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by global warming(passive) caused by1)climate change
by the fault(passive) caused byfault;The change
by the fact that (passive) is ... caused by24)the change
the fact(passive) is chiefly caused by24)the change
by technology(passive) heavily influenced bying change
these factors(passive) caused byhaemodynamic change
Syria conflictcausedby?climate change
the following factors : ( a ) site conditions , ( b ) the character of the events initiating succession ( perturbations ) , ( c(passive) can be influenced bySuccessional change
these conditions(passive) caused byhistologic change
two suites of variablesmay causeavifaunal change
outside influencescausingorganismal change
G , L , and T ... two suites of variablesmay causeavifaunal change
by outburst floods and debris flows(passive) caused byGeomorphic change
Outburst Floods and Debris Flows at Mount Rainier , Washington(passive) Caused byGeomorphic Change
long - term L - dopa treatment as evidenced by reducing the rise of nigral GABA and striatal Glu(passive) caused byneurochemical change
architect - artists Blaithin Quinn , Jo Anne Butler and Tara Kennedy and architects Dominic Stevens and Peter Tansey(passive) was led byDemense Change
architect - artists Blaithin Quinn , Jo Anne Butler and Tara Kennedy and architects Dominic Stevens and Peter Tansey , during which Blaithin Quinn concurrently led a workshop entitled Field Dynamics(passive) was led byDemense Change
by agonist binding to the receptor(passive) caused byconformational change
The binding of the ligandcausesconformational change
Ligand - bindingcausesconformational change
a binding ligandcausesconformational change
free Ligand - bindingcausesconformational change
The binding of two acetylcholine moleculescausesconformational change
by the binding to inhibitor(passive) caused byconformational change
Activation of the extracellular portion of this receptorcausesconformational change
Ca2 binds to troponin Ccausingconformational change
Binds repressor protein ... 17causingconformational change
Bindingcausesconformational change
Activation of the extracellular portion of these receptorscausesconformational change
proteincausingconformational change
by GTP hydrolysis(passive) caused byconformational change
hydrolysis of GTP(passive) is caused byConformational change
The binding of erythropoietin ( EPO ) to its dimerised cell surface receptorcausesconformational change
GTP hydrolysis in one tetramer(passive) caused byconformational change
by phosphorylation(passive) caused byconformational change
by binding and release of Ca(2(passive) caused byconformational change
mutationcausingconformational change
mutation ... causingconformational change
mutationcausingconformational change
by the mutation(passive) caused byconformational change
extinction is not on the tablecausedextinction is not on the table
alsoleadalso
less precipitation this yearcausesless precipitation this year
to the loss of the very occurrencewould leadto the loss of the very occurrence
further obstructioncausingfurther obstruction
from long - term erosionresultingfrom long - term erosion
CV abendcausesCV abend
alteration of snowcausingalteration of snow
from both natural and anthropogenic activities , primarily within San Francisco Bayresultingfrom both natural and anthropogenic activities , primarily within San Francisco Bay
in autophosphorylation and activation of the kinase catalytic domain of JAK2resultsin autophosphorylation and activation of the kinase catalytic domain of JAK2
to inhibition of dimerization and enzyme activationleadingto inhibition of dimerization and enzyme activation
in autophosphorylation and activation of the kinase catalytic domain of JAK2 , a member of the Janus kinase family ( Figresultsin autophosphorylation and activation of the kinase catalytic domain of JAK2 , a member of the Janus kinase family ( Fig
to opening of ion channelleadingto opening of ion channel
its activation through increase theresultsits activation through increase the
its activation through increaseresultsits activation through increase
in channel openingresultsin channel opening
to channel openingleadsto channel opening
to activationleadingto activation
its activationresultsits activation
to mitochondrial docking and to an increase of membrane contact sitesleadingto mitochondrial docking and to an increase of membrane contact sites
to coactivator activationleadingto coactivator activation
in dissociation of corepressorsresultingin dissociation of corepressors
to the exposure of more hydrophobic regionsleadingto the exposure of more hydrophobic regions
the dissociation of the tetramer to regulatory subunit dimer and two active catalytic subunitscausesthe dissociation of the tetramer to regulatory subunit dimer and two active catalytic subunits
to a cytoplasmic segment of the complexleadingto a cytoplasmic segment of the complex
to K release K Pleadsto K release K P
in the receptor s shape being in the active stateresultingin the receptor s shape being in the active state
in enzymatic activityresultingin enzymatic activity
the channel to open or closecausesthe channel to open or close
from eithercan originatefrom either
from either cytoplasmic extracellularcan originatefrom either cytoplasmic extracellular
to high affinity for extracellular ligandsleadingto high affinity for extracellular ligands
amino acid transportcausesamino acid transport
to functionally inactive formleadingto functionally inactive form
from either cytoplasmic or extracellular interactionscan originatefrom either cytoplasmic or extracellular interactions
to actin polymerizationleadsto actin polymerization
from the amino acid substitutionresultingfrom the amino acid substitution
T2 ) energy level(passive) caused byT2 ) energy level
to activation of G - protein present at the C - terminal cytosolic side , which leads to the regulation of various downstream signaling pathwaysleadsto activation of G - protein present at the C - terminal cytosolic side , which leads to the regulation of various downstream signaling pathways
to the development of steric hindrance and affects the ability of EGFR TKI to bind to the ATP - kinase pocketleadsto the development of steric hindrance and affects the ability of EGFR TKI to bind to the ATP - kinase pocket