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Qaagi - Book of Why

Causes

Effects

Long - term exposure of freshly isolated human CD4 + T cells tocausedIL-12 in vitro

Both 5beta,6beta - epoxycholesterol and its metabolite , cholestan-6-oxo-3,5-diol , have been shownto causecytotoxicity in vitro

Gorczynski R , Chen Z , Khatri I , Yu K. sCD200 present in mice receiving cardiac and skin allograftscausesimmunosuppression in vitro

their potentialto triggersynaptogenesis in vitro

conditionstriggeruncoating in vitro

Myr treatmentpreventedubiquitination in vitro

serial passaging [ 6 - 9(passive) caused byreplicative senescence in vitro

the abilityto preventAd5 transduction in vitro

a concentration - dependent net increase in superoxide anion in mitochondria from Sod2(+/- ... but not Sod2(+/+ ) mice [ 25].Furthermore , the nimesulide - treated mutant mice exhibited increased hepatic cytosolic levels of cytochrome c and caspase-3 activity , as well as increased numbers of apoptotic hepatocytes [ 25causeda concentration - dependent net increase in superoxide anion in mitochondria from Sod2(+/- ... but not Sod2(+/+ ) mice [ 25].Furthermore , the nimesulide - treated mutant mice exhibited increased hepatic cytosolic levels of cytochrome c and caspase-3 activity , as well as increased numbers of apoptotic hepatocytes [ 25

pallor of the regulation of signal function under controlled if artii cial conduit between the beginning of your family member expressed in the xenopus dorsalizing factor gremlin identifies a novel class of proteins that antagonize bmp activities mol cell biol a carroll t j and petit ccan causepallor of the regulation of signal function under controlled if artii cial conduit between the beginning of your family member expressed in the xenopus dorsalizing factor gremlin identifies a novel class of proteins that antagonize bmp activities mol cell biol a carroll t j and petit c

immediatelyleadsimmediately

rats(passive) caused byrats

in increased mitochondrial ROS production , responsible for reduced cell proliferationresultedin increased mitochondrial ROS production , responsible for reduced cell proliferation

platelet density(passive) caused byplatelet density

cells(passive) caused bycells

one form of DNA damage ( DNA strand breaks(passive) caused byone form of DNA damage ( DNA strand breaks

inhibition of [ 3H]dizocilpine binding onlycausedinhibition of [ 3H]dizocilpine binding only

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