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Qaagi - Book of Why

Causes

Effects

around 2ms • factorsinfluencingconduction velocity

a coating around axons , called myelin(passive) is influenced byConduction speed

The principal factorsinfluencingconduction velocity

amiodarone effect(passive) caused byconduction velocity

backward propagation > factorsinfluencingconduction velocity

1 ms . FactorsInfluencingthe Conduction Velocity

axon diameter ( larger � faster ) and myelin sheath ( presence � faster , through saltatory conduction(passive) influenced byConduction velocities

internodal length in fibers over a range of short internodal lengths between 0.2 and 0.6 mm in genetically modified mice(passive) are ... influenced byconduction velocities

The distance between the stimulating electrode and receiving electrode ... impulse latencyresultsin conduction velocity

The distance between the stimulating and receiving electrodes is divided by the impulse latencyresultingin conduction velocity

both axon diameter and the presence or absence of myelination(passive) is influenced byConduction Speed

15 mmol / L DAM(passive) caused byconduction velocities

abnormally(passive) may be caused异常大的传导速度大小可以由远场信号引起,因为这些可以经常产生非常小的峰峰Ea值 。 Conduction velocity

The rate of phase 0 depolarizationinfluencesconduction velocity

strongly(passive) is ... influencedConduction velocity

the altered source / sink ratios due to the changed axial / perpendicular resistance ratiomight have influencedthe conduction velocity

reduced nodal Na+ permeabilitycausingdiminished conduction velocity

Another factorinfluencesconduction velocity

As a determinant of intercellular conductance , gap - junctional coupling is consideredto influencemyocardial conduction velocity

acute ischemia and metabolic stress35–37 without affecting sarcolemmal ion channels.16,37–39(passive) caused byconduction velocity

The as yet unknown ion channel properties in these unmyelinated regions of cortical neuronswill also influenceconduction velocity

usually(passive) is ... setConduction velocity

Peripheral nervous systemcan resultin the conduction velocity

the depolarizing force(passive) is influenced byConduction velocity

Axonal diameterinfluencesconduction velocity

spatial heterogeneities of refractory periods(passive) caused bylocal conduction velocity

hypoxia ( 13(passive) caused byconduction velocity

atrial volume increase(passive) caused bya slowed conduction velocity

reduced K+ concentrations after hemodialysis(passive) caused byconduction velocity

large and systematic differences in these parametersinfluencingconduction velocity

to 0.25 m s−1(passive) was setConduction velocity

skin temperaturecan influenceconduction velocity

degree of myelination(passive) set byconduction velocity (

myocardial fibre orientation is knownto influenceconduction velocity

few patientscan causea - v conduction velocity

The axons in peripheral nerves supplying the bone ... both of whichcontributeto conduction velocity

changes in myelin for spared remyelinated RST axons with the physical parameters measured in our studies(passive) caused byconduction velocity

effectts ischemia or deposition of collagens after an acute myocardial infarctionresultingeffectts ischemia or deposition of collagens after an acute myocardial infarction

to heart block or other arrhythmiascan leadto heart block or other arrhythmias

in national conduction block)allofresultingin national conduction block)allof

conduction block and ventricular tachycardiacausesconduction block and ventricular tachycardia

to the discovery that one form ( type 1 ) has the features of a demyelinating neuropathyledto the discovery that one form ( type 1 ) has the features of a demyelinating neuropathy

WL shortening ( Figure 1BpreventingWL shortening ( Figure 1B

in conduction blockade with su ciently high frequenciesresultingin conduction blockade with su ciently high frequencies

in an increased temporal summation leading to an increased EMG amplitude response of the following muscle contractionmay resultin an increased temporal summation leading to an increased EMG amplitude response of the following muscle contraction

to a sudden ( within seconds to minutes ) decrease of the number of waveletsleadingto a sudden ( within seconds to minutes ) decrease of the number of wavelets

to higher contractility and thus an increase in mechanical response produced by the anodal stimulationmay leadto higher contractility and thus an increase in mechanical response produced by the anodal stimulation

to atrioventricular block ( 14leadingto atrioventricular block ( 14

from the match between current and loadresultingfrom the match between current and load

to locomotion defects and weakness in targeted limbs ( Kuwabara , 2007 ; Sun et al . , 2009leadingto locomotion defects and weakness in targeted limbs ( Kuwabara , 2007 ; Sun et al . , 2009

to arrhythmias and sudden cardiac death ( 12–14leadsto arrhythmias and sudden cardiac death ( 12–14

for each organismis ... designedfor each organism

as a biological reasonable value 2.4 mm /is setas a biological reasonable value 2.4 mm /

the duration of the action potential ( 2 , 30influencesthe duration of the action potential ( 2 , 30

in the animal being dead nearly instantlyresultingin the animal being dead nearly instantly

in increased motor functionresultsin increased motor function

in negative chronotropic , dromotropic , and inotropic changes in the heartresultingin negative chronotropic , dromotropic , and inotropic changes in the heart

in transverse propagation since the longitudinal conduction depends mainly on sodium channel availabilitywill resultin transverse propagation since the longitudinal conduction depends mainly on sodium channel availability

from loss of PKP2 expressionresultingfrom loss of PKP2 expression

in prolonged PR interval and progressive widening of the QRS complex and nodal and ventricular arrhythmiasresultingin prolonged PR interval and progressive widening of the QRS complex and nodal and ventricular arrhythmias

positive inotropic effectcausingpositive inotropic effect

to lengthening of the cardiac cycle and eventually ventricular tachycardia , fibrillation , and asystoleleadingto lengthening of the cardiac cycle and eventually ventricular tachycardia , fibrillation , and asystole

to an increased cardiac excitability ( hyper - excitabilityleadingto an increased cardiac excitability ( hyper - excitability

generallycausegenerally

Several cardiac dysfunctions and arrhythmia(passive) are caused bySeveral cardiac dysfunctions and arrhythmia

to be equal in all directionswas setto be equal in all directions

in less coordinated input to the somaresultingin less coordinated input to the soma

from short stretches of axons now supporting rapid saltatory conductionresultingfrom short stretches of axons now supporting rapid saltatory conduction

from Na+ channel inactivation owing to the strong dendritic depolarization during the EPSPmay have resultedfrom Na+ channel inactivation owing to the strong dendritic depolarization during the EPSP

to functional instabilitymay contributeto functional instability

from the various distributions of INa and INaP described in the Results sectionresultingfrom the various distributions of INa and INaP described in the Results section

to a functional block.8leadingto a functional block.8

from the 3-D correction procedureresultingfrom the 3-D correction procedure

in differences in the spatial summation at central synapsesresultingin differences in the spatial summation at central synapses

from prematurity and the presence of myofibroblast clustersresultingfrom prematurity and the presence of myofibroblast clusters

from stimulation of the lumbar regionresultingfrom stimulation of the lumbar region

up in the wake of the first responsesetup in the wake of the first response

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