CD4 + T cell intrinsic or extrinsic IRF3 activityinfluencedTh17 differentiation
by a recessive niche - filling allele of the Ikaros transcription factor(passive) caused byhematolymphoid differentiation
a recessive niche - filling allele of the Ikaros transcription factor(passive) caused byhematolymphoid differentiation
When Wnt is activated , KGF i.e. Keratinocyte Growth Factor is releasedcausesKeratinocyte differentiation
Most of these agentscausemyeloid differentiation
miRNAs alsoinfluenceadipocyte differentiation
that treatment with alkali results in the nanonetwork structures on the titanium surfaceinfluenceBMMSCs osteogenic differentiation
by the expression of PPAR&gamma(passive) is ... influenced byadipocyte differentiation
miRNAsinfluenceadipocyte differentiation
the powerto influenceTh17 differentiation
which in turn causes the expression of GFAPcausingglial differentiation
Certain proteins secreted or existing in the cell , and interactions between neighboring cells , are knownto causedifferentiation
Induced differentiation from stem cells Culture of human embryonic stem cells in mitotically inactivated porcine ovarian fibroblasts ( POFcausesdifferentiation
The growth factor deprivation ... which have been shownto influencetrophoblast differentiation
Vitamin D analogues are thoughtto influencekeratinocyte differentiation
hormonal factorscausegonadal differentiation
the expression of tissue specific transcription factors(passive) is caused byDifferentiation
Alteration in the levels of these factors in ES cellswill causedifferentiation
to tendon regeneration via the FAK / ERK1/2 pathwayleadingto tendon regeneration via the FAK / ERK1/2 pathway
to increased fat depositionleadingto increased fat deposition
to the mature adipocyte phenotypeleadingto the mature adipocyte phenotype
to the tumor vasculaturecontributingto the tumor vasculature
to tissue hypertrophy and insulin resistanceleadingto tissue hypertrophy and insulin resistance
in formation of the prochordal plate ( 1 , 2 , 3resultsin formation of the prochordal plate ( 1 , 2 , 3
in transient infertilityresultingin transient infertility
to bloodleadingto blood
the increase in plasmatocyte and crystal cells that we see following loss of the permeability barrier(passive) is caused bythe increase in plasmatocyte and crystal cells that we see following loss of the permeability barrier
of ganglion cellscomposedof ganglion cells
in tumorigenesisresultingin tumorigenesis
normal and tumoral breast epithelial cell proliferation and migrationinfluencesnormal and tumoral breast epithelial cell proliferation and migration
several days laterresultsseveral days later
in the lungmost likely originatingin the lung
to changes in the metabolism , structure , and gene expression patterns of cellsleadsto changes in the metabolism , structure , and gene expression patterns of cells
in the formation and maturation of cells responsible for bone remodeling 67 - 69resultingin the formation and maturation of cells responsible for bone remodeling 67 - 69
eventually to the hundreds of distinct cell types in our mature bodies , from retinal cells to those of heart muscle , from liver to bone , from brain to pancreasleadingeventually to the hundreds of distinct cell types in our mature bodies , from retinal cells to those of heart muscle , from liver to bone , from brain to pancreas
cells with different properties and capabilities including their tumorigenic potentialresultingcells with different properties and capabilities including their tumorigenic potential
to therapy resistance and disease progressionmay contributeto therapy resistance and disease progression
to insulin resistanceleadingto insulin resistance
a reduction in the number of new cells becoming neuronscausinga reduction in the number of new cells becoming neurons
to : Accumulation of immature myeloid forms ( blasts ) in the bone marrowleadingto : Accumulation of immature myeloid forms ( blasts ) in the bone marrow
type settles in Philadelphia hoping syndrome to further technologicalcausestype settles in Philadelphia hoping syndrome to further technological
to tumor formationleadingto tumor formation
to improved and faster tissue developmentleadingto improved and faster tissue development
to a particular mature cellleadingto a particular mature cell
to malignant cell evolutionleadingto malignant cell evolution
to an upregulation of FUT4 and MCL1 ( Fig . 6bleadingto an upregulation of FUT4 and MCL1 ( Fig . 6b
to the expression of the terminal phenotypeleadsto the expression of the terminal phenotype
to Differential advantage in which the firm gets the premium in the market , which is more than the cost of providing differentiationcan leadto Differential advantage in which the firm gets the premium in the market , which is more than the cost of providing differentiation
in a loss of mRNA expressionresultingin a loss of mRNA expression
to the production of the different body cellleadsto the production of the different body cell
in growth arrest , differentiation , and cell deathresultsin growth arrest , differentiation , and cell death
to the formation of various organs and organ systemsleadsto the formation of various organs and organ systems
in new fat creation ( adipogenesis ) and blood vessel formation ( angiogenesisresultingin new fat creation ( adipogenesis ) and blood vessel formation ( angiogenesis
to organ formationleadingto organ formation
in the specialization of cells for specific functionsresultsin the specialization of cells for specific functions
to non - coexistence of species and niche differentiationleadingto non - coexistence of species and niche differentiation
in the expansion of immature progenitor cellsresultingin the expansion of immature progenitor cells
from the controlled activation and de - activation of genesresultsfrom the controlled activation and de - activation of genes