoli

in worse ventilatory efficiency and greater impairment of exercise capacityresultedin worse ventilatory efficiency and greater impairment of exercise capacity

as pulmonary arterial perfusion had been excluded , which therefore created physiologic mismatching in the right lunghad resultedas pulmonary arterial perfusion had been excluded , which therefore created physiologic mismatching in the right lung

to the limits of acceptable respiratory acidosis in this caseleadingto the limits of acceptable respiratory acidosis in this case

in the microcirculation by neutrophilsis createdin the microcirculation by neutrophils

to a rise in PaCO2leadingto a rise in PaCO2

from the great reduction in tidal volumeresultingfrom the great reduction in tidal volume

from pulmonary micro - thrombi , right heart failure or AT1 receptor - mediated pulmonary vasoconstrictionresultingfrom pulmonary micro - thrombi , right heart failure or AT1 receptor - mediated pulmonary vasoconstriction

in lower PETCO 2 and an increased ( a - et)pco 2 gradientresultingin lower PETCO 2 and an increased ( a - et)pco 2 gradient

in heat loss without necessarily increasing oxygen uptake or carbon dioxide lossresultingin heat loss without necessarily increasing oxygen uptake or carbon dioxide loss

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