Long - term exposure of freshly isolated human CD4 + T cells tocausedIL-12 in vitro
Both 5beta,6beta - epoxycholesterol and its metabolite , cholestan-6-oxo-3,5-diol , have been shownto causecytotoxicity in vitro
Gorczynski R , Chen Z , Khatri I , Yu K. sCD200 present in mice receiving cardiac and skin allograftscausesimmunosuppression in vitro
their potentialto triggersynaptogenesis in vitro
conditionstriggeruncoating in vitro
Myr treatmentpreventedubiquitination in vitro
serial passaging [ 6 - 9(passive) caused byreplicative senescence in vitro
the abilityto preventAd5 transduction in vitro
a concentration - dependent net increase in superoxide anion in mitochondria from Sod2(+/- ... but not Sod2(+/+ ) mice [ 25].Furthermore , the nimesulide - treated mutant mice exhibited increased hepatic cytosolic levels of cytochrome c and caspase-3 activity , as well as increased numbers of apoptotic hepatocytes [ 25causeda concentration - dependent net increase in superoxide anion in mitochondria from Sod2(+/- ... but not Sod2(+/+ ) mice [ 25].Furthermore , the nimesulide - treated mutant mice exhibited increased hepatic cytosolic levels of cytochrome c and caspase-3 activity , as well as increased numbers of apoptotic hepatocytes [ 25
pallor of the regulation of signal function under controlled if artii cial conduit between the beginning of your family member expressed in the xenopus dorsalizing factor gremlin identifies a novel class of proteins that antagonize bmp activities mol cell biol a carroll t j and petit ccan causepallor of the regulation of signal function under controlled if artii cial conduit between the beginning of your family member expressed in the xenopus dorsalizing factor gremlin identifies a novel class of proteins that antagonize bmp activities mol cell biol a carroll t j and petit c
immediatelyleadsimmediately
rats(passive) caused byrats
in increased mitochondrial ROS production , responsible for reduced cell proliferationresultedin increased mitochondrial ROS production , responsible for reduced cell proliferation
platelet density(passive) caused byplatelet density
cells(passive) caused bycells
one form of DNA damage ( DNA strand breaks(passive) caused byone form of DNA damage ( DNA strand breaks
inhibition of [ 3H]dizocilpine binding onlycausedinhibition of [ 3H]dizocilpine binding only