NMDA and AMPA ( alpha amino 3 hydroxy 5 methyl isoxazolpropionic acid ) receptor activation and the development of central sensitization.[44preventsNMDA and AMPA ( alpha amino 3 hydroxy 5 methyl isoxazolpropionic acid ) receptor activation and the development of central sensitization.[44

The decrease in renal prostaglandin synthesis(passive) caused byThe decrease in renal prostaglandin synthesis

gastrointestinal side effectscausesgastrointestinal side effects

so many of the side effects of NSAIDscauseso many of the side effects of NSAIDs

gastrointestinal [ 20 ] and cardiovascular side effects.[21can causegastrointestinal [ 20 ] and cardiovascular side effects.[21

to the cord scdboth dorsal columns and the scans can also be encouraged in patients taking lithium for mixed episodes dosagecontributesto the cord scdboth dorsal columns and the scans can also be encouraged in patients taking lithium for mixed episodes dosage

ABLpreventedABL

the effect of astrocyte BAPTApreventsthe effect of astrocyte BAPTA

tumorigenesis in humans ( 34 ... and similarly , inhibition of COX reduced proliferation and cystogenesis in genetically nonorthologous models of ADPKD ( 35preventstumorigenesis in humans ( 34 ... and similarly , inhibition of COX reduced proliferation and cystogenesis in genetically nonorthologous models of ADPKD ( 35

Endothelium - dependent relaxation of HPRA resistant to NOS and(passive) was preventedEndothelium - dependent relaxation of HPRA resistant to NOS and

synthesis of certain prostaglandins [ physiologically active substances with diverse hormone - like effects ] , which augment the pain - inducing effects of certain chemicals released on tissue injury or in response to various kinds of stimulipreventssynthesis of certain prostaglandins [ physiologically active substances with diverse hormone - like effects ] , which augment the pain - inducing effects of certain chemicals released on tissue injury or in response to various kinds of stimuli

the generation of inflammatory mediators , like prostaglandins , that further increase the propagation of nociceptive painpreventsthe generation of inflammatory mediators , like prostaglandins , that further increase the propagation of nociceptive pain

cell proliferation and promotes apoptosispreventscell proliferation and promotes apoptosis

the airways(passive) caused bythe airways

in increased IFN - γ production in SCID / BALB / c mice treated with ODN 1826resultedin increased IFN - γ production in SCID / BALB / c mice treated with ODN 1826

to prevention of TXA2 synthesis and impairment of platelet secretion and aggregationleadsto prevention of TXA2 synthesis and impairment of platelet secretion and aggregation

in suppression of physiologically important prostaglandinsresultingin suppression of physiologically important prostaglandins

ocular angiogenesis in an animal modelpreventingocular angiogenesis in an animal model

any unexpected changes in neural activity(passive) caused byany unexpected changes in neural activity

to greater increases in blood pressure and limb vascular resistance compared with placebo during inspiratory muscle metaboreflex activationledto greater increases in blood pressure and limb vascular resistance compared with placebo during inspiratory muscle metaboreflex activation

to the right ventricle and the councils on clinical cardiology , stroke councilcontributesto the right ventricle and the councils on clinical cardiology , stroke council

to atherosclerotic plaque destabilization in human patients with recent ischemic events [ 69 ] and endothelial dysfunction during hypertension in humans [ 70leadsto atherosclerotic plaque destabilization in human patients with recent ischemic events [ 69 ] and endothelial dysfunction during hypertension in humans [ 70

gastrointestinal Inhibitors , Modulators , Libraries bleeding and ulcers , and COX 2 inhi bition is associated to anti inflammatory , antipyretic and analgesic effectsmay causegastrointestinal Inhibitors , Modulators , Libraries bleeding and ulcers , and COX 2 inhi bition is associated to anti inflammatory , antipyretic and analgesic effects

in decrease of prostaglandin E2 in the gastric mucosa [ 21 , 22resultingin decrease of prostaglandin E2 in the gastric mucosa [ 21 , 22

to virulence by binding reversibly to the patientcontributesto virulence by binding reversibly to the patient

differential effects on kynurenine metabolismprovokesdifferential effects on kynurenine metabolism

renal and splanchnic vasoconstriction and selectively increases the sensitivity of the renal vascular bed tocausesrenal and splanchnic vasoconstriction and selectively increases the sensitivity of the renal vascular bed to

to an increase in intracellular arachidonic acidcan leadto an increase in intracellular arachidonic acid

to acute vasorelaxation of AA despite continued ET administration , without affecting EA constriction in vivo , thereby resulting in a dramatic reversal of the effects of ET on GFRleadsto acute vasorelaxation of AA despite continued ET administration , without affecting EA constriction in vivo , thereby resulting in a dramatic reversal of the effects of ET on GFR

to a decrease in PGE2 levelsleadsto a decrease in PGE2 levels

in WT levels of IL-5 and IL-13 lung mRNA and cytokine protein in STAT6resultedin WT levels of IL-5 and IL-13 lung mRNA and cytokine protein in STAT6

to tau hyperphosphorylation and Aβ deposition ... whereas the activity of COX is inhibited by the increased levels of Aβ that eventually results in cell death ( Cassarino and Bennett , 1999leadsto tau hyperphosphorylation and Aβ deposition ... whereas the activity of COX is inhibited by the increased levels of Aβ that eventually results in cell death ( Cassarino and Bennett , 1999

gastrointestinal bleeding and ulcers ... and COX 2 inhi bition is associated to anti inflammatory , antipyretic and analgesic effectsmay causegastrointestinal bleeding and ulcers ... and COX 2 inhi bition is associated to anti inflammatory , antipyretic and analgesic effects

to decreases in cytoprotective prostaglandins ( PGsleadingto decreases in cytoprotective prostaglandins ( PGs

to longer icu stays , more days per month in patients requiring labyrinthectomy , the e ects m dic ion m y m a a a. Unit of the femur and is not achieved after years of age see tables , consequentlycontributesto longer icu stays , more days per month in patients requiring labyrinthectomy , the e ects m dic ion m y m a a a. Unit of the femur and is not achieved after years of age see tables , consequently

in reduction of prostaglandin in gastric mucosaresultingin reduction of prostaglandin in gastric mucosa

a reduction of PGE2 levels together with an enhanced AHRcauseda reduction of PGE2 levels together with an enhanced AHR

to decrease in inflammatory mediatorleadingto decrease in inflammatory mediator

in decreased PGE2resultingin decreased PGE2

to its anti - inflammatory and analgesic effectsleadingto its anti - inflammatory and analgesic effects